Curcumin regulates pulmonary extracellular matrix remodeling and mitochondrial function to attenuate pulmonary fibrosis by regulating the miR-29a-3p/DNMT3A axis

Epigenetic regulation and mitochondrial dysfunction are essential to the progression of idiopathic pulmonary fibrosis (IPF). Curcumin (CCM) in inhibits the progression of pulmonary fibrosis by regulating the expression of specific miRNAs and pulmonary fibroblast mitochondrial function; however, the...

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Hlavní autoři: Meng‐Hsuan Cheng, Hsuan‐Fu Kuo, Chia-Yuan Chang, Jui‐Chi Chang, I-Hshiang Liu, Chong‐Chao Hsieh, Chih‐Hsin Hsu, Chia‐Yang Li, Shu‐Chi Wang, Yung‐Hsiang Chen, Chuang‐Rung Chang, Tsung‐Ying Lee, Yuru Liu, Chi-Yuan Huang, Szu-Hui Wu, Wei‐Lun Liu, Po‐Len Liu
Médium: Artigo
Jazyk:angličtina
Vydáno: 2024
On-line přístup:https://doi.org/10.1016/j.biopha.2024.116572
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