نتائج البحث - Yutaka Harita

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  1. 1
    Neph1, a Component of the Kidney Slit Diaphragm, Is Tyrosine-phosphorylated by the Src Family Tyrosine Kinase and Modulates Intracellular Signaling by Binding to Grb2

    Neph1, a Component of the Kidney Slit Diaphragm, Is Tyrosine-phosphorylated by the Src Family Tyrosine Kinase and Modulates Intracellular Signaling by Binding to Grb2 حسب Yutaka Harita, Hidetake Kurihara, Hidetaka Kosako, Tohru Tezuka, Takashi Sekine, Takashi Igarashi, Seisuke Hattori

    منشور في 2008
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  2. 2
    Tyrosine phosphorylation–dependent activation of TRPC6 regulated by PLC-γ1 and nephrin: effect of mutations associated with focal segmental glomerulosclerosis

    Tyrosine phosphorylation–dependent activation of TRPC6 regulated by PLC-γ1 and nephrin: effect of mutations associated with focal segmental glomerulosclerosis حسب Shoichiro Kanda, Yutaka Harita, Yoshio Shibagaki, Takashi Sekine, Takashi Igarashi, Takafumi Inoue, Seisuke Hattori

    منشور في 2011
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    Artigo
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  3. 3
    ASPP2 Regulates Epithelial Cell Polarity through the PAR Complex

    ASPP2 Regulates Epithelial Cell Polarity through the PAR Complex حسب Wei-Li Cong, Tomonori Hirose, Yutaka Harita, Akio Yamashita, Keiko Mizuno, Hisashi Hirano, Shigeo Ohno

    منشور في 2010
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  4. 4
    Phosphorylation of Nephrin Triggers Ca2+ Signaling by Recruitment and Activation of Phospholipase C-γ1

    Phosphorylation of Nephrin Triggers Ca2+ Signaling by Recruitment and Activation of Phospholipase C-γ1 حسب Yutaka Harita, Hidetake Kurihara, Hidetaka Kosako, Tohru Tezuka, Takashi Sekine, Takashi Igarashi, Ikuroh Ohsawa, Shigeo Ohta, Seisuke Hattori

    منشور في 2009
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    Artigo
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  5. 5
    Treatment of Nephrotic Syndrome with Anti-Nephrin Antibodies Using Plasmapheresis, Rituximab, and Mycophenolate Mofetil

    Treatment of Nephrotic Syndrome with Anti-Nephrin Antibodies Using Plasmapheresis, Rituximab, and Mycophenolate Mofetil حسب Keiichi Takizawa, Yoko Shirai, Yuko Kajiho, Shoichiro Kanda, Motohiro Kato, Hiroyuki Abé, Kenichiro Miura, Motoshi Hattori, Yutaka Harita

    منشور في 2025
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    Artigo
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  6. 6
    LMX1B mutation with residual transcriptional activity as a cause of isolated glomerulopathy

    LMX1B mutation with residual transcriptional activity as a cause of isolated glomerulopathy حسب Tsuyoshi Isojima, Yutaka Harita, Masayuki Furuyama, Noriko Sugawara, Kiyonobu Ishizuka, Shigeru Horita, Yuko Kajiho, Kenichiro Miura, Takashi Igarashi, Motoshi Hattori, Sachiko Kitanaka

    منشور في 2013
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    Artigo
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  7. 7
    Patients with Epstein–Fechtner syndromes owing to MYH9 R702 mutations develop progressive proteinuric renal disease

    Patients with Epstein–Fechtner syndromes owing to MYH9 R702 mutations develop progressive proteinuric renal disease حسب Takashi Sekine, Mutsuko Konno, Satoshi Sasaki, Suzuko Moritani, Takuma Miura, Wai-shan Wong, Hisanori Nishio, Toshihiro Nishiguchi, Miyako Yoshinari Ohuchi, Shigeru Tsuchiya, Takeshi Matsuyama, Hirokazu Kanegane, Komei Ida, Kenichiro Miura, Yutaka Harita, Motoshi Hattori, Shigeru Horita, Takashi Igarashi, Hidehiko Saito, Shinji Kunishima

