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  1. 1
    Vitamin D supplementation for four months causes cardiac hypertrophy and diastolic dysfunction in normal rats

    Vitamin D supplementation for four months causes cardiac hypertrophy and diastolic dysfunction in normal rats by Dos Santos, Bruna Paola Murino Rafacho, Andréa Gonçalves, Renata Cândido, Vanessa M. Ferreira, Meliza Goi Roscani, Marcos Ferreira Minicucci, Álvaro Oscar Campana, Leonardo A. M. Zornoff, Paula S. Azevedo, Sérgio Alberto Rupp de Paiva

    Published 2013
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  2. 2
    Cardiac fibroblasts: from development to heart failure

    Cardiac fibroblasts: from development to heart failure by Thomas Moore‐Morris, Nuno Guimarães‐Camboa, Katherine E. Yutzey, Michel Pucéat, Sylvia Μ. Evans

    Published 2015
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  3. 3
    Influence of Physical Activity on Hypertension and Cardiac Structure and Function

    Influence of Physical Activity on Hypertension and Cardiac Structure and Function by Sheila M. Hegde, Scott D. Solomon

    Published 2015
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  4. 4
    Cardiotrophin-1 Activates a Distinct Form of Cardiac Muscle Cell Hypertrophy

    Cardiotrophin-1 Activates a Distinct Form of Cardiac Muscle Cell Hypertrophy by Kai C. Wollert, Tetsuya Taga, Mikiyoshi Saito, Masashi Narazaki, Tadamitsu Kishimoto, Christopher C. Glembotski, Ann B. Vernallis, John K. Heath, Diane Pennica, William I. Wood, Kenneth R. Chien

    Published 1996
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  5. 5
    Molecular characterization of the stretch-induced adaptation of cultured cardiac cells. An in vitro model of load-induced cardiac hypertrophy.

    Molecular characterization of the stretch-induced adaptation of cultured cardiac cells. An in vitro model of load-induced cardiac hypertrophy. by Junichi Sadoshima, L. Jahn, Toshiyuki Takahashi, Thomas J. Kulik, Seigo Izumo

    Published 1992
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  6. 6
    Raf-1 kinase activity is necessary and sufficient for gene expression changes but not sufficient for cellular morphology changes associated with cardiac myocyte hypertrophy.

    Raf-1 kinase activity is necessary and sufficient for gene expression changes but not sufficient for cellular morphology changes associated with cardiac myocyte hypertrophy. by J. Thorburn, Martin McMahon, Andrew Thorburn

    Published 1994
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  7. 7
    Sex Hormones Promote Opposite Effects on ACE and ACE2 Activity, Hypertrophy and Cardiac Contractility in Spontaneously Hypertensive Rats

    Sex Hormones Promote Opposite Effects on ACE and ACE2 Activity, Hypertrophy and Cardiac Contractility in Spontaneously Hypertensive Rats by Polyana Lima Meireles Dalpiaz, Aline Zandonadi Lamas, Izabela Facco Caliman, Rogério Faustino Ribeiro Júnior, G.R. Abreu, Margareth Ribeiro Moysés, Tadeu Uggere de Andrade, Sônia Alves Gouvêa, Márcio F.M. Alves, Adriana K. Carmona, Nazaré Souza Bissoli

    Published 2015
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  8. 8
    Cardiac Myocyte De Novo DNA Methyltransferases 3a/3b Are Dispensable for Cardiac Function and Remodeling after Chronic Pressure Overload in Mice

    Cardiac Myocyte De Novo DNA Methyltransferases 3a/3b Are Dispensable for Cardiac Function and Remodeling after Chronic Pressure Overload in Mice by Thomas Nührenberg, N. Hammann, Tilman Schnick, Sebastian Preißl, Anika Witten, Monika Stoll, Ralf Gilsbach, Franz‐Josef Neumann, Lutz Hein

    Published 2015
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  9. 9
    Myocardial pressure overload induces systemic inflammation through endothelial cell IL-33

    Myocardial pressure overload induces systemic inflammation through endothelial cell IL-33 by Wei‐Yu Chen, Jaewoo Hong, Joseph Gannon, Rahul Kakkar, Richard Lee

    Published 2015
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  10. 10
    Mechanical loading stimulates cell hypertrophy and specific gene expression in cultured rat cardiac myocytes. Possible role of protein kinase C activation.

