Fanconi anemia group J mutation abolishes its DNA repair function by uncoupling DNA translocation from helicase activity or disruption of protein-DNA complexes

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Main Authors: Yuliang Wu, Joshua A. Sommers, Avvaru N. Suhasini, Thomas A. Leonard, Julianna S. Deakyne, Alexander V. Mazin, Kazuo Shin‐ya, Hiroyuki Kitao, Robert Brosh
格式: Artigo
语言:英语
出版: 2010
在线阅读:https://doi.org/10.1182/blood-2009-11-256016
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