Developmental delays consistent with cochlear hypothyroidism contribute to failure to develop hearing in mice lacking <i>Slc26a4</i>/pendrin expression
Mutations of SLC26A4 cause an enlarged vestibular aqueduct, nonsyndromic deafness, and deafness as part of Pendred syndrome. SLC26A4 encodes pendrin, an anion exchanger located in the cochlea, thyroid, and kidney. The goal of the present study was to determine whether developmental delays, possibly...
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| Main Authors: | , , , , , , , , , |
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| Formato: | Artigo |
| Idioma: | inglés |
| Publicado: |
2009
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| Acceso en liña: | https://doi.org/10.1152/ajprenal.00011.2009 |
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