Multiple autophosphorylation sites are dispensable for murine ATM activation in vivo

Cellular responses to both physiological and pathological DNA double-strand breaks are initiated through activation of the evolutionarily conserved ataxia telangiectasia mutated (ATM) kinase. Upon DNA damage, an activation mechanism involving autophosphorylation has been reported to allow ATM to pho...

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Hlavní autoři: J. Daniel, Manuela Pellegrini, Ji‐Hoon Lee, Tanya T. Paull, Lionel Feigenbaum, André Nussenzweig
Médium: Artigo
Jazyk:angličtina
Vydáno: 2008
On-line přístup:https://doi.org/10.1083/jcb.200805154
https://rupress.org/jcb/article-pdf/183/5/777/1339781/jcb_200805154.pdf
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