Rezultati - Basavaraj Hooli
- Showing 1 - 9 results of 9
-
1
-
2
-
3
Gain-of-function mutations in protein kinase Cα (PKCα) may promote synaptic defects in Alzheimer’s disease od Stephanie Alfonso, Julia A. Callender, Basavaraj Hooli, Corina E. Antal, Kristina Mullin, Mathew A. Sherman, Sylvain Lesné, Michael Leitges, Alexandra C. Newton, Rudolph E. Tanzi, Roberto Malinow
Izdano 2016Artigo -
4
Assessment of Alzheimer’s disease case–control associations using family-based methods od Brit‐Maren M. Schjeide, Matthew B. McQueen, Kristina Mullin, Jason DiVito, Meghan F. Hogan, Michele Parkinson, Basavaraj Hooli, Christoph Lange, Deborah Blacker, Rudolph E. Tanzi, Lars Bertram
Izdano 2008Artigo -
5
Potential late-onset Alzheimer's disease-associated mutations in the ADAM10 gene attenuate α-secretase activity od Minji Kim, Jaehong Suh, Donna Romano, Mimy H. Truong, Kristina Mullin, Basavaraj Hooli, D. P. Norton, Giuseppina Tesco, Kathryn Elliott, Steven L. Wagner, Robert D. Moir, Kathrin Becker, Rudolph E. Tanzi
Izdano 2009Artigo -
6
Loss of Ataxin-1 Potentiates Alzheimer’s Pathogenesis by Elevating Cerebral BACE1 Transcription od Jaehong Suh, Donna Romano, Larissa Nitschke, Scott P. Herrick, Britt A. DiMarzio, Volodymyr Dzhala, Jun-Seok Bae, Mary K. Oram, Yuejiao Zheng, Basavaraj Hooli, Kristina Mullin, Vincenzo A. Gennarino, Wilma Wasco, Jeremy D. Schmahmann, Mark W. Albers, Huda Y. Zoghbi, Rudolph E. Tanzi
Izdano 2019Artigo -
7
A three-dimensional human neural cell culture model of Alzheimer’s disease od Se Hoon Choi, Young Hye Kim, Matthias Hebisch, Christopher Sliwinski, Seungkyu Lee, Carla D’Avanzo, Hechao Chen, Basavaraj Hooli, Caroline Asselin, Julien Muffat, Justin Klee, Can Zhang, Brian J. Wainger, Michael Peitz, Dora M. Kovacs, Clifford J. Woolf, Steven L. Wagner, Rudolph E. Tanzi, Doo Yeon Kim
Izdano 2014Artigo -
8
Genome-wide Association Analysis Reveals Putative Alzheimer's Disease Susceptibility Loci in Addition to APOE od Lars Bertram, Christoph Lange, Kristina Mullin, Michele Parkinson, Monica Hsiao, Meghan F. Hogan, Brit M.M. Schjeide, Basavaraj Hooli, Jason DiVito, Iuliana Ionita, Hongyu Jiang, Nan M. Laird, Thomas Moscarillo, Kari L. Ohlsen, Kathryn Elliott, Xin Wang, Diane Hu‐Lince, Marie Ryder, Amy Murphy, Steven L. Wagner, Deborah Blacker, Kathrin Becker, Rudolph E. Tanzi
Izdano 2008Artigo -
9
Genome-wide association identifies the first risk loci for psychosis in Alzheimer disease od Mary Ann A. DeMichele‐Sweet, Lambertus Klei, Byron Creese, Janet Harwood, Elise A. Weamer, Lora McClain, Rebecca Sims, Isabel Hernández, Sonia Moreno‐Grau, Lluís Tárraga, Merçé Boada, Emilio Alarcón‐Martín, Sergi Valero, Yushi Liu, Basavaraj Hooli, Dag Aarsland, Geir Selbæk, Sverre Bergh, Arvid Rongve, Ingvild Saltvedt, Håvard Kjesbu Skjellegrind, Bo Engdahl, Eystein Stordal, Ole A. Andreassen, Srdjan Djurovic, Lavinia Athanasiu, Davide Seripa, Barbara Borroni, Diego Albani, Gianluigi Forloni, Patrizia Mecocci, Alessandro Serretti, Diana De Ronchi, Antonis Politis, Julie Williams, Richard Mayeux, Tatiana Foroud, Agustı́n Ruiz, Clive Ballard, Peter Holmans, Oscar L. López, M. Ilyas Kamboh, Bernie Devlin, Robert A. Sweet
Izdano 2021Revisão
Iskalna orodja:
Sorodne teme
Biology
Disease
Medicine
Genetics
Internal medicine
Alzheimer's disease
Gene
Neuroscience
Cell biology
Amyloid precursor protein
Genetic association
Genome-wide association study
Genotype
Single-nucleotide polymorphism
Association (psychology)
Chemistry
Dementia
Immunology
Mutation
Neurodegeneration
Pathology
Phenotype
Psychology
Psychotherapist
ADAM10
Allele
Amyloid (mycology)
Amyloid beta
Amyloid precursor protein secretase
Antibody