    منشور في 2010
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    Artigo
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  8. 8
    Development of an exon skipping therapy for X-linked Alport syndrome with truncating variants in COL4A5

    Development of an exon skipping therapy for X-linked Alport syndrome with truncating variants in COL4A5 حسب Tomohiko Yamamura, Tomoko Horinouchi, Tomomi Adachi, Maki Terakawa, Yutaka Takaoka, Kohei Omachi, Minoru Takasato, Kiyosumi Takaishi, Takao Shoji, Yoshiyuki Onishi, Yoshito Kanazawa, Makoto Koizumi, Yasuko Tomono, Aki Sugano, Akemi Shono, Shogo Minamikawa, China Nagano, Nana Sakakibara, Shinya Ishiko, Yuya Aoto, Misato Kamura, Yutaka Harita, Kenichiro Miura, Shoichiro Kanda, Naoya Morisada, Rini Rossanti, Ming Juan Ye, Yoshimi Nozu, Masafumi Matsuo, Hirofumi Kai, Kazumoto Iijima, Kandai Nozu

    منشور في 2020
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    Artigo
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  9. 9
    Common risk variants in NPHS1 and TNFSF15 are associated with childhood steroid-sensitive nephrotic syndrome

    Common risk variants in NPHS1 and TNFSF15 are associated with childhood steroid-sensitive nephrotic syndrome حسب Xiaoyuan Jia, Tomohiko Yamamura, Rasheed Gbadegesin, Michelle Mcnulty, Kyuyong Song, China Nagano, Yuki Hitomi, Dongwon Lee, Yoshihiro Aiba, Seik‐Soon Khor, Kazuko Ueno, Yosuke Kawai, Masao Nagasaki, Eisei Noiri, Tomoko Horinouchi, Hiroshi Kaito, Riku Hamada, Takayuki Okamoto, Koichi Kamei, Yoshitsugu Kaku, Rika Fujimaru, Ryojiro Tanaka, Yuko Shima, Jiwon Baek, Hee Gyung Kang, Il Soo Ha, Kyoung Hee Han, Eun Mi Yang, Asiri Abeyagunawardena, Brandon M. Lane, Megan Chryst-Stangl, Christopher Imokhuede Esezobor, Adaobi Solarin, Claire Dossier, Georges Deschênes, Marina Vivarelli, Hanna Dębiec, Kenji Ishikura, Masafumi Matsuo, Kandai Nozu, Pierre Ronco, Hae Il Cheong, Matthew G. Sampson, Katsushi Tokunaga, Kazumoto Iijima, Yoshinori Araki, Yoshinobu Nagaoka, Takayuki Okamoto, Yasuyuki Sato, Asako Hayashi, Toshiyuki Takahashi, Hayato Aoyagi, Michihiko Ueno, Masanori Nakanishi, Nariaki Toita, Kimiaki Uetake, Norio Kobayashi, Shoji Fujita, Kazushi Tsuruga, Naonori Kumagai, Hiroki Kudo, Eriko Tanaka, Tae Omori, Mari Okada, Yoshiho Hatai, Tomohiro Udagawa, Yaeko Motoyoshi, Kenji Ishikura, Koichi Kamei, Masao Ogura, Mai Sato, Yuji Kano, Motoshi Hattori, Kenichiro Miura, Yutaka Harita, Shoichiro Kanda, Emi Sawanobori, Anna Kobayashi, Manabu Kojika, Yoko Ohwada, Kunimasa Yan, Hiroshi Hataya, Riku Hamada, Chikako Terano, Ryoko Harada, Yuko Hamasaki, Junya Hashimoto, Shuichi Ito, Hiroyuki Machida, Aya Inaba, Takeshi Matsuyama, Miwa Goto, Masaki Shimizu, Kazuhide Ohta, Yohei Ikezumi, Takeshi Yamada, Toshiaki Suzuki, Soichi Tamamura, Yukiko Mori, Yoshihiko Hidaka

    منشور في 2020
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    Artigo
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