Resultados da pesquisa - Vijayanand Modur

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  1. 1
    FOXO Proteins Regulate Tumor Necrosis Factor-related Apoptosis Inducing Ligand Expression

    FOXO Proteins Regulate Tumor Necrosis Factor-related Apoptosis Inducing Ligand Expression Por Vijayanand Modur, Rakesh Nagarajan, B. Mark Evers, Jeffrey Milbrandt

    Publicado em 2002
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  2. 2
    Borrelia burgdorferi outer membrane protein A induces nuclear translocation of nuclear factor-kappa B and inflammatory activation in human endothelial cells.

    Borrelia burgdorferi outer membrane protein A induces nuclear translocation of nuclear factor-kappa B and inflammatory activation in human endothelial cells. Por R. Mark Wooten, Vijayanand Modur, Thomas M. McIntyre, Janis J. Weis

    Publicado em 1996
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  3. 3
    Endothelial Cell Inflammatory Responses to Tumor Necrosis Factor α

    Endothelial Cell Inflammatory Responses to Tumor Necrosis Factor α Por Vijayanand Modur, Guy A. Zimmerman, Stephen M. Prescott, Thomas M. McIntyre

    Publicado em 1996
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  4. 4
    Retrograde inflammatory signaling from neutrophils to endothelial cells by soluble interleukin-6 receptor alpha.

    Retrograde inflammatory signaling from neutrophils to endothelial cells by soluble interleukin-6 receptor alpha. Por Vijayanand Modur, Yafei Li, Guy A. Zimmerman, Stephen M. Prescott, Thomas M. McIntyre

    Publicado em 1997
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  5. 5
    The necrotic venom of the brown recluse spider induces dysregulated endothelial cell-dependent neutrophil activation. Differential induction of GM-CSF, IL-8, and E-selectin expression.

    The necrotic venom of the brown recluse spider induces dysregulated endothelial cell-dependent neutrophil activation. Differential induction of GM-CSF, IL-8, and E-selectin express... Por Kamala D. Patel, Vijayanand Modur, Guy A. Zimmerman, S M Prescott, Thomas M. McIntyre

    Publicado em 1994
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  6. 6
    Oncostatin M is a proinflammatory mediator. In vivo effects correlate with endothelial cell expression of inflammatory cytokines and adhesion molecules.

    Oncostatin M is a proinflammatory mediator. In vivo effects correlate with endothelial cell expression of inflammatory cytokines and adhesion molecules. Por Vijayanand Modur, Michael J. Feldhaus, Andrew S. Weyrich, Douglas L. Jicha, Stephen M. Prescott, Gary A. Zimmerman, Thomas M. McIntyre

    Publicado em 1997
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