Oxygen radical production and thiol depletion are required for Ca(2+)-mediated endogenous endonuclease activation in apoptotic thymocytes.
Glucocorticoid hormones stimulate apoptosis in thymocytes via a mechanism that involves changes in intracellular Ca2+, and exogenous Ca2+ can also directly promote the nuclear alterations of apoptosis (lamin degradation and chromatin cleavage) in isolated nuclei. Here we report that glucocorticoid t...
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| Hlavní autoři: | , , , |
|---|---|
| Médium: | Artigo |
| Jazyk: | angličtina |
| Vydáno: |
1995
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| On-line přístup: | https://doi.org/10.4049/jimmunol.155.11.5133 https://journals.aai.org/jimmunol/article-pdf/155/11/5133/1068224/5133.pdf |
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