Multiple autophosphorylation sites are dispensable for murine ATM activation in vivo

Cellular responses to both physiological and pathological DNA double-strand breaks are initiated through activation of the evolutionarily conserved ataxia telangiectasia mutated (ATM) kinase. Upon DNA damage, an activation mechanism involving autophosphorylation has been reported to allow ATM to pho...

Бүрэн тодорхойлолт

-д хадгалсан:
Номзүйн дэлгэрэнгүй
Үндсэн зохиолчид: J. Daniel, Manuela Pellegrini, Ji‐Hoon Lee, Tanya T. Paull, Lionel Feigenbaum, André Nussenzweig
Формат: Artigo
Хэл сонгох:англи
Хэвлэсэн: 2008
Онлайн хандалт:https://doi.org/10.1083/jcb.200805154
https://rupress.org/jcb/article-pdf/183/5/777/1339781/jcb_200805154.pdf
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