Resultados de procura - Zhuang, Aowen

Globally elevating the AGE clearance receptor, OST48, does not protect against the development of diabetic kidney disease, despite improving insulin secretion por Zhuang, Aowen, Yap, Felicia Y. T., McCarthy, Domenica, Leung, Chris, Sourris, Karly C., Penfold, Sally A., Thallas-Bonke, Vicki, Coughlan, Melinda T., Schulz, Benjamin L., Forbes, Josephine M.

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The AGE receptor, OST48 drives podocyte foot process effacement and basement membrane expansion (alters structural composition) por Zhuang, Aowen, Yap, Felicia Y. T., Borg, Danielle J., McCarthy, Domenica, Fotheringham, Amelia, Leung, Sherman, Penfold, Sally A., Sourris, Karly C., Coughlan, Melinda T., Schulz, Benjamin L., Forbes, Josephine M.

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Perinatal exposure to high dietary advanced glycation end products in transgenic NOD8.3 mice leads to pancreatic beta cell dysfunction por Borg, Danielle J., Yap, Felicia Y. T., Keshvari, Sahar, Simmons, David G., Gallo, Linda A., Fotheringham, Amelia K., Zhuang, Aowen, Slattery, Robyn M., Hasnain, Sumaira Z., Coughlan, Melinda T., Kantharidis, Phillip, Forbes, Josephine M.

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SOD2 in skeletal muscle: New insights from an inducible deletion model por Zhuang, Aowen, Yang, Christine, Liu, Yingying, Tan, Yanie, Bond, Simon T., Walker, Shannen, Sikora, Tim, Laskowski, Adrienne, Sharma, Arpeeta, de Haan, Judy B., Meikle, Peter J., Shimizu, Takahiko, Coughlan, Melinda T., Calkin, Anna C., Drew, Brian G.

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Once daily administration of the SGLT2 inhibitor, empagliflozin, attenuates markers of renal fibrosis without improving albuminuria in diabetic db/db mice por Gallo, Linda A., Ward, Micheal S., Fotheringham, Amelia K., Zhuang, Aowen, Borg, Danielle J., Flemming, Nicole B., Harvie, Ben M., Kinneally, Toni L., Yeh, Shang-Ming, McCarthy, Domenica A., Koepsell, Hermann, Vallon, Volker, Pollock, Carol, Panchapakesan, Usha, Forbes, Josephine M.

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Erratum: Once daily administration of the SGLT2 inhibitor, empagliflozin, attenuates markers of renal fibrosis without improving albuminuria in diabetic db/db mice por Gallo, Linda A., Ward, Micheal S., Fotheringham, Amelia K., Zhuang, Aowen, Borg, Danielle J., Flemming, Nicole B., Harvie, Ben M., Kinneally, Toni L., Yeh, Shang-Ming, McCarthy, Domenica A., Koepsell, Hermann, Vallon, Volker, Pollock, Carol, Panchapakesan, Usha, Forbes, Josephine M.

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Increased liver AGEs induce hepatic injury mediated through an OST48 pathway por Zhuang, Aowen, Yap, Felicia YT., Bruce, Clinton, Leung, Chris, Plan, Manuel R., Sullivan, Mitchell A., Herath, Chandana, McCarthy, Domenica, Sourris, Karly C., Kantharidis, Phillip, Coughlan, Melinda T., Febbraio, Mark A., Hodson, Mark P., Watt, Matthew J., Angus, Peter, Schulz, Benjamin L., Forbes, Josephine M.

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Targeted mitochondrial therapy using MitoQ shows equivalent renoprotection to angiotensin converting enzyme inhibition but no combined synergy in diabetes por Ward, Micheal S., Flemming, Nicole B., Gallo, Linda A., Fotheringham, Amelia K., McCarthy, Domenica A., Zhuang, Aowen, Tang, Peter H., Borg, Danielle J., Shaw, Hannah, Harvie, Benjamin, Briskey, David R., Roberts, Llion A., Plan, Manuel R., Murphy, Michael P., Hodson, Mark P., Forbes, Josephine M.

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Tissue-specific expression of Cas9 has no impact on whole-body metabolism in four transgenic mouse lines por Bond, Simon T., Zhuang, Aowen, Yang, Christine, Gould, Eleanor A.M., Sikora, Tim, Liu, Yingying, Fu, Ying, Watt, Kevin I., Tan, Yanie, Kiriazis, Helen, Lancaster, Graeme I., Gregorevic, Paul, Henstridge, Darren C., McMullen, Julie R., Meikle, Peter J., Calkin, Anna C., Drew, Brian G.

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Loss of the long non-coding RNA OIP5-AS1 exacerbates heart failure in a sex-specific manner por Zhuang, Aowen, Calkin, Anna C., Lau, Shannen, Kiriazis, Helen, Donner, Daniel G., Liu, Yingying, Bond, Simon T., Moody, Sarah C., Gould, Eleanor A.M., Colgan, Timothy D., Carmona, Sergio Ruiz, Inouye, Michael, de Aguiar Vallim, Thomas Q., Tarling, Elizabeth J., Quaife-Ryan, Gregory A., Hudson, James E., Porrello, Enzo R., Gregorevic, Paul, Gao, Xiao-Ming, Du, Xiao-Jun, McMullen, Julie R., Drew, Brian G.

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