ω-3 DPA Protected Neurons from Neuroinflammation by Balancing Microglia M1/M2 Polarizations through Inhibiting NF-κB/MAPK p38 Signaling and Activating Neuron-BDNF-PI3K/AKT Pathways

Microglia M1 phenotype causes HPA axis hyperactivity, neurotransmitter dysfunction, and production of proinflammatory mediators and oxidants, which may contribute to the etiology of depression and neurodegenerative diseases. Eicosapentaenoic acid (EPA) may counteract neuroinflammation by increasing...

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Príomhchruthaitheoirí: Baiping Liu, Yongping Zhang, Zhiyou Yang, Meijun Liu, Cai Zhang, Yun‐Tao Zhao, Cai Song
Formáid: Artigo
Teanga:Béarla
Foilsithe / Cruthaithe: 2021
Rochtain ar líne:https://doi.org/10.3390/md19110587
https://www.mdpi.com/1660-3397/19/11/587/pdf?version=1634813299
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