Ngā hua rapu - Markus P. Kummer

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  1. 1
    PPARs in Alzheimer′s Disease

    PPARs in Alzheimer′s Disease mā Markus P. Kummer, Michael T. Heneka

    I whakaputaina 2008
    Whiwhi kuputuhi katoa Whiwhi kuputuhi katoa
    Artigo
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  2. 2
    Truncated and modified amyloid-beta species

    Truncated and modified amyloid-beta species mā Markus P. Kummer, Michael T. Heneka

    I whakaputaina 2014
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  3. 3
    Body Fluid Cytokine Levels in Mild Cognitive Impairment and Alzheimer’s Disease: a Comparative Overview

    Body Fluid Cytokine Levels in Mild Cognitive Impairment and Alzheimer’s Disease: a Comparative Overview mā Frederic Brosseron, Marius Krauthausen, Markus P. Kummer, Michael T. Heneka

    I whakaputaina 2014
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  4. 4
    Impact and Therapeutic Potential of PPARs in Alzheimers Disease

    Impact and Therapeutic Potential of PPARs in Alzheimers Disease mā Michael T. Heneka, Elisabet Reyes-Irisarri, Michael Hull, Markus P. Kummer

    I whakaputaina 2011
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    Artigo
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  5. 5
    PPARγ/RXRα-Induced and CD36-Mediated Microglial Amyloid-β Phagocytosis Results in Cognitive Improvement in Amyloid Precursor Protein/Presenilin 1 Mice

    PPARγ/RXRα-Induced and CD36-Mediated Microglial Amyloid-β Phagocytosis Results in Cognitive Improvement in Amyloid Precursor Protein/Presenilin 1 Mice mā Mitsugu Yamanaka, Taizo Ishikawa, Angelika Griep, Daisy Axt, Markus P. Kummer, Michael T. Heneka

    I whakaputaina 2012
    Whiwhi kuputuhi katoa Whiwhi kuputuhi katoa
    Artigo
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  6. 6
    Formation of Pmel17 Amyloid Is Regulated by Juxtamembrane Metalloproteinase Cleavage, and the Resulting C-terminal Fragment Is a Substrate for γ-Secretase

    Formation of Pmel17 Amyloid Is Regulated by Juxtamembrane Metalloproteinase Cleavage, and the Resulting C-terminal Fragment Is a Substrate for γ-Secretase mā Markus P. Kummer, Hiroko Maruyama, Claudia Huelsmann, Sandra Baches, Sascha Weggen, Edward H. Koo

    I whakaputaina 2008
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    Artigo
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  7. 7
    Aβ induces cell death by direct interaction with its cognate extracellular domain on APP (APP 597–624)

    Aβ induces cell death by direct interaction with its cognate extracellular domain on APP (APP 597–624) mā Gideon M. Shaked, Markus P. Kummer, Daniel C. Lu, Verónica Galván, Dale E. Bredesen, Edward H. Koo, Gideon M. Shaked, Markus P. Kummer, Daniel C. Lu, Verónica Galván, Dale E. Bredesen, Edward H. Koo

    I whakaputaina 2006
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    Artigo
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  8. 8
    Distinct modulation of microglial amyloid β phagocytosis and migration by neuropeptidesi

    Distinct modulation of microglial amyloid β phagocytosis and migration by neuropeptidesi mā Sigal Fleisher‐Berkovich, Talia Filipovich‐Rimon, Sarit Ben-Shmuel, Claudia Hülsmann, Markus P. Kummer, Michael T. Heneka

    I whakaputaina 2010
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    Artigo
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  9. 9
    Characterization and clinical use of inflammatory cerebrospinal fluid protein markers in Alzheimer’s disease

    Characterization and clinical use of inflammatory cerebrospinal fluid protein markers in Alzheimer’s disease mā Frederic Brosseron, Andreas Traschütz, Catherine Widmann, Markus P. Kummer, Paweł Tacik, Francesco Santarelli, Frank Jessen, Michael T. Heneka

    I whakaputaina 2018
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    Artigo
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  10. 10
    Targeting norepinephrine in mild cognitive impairment and Alzheimer's disease

    Targeting norepinephrine in mild cognitive impairment and Alzheimer's disease mā Termpanit Chalermpalanupap, Becky Kinkead, William T. Hu, Markus P. Kummer, Thea Hammerschmidt, Michael T. Heneka, David Weinshenker, Allan I. Levey

    I whakaputaina 2013
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    Revisão
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  11. 11
    CXCR3 promotes plaque formation and behavioral deficits in an Alzheimer’s disease model

    CXCR3 promotes plaque formation and behavioral deficits in an Alzheimer’s disease model mā Marius Krauthausen, Markus P. Kummer, Julian Zimmermann, Elisabet Reyes-Irisarri, Dick Terwel, Bruno Bulic, Michael T. Heneka, Marcus Müller

    I whakaputaina 2014
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    Artigo
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  12. 12
    <i>NOS2</i>Gene Deficiency Protects from Sepsis-Induced Long-Term Cognitive Deficits

