Oligomeric and fibrillar species of β-amyloid (Aβ42) both impair mitochondrial function in P301L tau transgenic mice

Oligomeric and fibrillar species of β-amyloid (Aβ42) both impair mitochondrial function in P301L tau transgenic mice

We recently provided evidence for a mitochondrial dysfunction in P301L tau transgenic mice, a strain modeling the tau pathology of Alzheimer's disease (AD) and frontotemporal dementia (FTD). In addition to tau aggregates, the AD brain is further characterized by A beta peptide-containing plaque...

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Detaylı Bibliyografya
Asıl Yazarlar: Anne Eckert, Susanne Hauptmann, Isabel Scherping, Jessica Meinhardt, Virginie Rhein, Stefan Dröse, Ulrich Brandt, Marcus Fändrich, Wernér E.G. Müller, Jürgen Götz
Materyal Türü: Artigo
Dil:İngilizce
Baskı/Yayın Bilgisi: 2008
Online Erişim:https://doi.org/10.1007/s00109-008-0391-6
https://link.springer.com/content/pdf/10.1007/s00109-008-0391-6.pdf
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Internet

https://doi.org/10.1007/s00109-008-0391-6
https://link.springer.com/content/pdf/10.1007/s00109-008-0391-6.pdf

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