Søgeresultater - Tsukasa Oikawa

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  1. 1
    Membrane lipids in invadopodia and podosomes: key structures for cancer invasion and metastasis.

    Membrane lipids in invadopodia and podosomes: key structures for cancer invasion and metastasis. af Hideki Yamaguchi, Tsukasa Oikawa

    Udgivet 2010
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    Revisão
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  2. 2
    Sequential signals toward podosome formation in NIH-src cells

    Sequential signals toward podosome formation in NIH-src cells af Tsukasa Oikawa, Toshiki Itoh, Tadaomi Takenawa

    Udgivet 2008
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    Artigo
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  3. 3
    Optimization of WAVE2 complex–induced actin polymerization by membrane-bound IRSp53, PIP3, and Rac

    Optimization of WAVE2 complex–induced actin polymerization by membrane-bound IRSp53, PIP3, and Rac af Shiro Suetsugu, Shusaku Kurisu, Tsukasa Oikawa, Daisuke Yamazaki, Atsushi Oda, Tadaomi Takenawa

    Udgivet 2006
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    Artigo
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  4. 4
    Coordination between the actin cytoskeleton and membrane deformation by a novel membrane tubulation domain of PCH proteins is involved in endocytosis

    Coordination between the actin cytoskeleton and membrane deformation by a novel membrane tubulation domain of PCH proteins is involved in endocytosis af Kazuya Tsujita, Shiro Suetsugu, Nobunari Sasaki, Masahiro Furutani, Tsukasa Oikawa, Tadaomi Takenawa

    Udgivet 2006
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    Artigo
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  5. 5
    Tks5-dependent formation of circumferential podosomes/invadopodia mediates cell–cell fusion

    Tks5-dependent formation of circumferential podosomes/invadopodia mediates cell–cell fusion af Tsukasa Oikawa, Masaaki Oyama, Hiroko Kozuka‐Hata, Shunsuke Uehara, Nobuyuki Udagawa, Hideyuki Saya, Koichi Matsuo

    Udgivet 2012
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    Artigo
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  6. 6
    ZEB1 induces EPB41L5 in the cancer mesenchymal program that drives ARF6-based invasion, metastasis and drug resistance

    ZEB1 induces EPB41L5 in the cancer mesenchymal program that drives ARF6-based invasion, metastasis and drug resistance af Ari Hashimoto, Shigeru Hashimoto, Hiroaki Sugino, A. Yoshikawa, Yasuhito Onodera, Haruka Handa, Tsukasa Oikawa, Hisataka Sabe

    Udgivet 2016
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    Artigo
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  7. 7
    Frequent overexpression of AMAP1, an Arf6 effector in cell invasion, is characteristic of the MMTV-PyMT rather than the MMTV-Neu human breast cancer model

    Frequent overexpression of AMAP1, an Arf6 effector in cell invasion, is characteristic of the MMTV-PyMT rather than the MMTV-Neu human breast cancer model af Yutaro Otsuka, Tsukasa Oikawa, Hinako Yoshino, Shigeru Hashimoto, Haruka Handa, Hiroki Yamamoto, Ari Hashimoto, Hisataka Sabe

    Udgivet 2018
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    Artigo
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  8. 8
    The RAC Binding Domain/IRSp53-MIM Homology Domain of IRSp53 Induces RAC-dependent Membrane Deformation

    The RAC Binding Domain/IRSp53-MIM Homology Domain of IRSp53 Induces RAC-dependent Membrane Deformation af Shiro Suetsugu, Kazutaka Murayama, Ayako Sakamoto, Kyoko Hanawa‐Suetsugu, Azusa Seto, Tsukasa Oikawa, Chiemi Mishima, Mikako Shirouzu, Tadaomi Takenawa, Shigeyuki Yokoyama

    Udgivet 2006
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    Artigo
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  9. 9
    Lysophosphatidic acid activates Arf6 to promote the mesenchymal malignancy of renal cancer

    Lysophosphatidic acid activates Arf6 to promote the mesenchymal malignancy of renal cancer af Shigeru Hashimoto, Shuji Mikami, Hirokazu Sugino, Ayumu Yoshikawa, Ari Hashimoto, Yasuhito Onodera, Shotaro Furukawa, Haruka Handa, Tsukasa Oikawa, Yasunori Okada, Mototsugu Oya, Hisataka Sabe

    Udgivet 2016
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    Artigo
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  10. 10
    P53- and mevalonate pathway–driven malignancies require Arf6 for metastasis and drug resistance

    P53- and mevalonate pathway–driven malignancies require Arf6 for metastasis and drug resistance af Ari Hashimoto, Tsukasa Oikawa, Shigeru Hashimoto, Hirokazu Sugino, Ayumu Yoshikawa, Yutaro Otsuka, Haruka Handa, Yasuhito Onodera, Jin‐Min Nam, Chitose Oneyama, Masato Okada, Mitsunori Fukuda, Hisataka Sabe

    Udgivet 2016
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    Artigo
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  11. 11
    ARF6 and AMAP1 are major targets of <i>KRAS</i> and <i>TP53</i> mutations to promote invasion, PD-L1 dynamics, and immune evasion of pancreatic cancer

    ARF6 and AMAP1 are major targets of <i>KRAS</i> and <i>TP53</i> mutations to promote invasion, PD-L1 dynamics, and immune evasion of pancreatic cancer af Shigeru Hashimoto, Shotaro Furukawa, Ari Hashimoto, Akio Tsutaho, Akira Fukao, Yurika Sakamura, Gyanu Parajuli, Yasuhito Onodera, Yutaro Otsuka, Haruka Handa, Tsukasa Oikawa, Soichiro Hata, Yoshihiro Nishikawa, Yusuke Mizukami, Yuzo Kodama, Masaaki Murakami, Toshinobu Fujiwara, Satoshi Hirano, Hisataka Sabe

    Udgivet 2019
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    Artigo
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