    <i>NOS2</i>Gene Deficiency Protects from Sepsis-Induced Long-Term Cognitive Deficits mā Marc Weberpals, Michael Hermes, Sven Hermann, Markus P. Kummer, Dick Terwel, Alexander Semmler, Meike Berger, Michael Schäfers, Michael T. Heneka

    I whakaputaina 2009
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    Artigo
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  13. 13
    Critical Role of Astroglial Apolipoprotein E and Liver X Receptor-α Expression for Microglial Aβ Phagocytosis

    Critical Role of Astroglial Apolipoprotein E and Liver X Receptor-α Expression for Microglial Aβ Phagocytosis mā Dick Terwel, Knut R. Steffensen, Philip B. Verghese, Markus P. Kummer, Jan-Ακε Gustafsson, David M. Holtzman, Michael T. Heneka

    I whakaputaina 2011
    Whiwhi kuputuhi katoa Whiwhi kuputuhi katoa
    Artigo
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  14. 14
    Mrp14 Deficiency Ameliorates Amyloid β Burden by Increasing Microglial Phagocytosis and Modulation of Amyloid Precursor Protein Processing

    Mrp14 Deficiency Ameliorates Amyloid β Burden by Increasing Microglial Phagocytosis and Modulation of Amyloid Precursor Protein Processing mā Markus P. Kummer, Thomas Vogl, Daisy Axt, Angelika Griep, Ana Vieira‐Saecker, Frank Jessen, Ellen Gelpí, Johannes Roth, Michael T. Heneka

    I whakaputaina 2012
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    Artigo
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  15. 15
    Pan-PPAR Modulation Effectively Protects APP/PS1 Mice from Amyloid Deposition and Cognitive Deficits

    Pan-PPAR Modulation Effectively Protects APP/PS1 Mice from Amyloid Deposition and Cognitive Deficits mā Markus P. Kummer, Rafael Schwarzenberger, Sakina Sayah‐Jeanne, Mathieu Dubernet, Robert Walczak, Dean W. Hum, Stephanie Schwartz, Daisy Axt, Michael T. Heneka

    I whakaputaina 2014
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    Artigo
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  16. 16
    Sepsis causes neuroinflammation and concomitant decrease of cerebral metabolism

    Sepsis causes neuroinflammation and concomitant decrease of cerebral metabolism mā Alexander Semmler, Sven Hermann, Florian Mormann, Marc Weberpals, Stephan Paxian, Thorsten Okulla, Michael Schäfers, Markus P. Kummer, Thomas Klockgether, Michael T. Heneka

    I whakaputaina 2008
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    Artigo
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  17. 17
    Distinct and Non-Redundant Roles of Microglia and Myeloid Subsets in Mouse Models of Alzheimer's Disease

    Distinct and Non-Redundant Roles of Microglia and Myeloid Subsets in Mouse Models of Alzheimer's Disease mā Alexander Mildner, Bernhard Schlevogt, Katrin Kierdorf, Chotima Böttcher, Daniel Erny, Markus P. Kummer, Michael Quinn, Wolfgang Brück, Ingo Bechmann, Michael T. Heneka, Josef Priller, Marco Prinz

    I whakaputaina 2011
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    Artigo
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  18. 18
    Imaging microglial activation and glucose consumption in a mouse model of Alzheimer's disease

    Imaging microglial activation and glucose consumption in a mouse model of Alzheimer's disease mā Sara Rapic, Heiko Backes, Thomas Viel, Markus P. Kummer, Parisa Monfared, Bernd Neumaier, Stefan Vollmar, Mathias Hoehn, Annemie Van der Linden, Michael T. Heneka, Andreas H. Jacobs

    I whakaputaina 2012
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    Artigo
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  19. 19
    Locus ceruleus controls Alzheimer's disease pathology by modulating microglial functions through norepinephrine

    Locus ceruleus controls Alzheimer's disease pathology by modulating microglial functions through norepinephrine mā Michael T. Heneka, Fabian Nadrigny, Tommy Regen, Ana Martinez-Hernandez, Lucia Dumitrescu‐Ozimek, Dick Terwel, Daniel Jardanhazi‐Kurutz, Jochen Walter, Frank Kirchhoff, Uwe‐Karsten Hanisch, Markus P. Kummer

    I whakaputaina 2010
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    Artigo
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  20. 20
    Ear2 Deletion Causes Early Memory and Learning Deficits in APP/PS1 Mice

    Ear2 Deletion Causes Early Memory and Learning Deficits in APP/PS1 Mice mā Markus P. Kummer, Thomas Hammerschmidt, Alfredo Martı́nez, Dick Terwel, Gregor Eichele, Anika Witten, S. Figura, Monika Stoll, Stephanie Schwartz, Hans‐Christian Pape, Joachim L. Schultze, David Weinshenker, Michael T. Heneka

    I whakaputaina 2014
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    Artigo
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