检索结果 - Wei, Xiaomu

Mob as tumor suppressor is activated by Hippo kinase for growth inhibition in Drosophila 由 Wei, Xiaomu, Shimizu, Takeshi, Lai, Zhi-Chun

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Three-dimensional reconstruction of protein networks provides insight into human genetic disease 由 Wang, Xiujuan, Wei, Xiaomu, Thijssen, Bram, Das, Jishnu, Lipkin, Steven M, Yu, Haiyuan

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Dissecting Disease Inheritance Modes in a Three-Dimensional Protein Network Challenges the “Guilt-by-Association” Principle 由 Guo, Yu, Wei, Xiaomu, Das, Jishnu, Grimson, Andrew, Lipkin, Steven M., Clark, Andrew G., Yu, Haiyuan

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A full-proteome, interaction-specific characterization of mutational hotspots across human cancers 由 Chen, Siwei, Liu, Yuan, Zhang, Yingying, Wierbowski, Shayne D., Lipkin, Steven M., Wei, Xiaomu, Yu, Haiyuan

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Interactome INSIDER: a structural interactome browser for genomic studies 由 Meyer, Michael J., Beltrán, Juan Felipe, Liang, Siqi, Fragoza, Robert, Rumack, Aaron, Liang, Jin, Wei, Xiaomu, Yu, Haiyuan

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GeMSTONE: orchestrated prioritization of human germline mutations in the cloud 由 Chen, Siwei, Beltrán, Juan F., Esteban-Jurado, Clara, Franch-Expósito, Sebastià, Castellví-Bel, Sergi, Lipkin, Steven, Wei, Xiaomu, Yu, Haiyuan

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Comparative exome sequencing of metastatic lesions provides insights into the mutational progression of melanoma 由 Gartner, Jared J, Davis, Sean, Wei, Xiaomu, Lin, Jimmy C, Trivedi, Niraj S, Teer, Jamie K, Meltzer, Paul S, Rosenberg, Steven A, Samuels, Yardena

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Analysis of the Tyrosine Kinome in Melanoma Reveals Recurrent Mutations in ERBB4 由 Prickett, Todd D., Agrawal, Neena S., Wei, Xiaomu, Yates, Kristin E., Lin, Jimmy C., Wunderlich, John, Cronin, Julia C., Cruz, Pedro, Rosenberg, Steven A., Samuels, Yardena

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Analysis of the Disintegrin-metalloproteinases Family Reveals ADAM29 and ADAM7 Are Often Mutated in Melanoma 由 Wei, Xiaomu, Moncada-Pazos, Angela, Cal, Santiago, Soria-Valles, Clara, Gartner, Jared, Rudloff, Udo, Lin, Jimmy C., Rosenberg, Steven A., López-Otín, Carlos, Samuels, Yardena

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Elucidating Common Structural Features of Human Pathogenic Variations Using Large-Scale Atomic-Resolution Protein Networks 由 Das, Jishnu, Lee, Hao Ran, Sagar, Adithya, Fragoza, Robert, Liang, Jin, Wei, Xiaomu, Wang, Xiujuan, Mort, Matthew, Stenson, Peter D., Cooper, David N., Yu, Haiyuan

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Mutational and Functional Analysis Reveals ADAMTS18 Metalloproteinase as a Novel Oncogene in Melanoma 由 Wei, Xiaomu, Prickett, Todd D., Viloria, Cristina G., Molinolo, Alfredo, Lin, Jimmy C., Cardenas-Navia, Isabel, Cruz, Pedro, Rosenberg, Steven A., Davies, Michael A., Gershenwald, Jeffrey E., López-Otín, Carlos, Samuels, Yardena

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Inactivation of YAP oncoprotein by the Hippo pathway is involved in cell contact inhibition and tissue growth control 由 Zhao, Bin, Wei, Xiaomu, Li, Weiquan, Udan, Ryan S., Yang, Qian, Kim, Joungmok, Xie, Joe, Ikenoue, Tsuneo, Yu, Jindan, Li, Li, Zheng, Pan, Ye, Keqiang, Chinnaiyan, Arul, Halder, Georg, Lai, Zhi-Chun, Guan, Kun-Liang

