Rezultati - Michael Arad

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  1. 1
    Phenotypic diversity in hypertrophic cardiomyopathy

    Phenotypic diversity in hypertrophic cardiomyopathy od Michael Arad

    Izdano 2002
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  2. 2
    Efficacy of exercise training in symptomatic patients with hypertrophic cardiomyopathy: Results of a structured exercise training program in a cardiac rehabilitation center

    Efficacy of exercise training in symptomatic patients with hypertrophic cardiomyopathy: Results of a structured exercise training program in a cardiac rehabilitation center od Robert Klempfner, Tamir Kamerman, Ehud Schwammenthal, Amira Nahshon, Ilan Hay, Ilan Goldenberg, Freimark Dov, Michael Arad

    Izdano 2013
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  3. 3
    Adipocyte-Specific Expression of PGC1α Promotes Adipocyte Browning and Alleviates Obesity-Induced Metabolic Dysfunction in an HO-1-Dependent Fashion

    Adipocyte-Specific Expression of PGC1α Promotes Adipocyte Browning and Alleviates Obesity-Induced Metabolic Dysfunction in an HO-1-Dependent Fashion od Hsin‐Hsueh Shen, Shailendra Pratap Singh, Marco Raffaele, Maayan Waldman, Edith Hochhauser, Juancarlos Ospino, Michael Arad, Stephen J. Peterson

    Izdano 2022
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  4. 4
    Angiogenic Imbalance and Residual Myocardial Injury in Recovered Peripartum Cardiomyopathy Patients

    Angiogenic Imbalance and Residual Myocardial Injury in Recovered Peripartum Cardiomyopathy Patients od Sorel Goland, Jean Marc Weinstein, Adi Zalik, Rafael Kuperstein, Liaz Zilberman, Sara Shimoni, Michael Arad, Tuvia Ben Gal, Jacob George

    Izdano 2016
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  5. 5
    Functional abnormalities in iPSC‐derived cardiomyocytes generated from CPVT1 and CPVT2 patients carrying ryanodine or calsequestrin mutations

    Functional abnormalities in iPSC‐derived cardiomyocytes generated from CPVT1 and CPVT2 patients carrying ryanodine or calsequestrin mutations od Atara Novak, Lili Barad, Avraham Lorber, Mihaela Gherghiceanu, Irina Reiter, Binyamin Eisen, Liron Eldor, Joseph Itskovitz‐Eldor, Michael Eldar, Michael Arad, Ofer Binah

    Izdano 2015
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  6. 6
    Sodium–glucose cotransporter 2 inhibitor Dapagliflozin attenuates diabetic cardiomyopathy

    Sodium–glucose cotransporter 2 inhibitor Dapagliflozin attenuates diabetic cardiomyopathy od Mohamad Arow, Maayan Waldman, Dor Yadin, Vadim Nudelman, Asher Shainberg, Nader G. Abraham, Dov Freimark, Ran Kornowski, Dan Aravot, Edith Hochhauser, Michael Arad

    Izdano 2020
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  7. 7
    Expression of the SARS-CoV-2 receptorACE2 in human heart is associated with uncontrolled diabetes, obesity, and activation of the renin angiotensin system

    Expression of the SARS-CoV-2 receptorACE2 in human heart is associated with uncontrolled diabetes, obesity, and activation of the renin angiotensin system od Michal Herman‐Edelstein, Tali Guetta, Amir Barnea, Maayan Waldman, Naomi Ben-Dor, Yaron D. Barac, Ran Kornowski, Michael Arad, Edith Hochhauser, Dan Aravot

    Izdano 2021
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  8. 8
    Regulation of diabetic cardiomyopathy by caloric restriction is mediated by intracellular signaling pathways involving ‘SIRT1 and PGC-1α’

    Regulation of diabetic cardiomyopathy by caloric restriction is mediated by intracellular signaling pathways involving ‘SIRT1 and PGC-1α’ od Maayan Waldman, Keren Cohen, Dor Yadin, Vadim Nudelman, Dan Gorfil, Michal L. Schwartzman, Ran Kornwoski, Dan Aravot, Nader G. Abraham, Michael Arad, Edith Hochhauser

    Izdano 2018
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  9. 9
    Calsequestrin 2 (CASQ2) mutations increase expression of calreticulin and ryanodine receptors, causing catecholaminergic polymorphic ventricular tachycardia

    Calsequestrin 2 (CASQ2) mutations increase expression of calreticulin and ryanodine receptors, causing catecholaminergic polymorphic ventricular tachycardia od Song Lei, Ronny Alcalai, Michael Arad, Cordula M. Wolf, Okan Toka, David A. Conner, Charles I. Berul, Michael Eldar, Christine E. Seidman, Jonathan G. Seidman

    Izdano 2007
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  10. 10
    Constitutively active AMP kinase mutations cause glycogen storage disease mimicking hypertrophic cardiomyopathy

    Constitutively active AMP kinase mutations cause glycogen storage disease mimicking hypertrophic cardiomyopathy od Michael Arad, D. Woodrow Benson, Antonio R. Pérez‐Atayde, William J. McKenna, Elizabeth Sparks, Ronald J. Kanter, Kate McGarry, Jonathan G. Seidman, Christine E. Seidman

    Izdano 2002
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  11. 11
    Constitutively active AMP kinase mutations cause glycogen storage disease mimicking hypertrophic cardiomyopathy

    Constitutively active AMP kinase mutations cause glycogen storage disease mimicking hypertrophic cardiomyopathy od Michael Arad, D. Woodrow Benson, Antonio R. Pérez‐Atayde, William J. McKenna, Elizabeth Sparks, Ronald J. Kanter, Kate McGarry, Jonathan G. Seidman, Christine E. Seidman

