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Dissecting the role of sodium currents in visceral sensory neurons in a model of chronic hyperexcitability using Na_v1.8 and Na_v1.9 null mice por Kirk Hillsley, Jiahui Lin, Andre Stanisz, David Grundy, Jeroen Aerssens, Pieter J. Peeters, Diederik Moechars, Bernard Coulie, R. H. Stead

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Nav1.3 Sodium Channels: Rapid Repriming and Slow Closed-State Inactivation Display Quantitative Differences after Expression in a Mammalian Cell Line and in Spinal Sensory Neurons por Theodore Cummins, Fabio Aglieco, Muthukrishnan Renganathan, Raimund I. Herzog, Sulayman D. Dib‐Hajj, Stephen G. Waxman

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Mechanisms by which a <i>CACNA1H</i> mutation in epilepsy patients increases seizure susceptibility por Veit‐Simon Eckle, Aleksandr Shcheglovitov, Iuliia Vitko, Deblina Dey, Chan Choo Yap, Bettina Winckler, Edward Perez‐Reyes

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De novo KCNB1 mutations in infantile epilepsy inhibit repetitive neuronal firing por Hirotomo Saitsu, Tenpei Akita, Jun Tohyama, Hadassa Goldberg‐Stern, Yu Kobayashi, Roni Cohen, Mitsuhiro Kato, Chihiro Ohba, Satoko Miyatake, Yoshinori Tsurusaki, Mitsuko Nakashima, Noriko Miyake, Atsuo Fukuda, Naomichi Matsumoto

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Nav1.1 dysfunction in genetic epilepsy with febrile seizures-plus or Dravet syndrome por Linda Volkers, Kristopher M. Kahlig, Nienke E. Verbeek, Joost H.G. Das, Marjan J. A. van Kempen, Hans Stroink, Paul B. Augustijn, Onno van Nieuwenhuizen, Dick Lindhout, Alfred L. George, Bobby P.C. Koeleman, Martin B. Rook

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Downregulation of Tetrodotoxin-Resistant Sodium Currents and Upregulation of a Rapidly Repriming Tetrodotoxin-Sensitive Sodium Current in Small Spinal Sensory Neurons after Nerve I... por Theodore Cummins, Stephen G. Waxman

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Reduced dendritic arborization and hyperexcitability of pyramidal neurons in a Scn1b-based model of Dravet syndrome por Christopher A. Reid, Bryan Leaw, Kay Richards, Robert J. Richardson, Verena C. Wimmer, Christiaan Yu, Elisa L. Hill‐Yardin, Holger Lerche, Ingrid E. Scheffer, Samuel F. Berkovic, Steven Petrou

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Size Matters: Erythromelalgia Mutation S241T in Nav1.7 Alters Channel Gating por Angelika Lampert, Sulayman D. Dib‐Hajj, Lynda Tyrrell, Stephen G. Waxman

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Tetrodotoxin-Resistant Sodium Channels in Sensory Neurons Generate Slow Resurgent Currents That Are Enhanced by Inflammatory Mediators por Zhi‐Yong Tan, Andrew D. Piekarz, Birgit T. Priest, Kelly L. Knopp, Jeffrey L. Krajewski, Jeff S. McDermott, Eric S. Nisenbaum, Theodore Cummins

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A Mutation in Telethonin Alters Nav1.5 Function por Amelia Mazzone, Peter R. Strege, David J. Tester, Cheryl E. Bernard, Georgine Faulkner, Roberto De Giorgio, Jonathan C. Makielski, Vincenzo Stanghellini, Simon J. Gibbons, Michael J. Ackerman, Gianrico Farrugia

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A sodium channel knockin mutant (NaV1.4-R669H) mouse model of hypokalemic periodic paralysis por Fenfen Wu, Wentao Mi, Dennis K. Burns, Yu Fu, Hillery F. Gray, Arie Struyk, Stephen C. Cannon

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A rescuable folding defective Na_v1.1 (<i>SCN1A</i>) sodium channel mutant causes GEFS+: Common mechanism in Na_v1.1 related epilepsies? por Raffaella Rusconi, Romina Combi, Sandrine Cestèle, Daniele Grioni, Silvana Franceschetti, Leda DalprÃ, Massimo Mantegazza

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The Tetrodotoxin‐Resistant Na⁺ Channel Nav1.8 is Essential for the Expression of Spontaneous Activity in Damaged Sensory Axons of Mice por Carolina Roza, Jennifer M.A. Laird, Veronika Souslova, John N. Wood, Fernando Cerveró

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Correlations in timing of sodium channel expression, epilepsy, and sudden death in Dravet syndrome por Christine S. Cheah, Ruth E. Westenbroek, William H. Roden, Franck Kalume, John C. Oakley, Laura A. Jansen, William A. Catterall

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CRMP2 Protein SUMOylation Modulates NaV1.7 Channel Trafficking por Erik T. Dustrude, Sarah M. Wilson, Weina Ju, Yucheng Xiao, Rajesh Khanna

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Sodium channel SCN8A (Nav1.6): properties and de novo mutations in epileptic encephalopathy and intellectual disability por Janelle E. O’Brien, Miriam H. Meisler

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Specialized Functions of Nav1.5 and Nav1.9 Channels in Electrogenesis of Myenteric Neurons in Intact Mouse Ganglia por Nancy Osorio, S. М. Коrogod, Patrick Delmas

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Voltage-gated sodium channel organization in neurons: Protein interactions and trafficking pathways por Christophe Leterrier, Anna Brachet, Marie‐Pierre Fache, Bénédicte Dargent

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Phosphorylation of Sodium Channel Na_v1.8 by p38 Mitogen-Activated Protein Kinase Increases Current Density in Dorsal Root Ganglion Neurons por Andy Hudmon, Jin‐Sung Choi, Lynda Tyrrell, Joel A. Black, Anthony M. Rush, Stephen G. Waxman, Sulayman D. Dib‐Hajj

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Osteoarthritis-dependent changes in antinociceptive action of Nav1.7 and Nav1.8 sodium channel blockers: An in vivo electrophysiological study in the rat por Wael A. Rahman, Anthony H. Dickenson

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