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A Massively Parallel Pipeline to Clone DNA Variants and Examine Molecular Phenotypes of Human Disease Mutations 由 Wei, Xiaomu, Das, Jishnu, Fragoza, Robert, Liang, Jin, Bastos de Oliveira, Francisco M., Lee, Hao Ran, Wang, Xiujuan, Mort, Matthew, Stenson, Peter D., Cooper, David N., Lipkin, Steven M., Smolka, Marcus B., Yu, Haiyuan

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Exome sequencing identifies GRIN2A as frequently mutated in melanoma 由 Wei, Xiaomu, Walia, Vijay, Lin, Jimmy C, Teer, Jamie K, Prickett, Todd D, Gartner, Jared, Davis, Sean, Stemke-Hale, Katherine, Davies, Michael A, Gershenwald, Jeffrey E, Robinson, William, Robinson, Steven, Rosenberg, Steven A, Samuels, Yardena

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Frequent Mutations in the MITF Pathway in Melanoma 由 Cronin, Julia C., Wunderlich, John, Loftus, Stacie K., Prickett, Todd D., Wei, Xiaomu, Ridd, Katie, Vemula, Swapna, Burrell, Allison S., Agrawal, Neena S., Lin, Jimmy C., Banister, Carolyn E., Buckhaults, Phillip, Rosenberg, Steven A., Bastian, Boris C., Pavan, William J., Samuels, Yardena

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Cross-Species Protein Interactome Mapping Reveals Species-Specific Wiring of Stress-Response Pathways() 由 Das, Jishnu, Vo, Tommy V., Wei, Xiaomu, Mellor, Joseph C., Tong, Virginia, Degatano, Andrew G., Wang, Xiujuan, Wang, Lihua, Cordero, Nicolas A., Kruer-Zerhusen, Nathan, Matsuyama, Akihisa, Pleiss, Jeffrey A., Lipkin, Steven M., Yoshida, Minoru, Roth, Frederick P., Yu, Haiyuan

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Sequencing at lymphoid neoplasm susceptibility loci maps six myeloma risk genes 由 Waller, Rosalie Griffin, Klein, Robert J, Vijai, Joseph, McKay, James D, Clay-Gilmour, Alyssa, Wei, Xiaomu, Madsen, Michael J, Sborov, Douglas W, Curtin, Karen, Slager, Susan L, Offit, Kenneth, Vachon, Celine M, Lipkin, Steven M, Dumontet, Charles, Camp, Nicola J

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Exon capture analysis of G protein-coupled receptors identifies activating mutations in GRM3 in melanoma 由 Prickett, Todd D, Wei, Xiaomu, Cardenas-Navia, Isabel, Teer, Jamie K, Lin, Jimmy C, Walia, Vijay, Gartner, Jared, Jiang, Jiji, Cherukuri, Praveen F, Molinolo, Alfredo, Davies, Michael A, Gershenwald, Jeffrey E, Stemke-Hale, Katherine, Rosenberg, Steven A, Margulies, Elliott H, Samuels, Yardena

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Somatic mutations in MAP3K5 attenuate its pro-apoptotic function in melanoma through increased binding to Thioredoxin 由 Prickett, Todd D., Zerlanko, Brad, Gartner, Jared J., Parker, Stephen C. J., Dutton-Regester, Ken, Lin, Jimmy C., Teer, Jamie K., Wei, Xiaomu, Jiang, Jiji, Chen, Guo, Davies, Michael A., Gershenwald, Jeffrey E., Robinson, William, Robinson, Steven, Hayward, Nicholas K., Rosenberg, Steven, A., Margulies, Elliott H., Samuels, Yardena

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Extensive disruption of protein interactions by genetic variants across the allele frequency spectrum in human populations 由 Fragoza, Robert, Das, Jishnu, Wierbowski, Shayne D., Liang, Jin, Tran, Tina N., Liang, Siqi, Beltran, Juan F., Rivera-Erick, Christen A., Ye, Kaixiong, Wang, Ting-Yi, Yao, Li, Mort, Matthew, Stenson, Peter D., Cooper, David N., Wei, Xiaomu, Keinan, Alon, Schimenti, John C., Clark, Andrew G., Yu, Haiyuan

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