Efeitos da intensidade do exercício e da sobrecarga de creatina na hipotensão pós-exercício resistido

Postexercise hypotension plays an important role in the non-pharmacological treat-ment of hypertension and is characterized by a decrease in blood pressure after a single exercise bout in relation to pre-exercise levels. This study investigated the effects of exercise intensity and creatine monohydr...

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Autors principals: Moreno Rodrigues Moreno, Gisela Arsa, Pedro Luiz Garcia Braga, Juliana Hott de Fúcio Lizardo, Carmen Sílvia Grubert Campbell, Mara Lucy Dompietro Ruiz Denadai, Herbert Gustavo Simões
Format: Artigo
Idioma:anglès
Publicat: 2011
Accés en línia:https://doi.org/10.5007/1980-0037.2009v11n4p373
https://periodicos.ufsc.br/index.php/rbcdh/article/download/1980-0037.2009v11n4p373/10918
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Sumari:Postexercise hypotension plays an important role in the non-pharmacological treat-ment of hypertension and is characterized by a decrease in blood pressure after a single exercise bout in relation to pre-exercise levels. This study investigated the effects of exercise intensity and creatine monohydrate supplementation on postexercise hypotension, as well as the possible role of blood lactate in this response. Ten normotensive subjects underwent resistance exercise sessions before (BC) and after (AC) creatine supplementation: 1) muscle endurance (ME) consisting of 30 repetitions at 30% of one-repetition maximum; 2) hypertrophy (HP) consisting of 8 repetitions at 75% of one-repetition maximum. Blood pressure was measured before and after the exercise bout. Blood lactate was measured after the exercise bout. The HP and ME sessions promoted a decrease in systolic blood pressure (∆ -19 ± 1.0 mmHg; ∆ -15 ± 0.9 mmHg, respectively, P< 0.05), which was attenuated after creatine supplementation (∆ -7.1 ± 1.0 mmHg; ∆ -11 ± 1.0 mmHg, respectively, P<0.05). Peak blood lactate was attenuated after creatine supplementation in the HP session (BC: 19 ± 0.4 mM; AC: 15 ± 0.4 mM, P< 0.05) and remained unchanged after creatine supplementation in the ME session (BC: 16 ± 0.8 mM; AC: 14 ± 0.4 mM, P> 0.05). In conclusion, resistance exercise intensity did not influence postexercise hypotension. Creatine supplementation attenuated the decrease in blood pressure after resistance exercise. The results suggest the involvement of blood lactate in post-resistance exercise hypotension.