Spliceosomal protein E regulates neoplastic cell growth by modulating expression of Cyclin E/CDK2 and G2/M checkpoint proteins
Small nuclear ribonucleoproteins are essential splicing factors. We previously identified the spliceosomal protein E (SmE) as a downstream effector of E2F1 in p53-deficient human carcinoma cells. Here, we investigated the biological relevance of SmE in determining the fate of cancer and non-tumourig...
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| Hoofdauteurs: | , |
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| Formaat: | Text |
| Taal: | Engels |
| Gepubliceerd in: |
John Wiley & Sons, Ltd
2008
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| Onderwerpen: | |
| Online toegang: | https://pmc.ncbi.nlm.nih.gov/articles/PMC4514120/ https://pubmed.ncbi.nlm.nih.gov/18208561 http://dx.doi.org/10.1111/j.1582-4934.2008.00244.x |
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