    Mechanical loading stimulates cell hypertrophy and specific gene expression in cultured rat cardiac myocytes. Possible role of protein kinase C activation. by Issei Komuro, Youichi Katoh, T Kaida, Y Shibazaki, Masahiko Kurabayashi, Eitetsu Hoh, Fumimaro Takaku, Y Yazaki

    Published 1991
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  11. 11
    Early Spironolactone Treatment Attenuates Heart Failure Development by Improving Myocardial Function and Reducing Fibrosis in Spontaneously Hypertensive Rats

    Early Spironolactone Treatment Attenuates Heart Failure Development by Improving Myocardial Function and Reducing Fibrosis in Spontaneously Hypertensive Rats by M. D. M. Cezar, R. L. Damatto, Luana Urbano Pagan, A. R. R. Lima, Paula Felippe Martinez, Camila Bonomo, C. M. Rosa, Dijon Henrique Salomé de Campos, Antônio Carlos Cicogna, Mariana Janini Gomes, S. A. Oliveira, Daniella Audi Blotta, Marina Politi Okoshi, Katashi Okoshi

    Published 2015
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  12. 12
    The effect of enalapril on the cardiac remodelling in ovariectomized spontaneously hypertensive rats

    The effect of enalapril on the cardiac remodelling in ovariectomized spontaneously hypertensive rats by Wellington V. Santos, Leila Maria Meirelles Pereira, Carlos Alberto Mandarim‐de‐Lacerda

    Published 2004
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  13. 13
    Sexual dimorphism in autonomic changes and in the renin–angiotensin system in the hearts of mice subjected to thyroid hormone‐induced cardiac hypertrophy

    Sexual dimorphism in autonomic changes and in the renin–angiotensin system in the hearts of mice subjected to thyroid hormone‐induced cardiac hypertrophy by Anderson Luiz Bezerra da Silveira, Manuela França de Souza Miranda, André S. Mecawi, Roberto Laureano‐Melo, Michelle Porto Marassi, Alba Cenélia Matos da Silva, José Antunes‐Rodrigues, Emerson L. Olivares

    Published 2014
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  14. 14
    Myotrophin: purification of a novel peptide from spontaneously hypertensive rat heart that influences myocardial growth.

    Myotrophin: purification of a novel peptide from spontaneously hypertensive rat heart that influences myocardial growth. by S Sen, Gopal C. Kundu, Nagy Mekhail, Julien Castel, K S Misono, Bernadine Healy

    Published 1990
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  15. 15
    Emerging concepts for the role of TRP channels in the cardiovascular system

    Emerging concepts for the role of TRP channels in the cardiovascular system by Rudi Vennekens

    Published 2010
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  16. 16
    Matricellular Proteins: New Molecular Targets To Prevent Heart Failure

    Matricellular Proteins: New Molecular Targets To Prevent Heart Failure by Hiroshi Okamoto, Kyoko Imanaka‐Yoshida

    Published 2011
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  17. 17
    Natriuretic Peptides and Cardiometabolic Health

    Natriuretic Peptides and Cardiometabolic Health by Deepak K. Gupta, Thomas J. Wang

    Published 2015
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  18. 18
    Blood-based microRNA signatures differentiate various forms of cardiac hypertrophy

    Blood-based microRNA signatures differentiate various forms of cardiac hypertrophy by Anselm A. Derda, Sabrina Thum, Johan M. Lorenzen, Udo Bavendiek, Joerg Heineke, Britta Keyser, Manfred Stuhrmann, Raymond C. Givens, Peter J. Kennel, P. Christian Schulze, Julian D. Widder, Johann Bauersachs, Thomas Thum

    Published 2015
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  19. 19
    Monoamine Oxidase B Prompts Mitochondrial and Cardiac Dysfunction in Pressure Overloaded Hearts

    Monoamine Oxidase B Prompts Mitochondrial and Cardiac Dysfunction in Pressure Overloaded Hearts by Nina Kaludercic, Andrea Carpi, Takahiro Nagayama, Vidhya Sivakumaran, Guangshuo Zhu, Edwin W. Lai, Djahida Bedja, Agnese De Mario, Kai Chen, Kathleen L. Gabrielson, Merry L. Lindsey, Karel Pacák, Eiki Takimoto, Jean C. Shih, David A. Kass, Fabio Di Lisa, Nazareno Paolocci

    Published 2013
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  20. 20
    Caveolin-3 Overexpression Attenuates Cardiac Hypertrophy via Inhibition of T-type Ca2+ Current Modulated by Protein Kinase Cα in Cardiomyocytes

    Caveolin-3 Overexpression Attenuates Cardiac Hypertrophy via Inhibition of T-type Ca2+ Current Modulated by Protein Kinase Cα in Cardiomyocytes by Yogananda S. Markandeya, Laura Phelan, Marites T. Woon, Alexis M. Keefe, Courtney Reynolds, Benjamin K. August, Timothy A. Hacker, David M. Roth, Hemal H. Patel, Ravi C. Balijepalli

    Published 2015
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