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Presenilin-1 P264L Knock-In Mutation: Differential Effects on Aβ Production, Amyloid Deposition, and Neuronal Vulnerability por Robert Siman, Andrew G. Reaume, Mary J. Savage, Stephen P. Trusko, Yin‐Guo Lin, Richard Scott, Dorothy G. Flood
Publicado em 2000Artigo -
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Biology
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Internal medicine
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Alzheimer's disease
Amyloid precursor protein
Biochemistry
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Genetically modified mouse
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Presenilin
Transgene
Amyotrophic lateral sclerosis
Cyclin-dependent kinase 5
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Dismutase
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Gene knockin
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Knockout mouse
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Mitogen-activated protein kinase kinase
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