    Izdano 2002
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  12. 12
    EET intervention on Wnt1, NOV, and HO-1 signaling prevents obesity-induced cardiomyopathy in obese mice

    EET intervention on Wnt1, NOV, and HO-1 signaling prevents obesity-induced cardiomyopathy in obese mice od Jian Cao, Shailendra Pratap Singh, John A. McClung, Gregory Joseph, Luca Vanella, Ignazio Barbagallo, Houli Jiang, John R. Falck, Michael Arad, Joseph I. Shapiro, Nader G. Abraham

    Izdano 2017
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  13. 13
    The effect of enzyme replacement therapy on clinical outcomes in female patients with Fabry disease – A systematic literature review by a European panel of experts

    The effect of enzyme replacement therapy on clinical outcomes in female patients with Fabry disease – A systematic literature review by a European panel of experts od Dominique P. Germain, Michael Arad, Alessandro P. Burlina, Perry Elliott, Bruno Falissard, Ulla Feldt‐Rasmussen, Max J. Hilz, Derralynn Hughes, Alberto Ortíz, Christoph Wanner, Frank Weidemann, Marco Spada

    Izdano 2018
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  14. 14
    Reversibility of <i>PRKAG2</i> Glycogen-Storage Cardiomyopathy and Electrophysiological Manifestations

    Reversibility of <i>PRKAG2</i> Glycogen-Storage Cardiomyopathy and Electrophysiological Manifestations od Cordula M. Wolf, Michael Arad, Ferhaan Ahmad, Atsushi Sanbe, Scott Bernstein, Okan Toka, Tetsuo Konno, Gregory E. Morley, Jeffrey Robbins, J.G. Seidman, Christine E. Seidman, Charles I. Berul

    Izdano 2007
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  15. 15
    Gene Mutations in Apical Hypertrophic Cardiomyopathy

    Gene Mutations in Apical Hypertrophic Cardiomyopathy od Michael Arad, Manual Penas-Lado, Lorenzo Monserrat, Barry J. Maron, Mark V. Sherrid, Carolyn Y. Ho, Scott Barr, Ahmad Karim, Timothy M. Olson, Mitsohiro Kamisago, Jonathan G. Seidman, Christine E. Seidman

    Izdano 2005
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  16. 16
    Epoxyeicosatrienoic Acids Regulate Adipocyte Differentiation of Mouse 3T3 Cells, Via PGC-1α Activation, Which Is Required for HO-1 Expression and Increased Mitochondrial Function

    Epoxyeicosatrienoic Acids Regulate Adipocyte Differentiation of Mouse 3T3 Cells, Via PGC-1α Activation, Which Is Required for HO-1 Expression and Increased Mitochondrial Function od Maayan Waldman, Lars Bellner, Luca Vanella, Joseph Schragenheim, Komal Sodhi, Shailendra Pratap Singh, Dao‐Hong Lin, Anand Lakhkar, Jiangwei Li, Edith Hochhauser, Michael Arad, Zbigniew Darżynkiewicz, Attallah Kappas, Nader G. Abraham

    Izdano 2016
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  17. 17
    Glycogen Storage Diseases Presenting as Hypertrophic Cardiomyopathy

    Glycogen Storage Diseases Presenting as Hypertrophic Cardiomyopathy od Michael Arad, Barry J. Maron, Joshua M. Gorham, Walter H. Johnson, J. Philip Saul, Antonio R. Pérez‐Atayde, Paolo Spirito, Gregory B. Wright, Ronald J. Kanter, Christine E. Seidman, Jonathan G. Seidman

    Izdano 2005
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  18. 18
    Aberrant activation of AMP-activated protein kinase remodels metabolic network in favor of cardiac glycogen storage

    Aberrant activation of AMP-activated protein kinase remodels metabolic network in favor of cardiac glycogen storage od Ivan Luptak, Mei Shen, Huamei He, Michael F. Hirshman, Nicolas Musi, Laurie J. Goodyear, Jie Yan, Hiroko Wakimoto, Hiroyuki Morita, Michael Arad, Christine E. Seidman, Jonathan G. Seidman, Joanne S. Ingwall, James A. Balschi, Rong Tian

    Izdano 2007
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  19. 19
    Electrophysiological abnormalities in induced pluripotent stem cell-derived cardiomyocytes generated from Duchenne muscular dystrophy patients

    Electrophysiological abnormalities in induced pluripotent stem cell-derived cardiomyocytes generated from Duchenne muscular dystrophy patients od Binyamin Eisen, Ronen Ben Jehuda, Ashley J. Cuttitta, Lucy N. Mekies, Yuval Shemer, Polina Baskin, Irina Reiter, Lubna Willi, Dov Freimark, Mihaela Gherghiceanu, Lorenzo Monserrat, Michaela Scherr, Denise Hilfiker‐Kleiner, Michael Arad, Daniel E. Michele, Ofer Binah

    Izdano 2019
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  20. 20
    Genetic counselling and testing in cardiomyopathies: a position statement of the European Society of Cardiology Working Group on Myocardial and Pericardial Diseases

    Genetic counselling and testing in cardiomyopathies: a position statement of the European Society of Cardiology Working Group on Myocardial and Pericardial Diseases od Philippe Charron, Michael Arad, Eloisa Arbustini, Cristina Basso, Zofia T. Bilińska, Perry Elliott, Tiina Heliö, Andre Keren, William J. McKenna, Lorenzo Monserrat, Sabine Pankuweit, Andreas Perrot, Claudio Rapezzi, Arsen Ristić, Hubert Seggewiß, Irene M. van Langen, Luigi Tavazzi

    Izdano 2010
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