Kết quả tìm kiếm - Markus P. Kummer
- Đang hiển thị 1 - 20 kết quả của 30
- Chuyển đến trang tiếp theo
-
1
PPARs in Alzheimer′s Disease Bằng Markus P. Kummer, Michael T. Heneka
Được phát hành 2008Artigo -
2
Truncated and modified amyloid-beta species Bằng Markus P. Kummer, Michael T. Heneka
Được phát hành 2014Revisão -
3
-
4
Impact and Therapeutic Potential of PPARs in Alzheimers Disease Bằng Michael T. Heneka, Elisabet Reyes-Irisarri, Michael Hull, Markus P. Kummer
Được phát hành 2011Artigo -
5
PPARγ/RXRα-Induced and CD36-Mediated Microglial Amyloid-β Phagocytosis Results in Cognitive Improvement in Amyloid Precursor Protein/Presenilin 1 Mice Bằng Mitsugu Yamanaka, Taizo Ishikawa, Angelika Griep, Daisy Axt, Markus P. Kummer, Michael T. Heneka
Được phát hành 2012Artigo -
6
-
7
Aβ induces cell death by direct interaction with its cognate extracellular domain on APP (APP 597–624) Bằng Gideon M. Shaked, Markus P. Kummer, Daniel C. Lu, Verónica Galván, Dale E. Bredesen, Edward H. Koo, Gideon M. Shaked, Markus P. Kummer, Daniel C. Lu, Verónica Galván, Dale E. Bredesen, Edward H. Koo
Được phát hành 2006Artigo -
8
-
9
-
10
-
11
-
12
-
13
-
14
Mrp14 Deficiency Ameliorates Amyloid β Burden by Increasing Microglial Phagocytosis and Modulation of Amyloid Precursor Protein Processing Bằng Markus P. Kummer, Thomas Vogl, Daisy Axt, Angelika Griep, Ana Vieira‐Saecker, Frank Jessen, Ellen Gelpí, Johannes Roth, Michael T. Heneka
Được phát hành 2012Artigo -
15
Pan-PPAR Modulation Effectively Protects APP/PS1 Mice from Amyloid Deposition and Cognitive Deficits Bằng Markus P. Kummer, Rafael Schwarzenberger, Sakina Sayah‐Jeanne, Mathieu Dubernet, Robert Walczak, Dean W. Hum, Stephanie Schwartz, Daisy Axt, Michael T. Heneka
Được phát hành 2014Artigo -
16
Sepsis causes neuroinflammation and concomitant decrease of cerebral metabolism Bằng Alexander Semmler, Sven Hermann, Florian Mormann, Marc Weberpals, Stephan Paxian, Thorsten Okulla, Michael Schäfers, Markus P. Kummer, Thomas Klockgether, Michael T. Heneka
Được phát hành 2008Artigo -
17
Distinct and Non-Redundant Roles of Microglia and Myeloid Subsets in Mouse Models of Alzheimer's Disease Bằng Alexander Mildner, Bernhard Schlevogt, Katrin Kierdorf, Chotima Böttcher, Daniel Erny, Markus P. Kummer, Michael Quinn, Wolfgang Brück, Ingo Bechmann, Michael T. Heneka, Josef Priller, Marco Prinz
Được phát hành 2011Artigo -
18
Imaging microglial activation and glucose consumption in a mouse model of Alzheimer's disease Bằng Sara Rapic, Heiko Backes, Thomas Viel, Markus P. Kummer, Parisa Monfared, Bernd Neumaier, Stefan Vollmar, Mathias Hoehn, Annemie Van der Linden, Michael T. Heneka, Andreas H. Jacobs
Được phát hành 2012Artigo -
19
Locus ceruleus controls Alzheimer's disease pathology by modulating microglial functions through norepinephrine Bằng Michael T. Heneka, Fabian Nadrigny, Tommy Regen, Ana Martinez-Hernandez, Lucia Dumitrescu‐Ozimek, Dick Terwel, Daniel Jardanhazi‐Kurutz, Jochen Walter, Frank Kirchhoff, Uwe‐Karsten Hanisch, Markus P. Kummer
Được phát hành 2010Artigo -
20
Ear2 Deletion Causes Early Memory and Learning Deficits in APP/PS1 Mice Bằng Markus P. Kummer, Thomas Hammerschmidt, Alfredo Martı́nez, Dick Terwel, Gregor Eichele, Anika Witten, S. Figura, Monika Stoll, Stephanie Schwartz, Hans‐Christian Pape, Joachim L. Schultze, David Weinshenker, Michael T. Heneka
Được phát hành 2014Artigo
Công cụ tìm kiếm:
Các môn học liên quan
Medicine
Biology
Disease
Inflammation
Alzheimer's disease
Internal medicine
Immunology
Neuroscience
Microglia
Neuroinflammation
Pathology
Endocrinology
Biochemistry
Chemistry
Cell biology
Gene
Receptor
Amyloid precursor protein
Central nervous system
Neurodegeneration
Amyloid (mycology)
Cerebrospinal fluid
Dementia
Pathogenesis
Phagocytosis
Proinflammatory cytokine
Apolipoprotein E
Hippocampus
Psychology
Agonist