Lack of resolution sensor drives age-related cardiometabolic and cardiorenal defects and impedes inflammation-resolution in heart failure
Recently, we observed that the specialized proresolving mediator (SPM) entity resolvin D1 activates lipoxin A4/formyl peptide receptor 2 (ALX/FPR2), which facilitates cardiac healing and persistent inflammation is a hallmark of impaired cardiac repair in aging. Splenic leukocyte-directed SPMs are es...
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| Главные авторы: | , , , , , , , , |
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| Формат: | Artigo |
| Язык: | английский |
| Опубликовано: |
2019
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| Online-ссылка: | https://doi.org/10.1016/j.molmet.2019.10.008 |
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