Evasion of alternative complement pathway by Trypanosoma cruzi results from inefficient binding of factor B.
During its differentiation in the insect vector to a stage infective for the mammalian host, Trypanosoma cruzi becomes resistant to lysis by the alternative pathway of complement. To elucidate the mechanism of complement evasion, we studied control of complement activation on the surface of the noni...
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| Main Authors: | , , , |
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| Format: | Text |
| Language: | English |
| Published: |
1986
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| Subjects: | |
| Online Access: | https://pmc.ncbi.nlm.nih.gov/articles/PMC386550/ https://pubmed.ncbi.nlm.nih.gov/2944112 |
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