Cyclic adenosine monophosphate acutely inhibits and chronically stimulates Na/H antiporter in OKP cells.
Parathyroid hormone, dopamine, alpha-adrenergic catecholamines, and angiotensin II regulate renal Na excretion, at least in part through modulation of acute cyclic (c)AMP-induced proximal tubule Na/H antiporter inhibition. The present studies examined the effect of chronic increases in cell cAMP on...
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Main Authors: | , , |
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Format: | Text |
Language: | English |
Published: |
1993
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Online Access: | https://pmc.ncbi.nlm.nih.gov/articles/PMC288321/ https://pubmed.ncbi.nlm.nih.gov/7691881 |
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