ATM phosphorylates the FATC domain of DNA-PKcs at threonine 4102 to promote non-homologous end joining
Ataxia-telangiectasia mutated (ATM) drives the DNA damage response via modulation of multiple signal transduction and DNA repair pathways. Previously, ATM activity was implicated in promoting the non-homologous end joining (NHEJ) pathway to repair a subset of DNA double-stranded breaks (DSBs), but h...
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| Автори: | , , , , , , , , |
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| Формат: | Artigo |
| Мова: | Англійська |
| Опубліковано: |
2023
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| Онлайн доступ: | https://doi.org/10.1093/nar/gkad505 https://academic.oup.com/nar/advance-article-pdf/doi/10.1093/nar/gkad505/50580076/gkad505.pdf |
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