Canlyniadau Chwilio am Awdur

1

Erythromelalgia: A hereditary pain syndrome enters the molecular era gan Stephen G. Waxman, Sulayman D. Dib‐Hajj

Cyhoeddwyd 2005

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Artigo

Ychwanegu at ffefrynnau

Wedi'i Gadw mewn:
2

Fibroblast Growth Factor Homologous Factor 1B Binds to the C Terminus of the Tetrodotoxin-resistant Sodium Channel rNav1.9a (NaN) gan Liu C, Sulayman D. Dib‐Hajj, Stephen G. Waxman

Cyhoeddwyd 2001

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Artigo

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Wedi'i Gadw mewn:
3

Electrophysiological Properties of Mutant Nav1.7 Sodium Channels in a Painful Inherited Neuropathy gan Theodore Cummins, Sulayman D. Dib‐Hajj, Stephen G. Waxman

Cyhoeddwyd 2004

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Artigo

Ychwanegu at ffefrynnau

Wedi'i Gadw mewn:
4

Sodium channels in pain disorders: pathophysiology and prospects for treatment gan Sulayman D. Dib‐Hajj, Paul Geha, Stephen G. Waxman

Cyhoeddwyd 2017

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Revisão

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Wedi'i Gadw mewn:
5

Electrophysiological properties of two axonal sodium channels, Nav1.2 and Nav1.6, expressed in mouse spinal sensory neurones gan Anthony M. Rush, Sulayman D. Dib‐Hajj, Stephen G. Waxman

Cyhoeddwyd 2005

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Artigo

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Wedi'i Gadw mewn:
6

Voltage-Gated Sodium Channels: Therapeutic Targets for Pain gan Sulayman D. Dib‐Hajj, Joel A. Black, Stephen G. Waxman

Cyhoeddwyd 2009

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Revisão

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Wedi'i Gadw mewn:
7

Size Matters: Erythromelalgia Mutation S241T in Nav1.7 Alters Channel Gating gan Angelika Lampert, Sulayman D. Dib‐Hajj, Lynda Tyrrell, Stephen G. Waxman

Cyhoeddwyd 2006

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Artigo

Ychwanegu at ffefrynnau

Wedi'i Gadw mewn:
8

Mutation I136V Alters Electrophysiological Properties of the NaV1.7 Channel in a Family with Onset of Erythromelalgia in the Second Decade gan Xiaoyang Cheng, Sulayman D. Dib‐Hajj, Lynda Tyrrell, Stephen G. Waxman

Cyhoeddwyd 2008

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Artigo

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Wedi'i Gadw mewn:
9

Glycosylation Alters Steady-State Inactivation of Sodium Channel Nav1.9/NaN in Dorsal Root Ganglion Neurons and Is Developmentally Regulated gan Lynda Tyrrell, Muthukrishnan Renganathan, Sulayman D. Dib‐Hajj, Stephen G. Waxman

Cyhoeddwyd 2001

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Artigo

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Wedi'i Gadw mewn:
10

Glial-Derived Neurotrophic Factor Upregulates Expression of Functional SNS and NaN Sodium Channels and Their Currents in Axotomized Dorsal Root Ganglion Neurons gan Theodore Cummins, Joel A. Black, Sulayman D. Dib‐Hajj, Stephen G. Waxman

Cyhoeddwyd 2000

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Artigo

Ychwanegu at ffefrynnau

Wedi'i Gadw mewn:
11

Inherited Neuronal Ion Channelopathies: New Windows on Complex Neurological Diseases gan William A. Catterall, Sulayman D. Dib‐Hajj, Miriam H. Meisler, Daniela Pietrobon

Cyhoeddwyd 2008

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Revisão

Ychwanegu at ffefrynnau

Wedi'i Gadw mewn:
12

NaN, a novel voltage-gated Na channel, is expressed preferentially in peripheral sensory neurons and down-regulated after axotomy gan Sulayman D. Dib‐Hajj, Lynda Tyrrell, J.A. Black, Stephen G. Waxman

Cyhoeddwyd 1998

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Artigo

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Wedi'i Gadw mewn:
13

Nav1.6 channels generate resurgent sodium currents in spinal sensory neurons gan Theodore Cummins, Sulayman D. Dib‐Hajj, Raimund I. Herzog, Stephen G. Waxman

Cyhoeddwyd 2005

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Artigo

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Wedi'i Gadw mewn:
14

Sodium channels and pain gan Stephen G. Waxman, Sulayman D. Dib‐Hajj, Theodore Cummins, J.A. Black

Cyhoeddwyd 1999

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Revisão

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Wedi'i Gadw mewn:
15

Expression of Nav1.7 in DRG Neurons Extends from Peripheral Terminals in the Skin to Central Preterminal Branches and Terminals in the Dorsal Horn gan Joel A. Black, Noémie Frezel, Sulayman D. Dib‐Hajj, Stephen G. Waxman

Cyhoeddwyd 2012

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Artigo

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Wedi'i Gadw mewn:
16

Down-regulation of transcripts for Na channel α-SNS in spinal sensory neurons following axotomy gan Sulayman D. Dib‐Hajj, J.A. Black, Paul A. Felts, Stephen G. Waxman

Cyhoeddwyd 1996

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Artigo

Ychwanegu at ffefrynnau

Wedi'i Gadw mewn:
17

Virus-Mediated Knockdown of Nav1.3 in Dorsal Root Ganglia of STZ-Induced Diabetic Rats Alleviates Tactile Allodynia gan Andrew M. Tan, Omar Abdel Samad, Sulayman D. Dib‐Hajj, Stephen G. Waxman

Cyhoeddwyd 2015

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Artigo

Ychwanegu at ffefrynnau

Wedi'i Gadw mewn:
18

The Role of Voltage-Gated Sodium Channels in Pain Signaling gan David Bennett, Alex Clark, Jianying Huang, Stephen G. Waxman, Sulayman D. Dib‐Hajj

Cyhoeddwyd 2019

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Revisão

Ychwanegu at ffefrynnau

Wedi'i Gadw mewn:
19

Distinct repriming and closed-state inactivation kinetics of Nav1.6 and Nav1.7 sodium channels in mouse spinal sensory neurons gan Raimund I. Herzog, Theodore Cummins, Farshid Ghassemi, Sulayman D. Dib‐Hajj, Stephen G. Waxman

Cyhoeddwyd 2003

Cael y testun llawn
Artigo

Ychwanegu at ffefrynnau

Wedi'i Gadw mewn:
20

Two Nedd4-binding Motifs Underlie Modulation of Sodium Channel Nav1.6 by p38 MAPK gan Andreas Gasser, Xiaoyang Cheng, Elaine S. Gilmore, Lynda Tyrrell, Stephen G. Waxman, Sulayman D. Dib‐Hajj

Cyhoeddwyd 2010

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Artigo

Ychwanegu at ffefrynnau

Wedi'i Gadw mewn:

Erythromelalgia: A hereditary pain syndrome enters the molecular era gan Stephen G. Waxman, Sulayman D. Dib‐Hajj

Fibroblast Growth Factor Homologous Factor 1B Binds to the C Terminus of the Tetrodotoxin-resistant Sodium Channel rNav1.9a (NaN) gan Liu C, Sulayman D. Dib‐Hajj, Stephen G. Waxman

Electrophysiological Properties of Mutant Na<sub>v</sub>1.7 Sodium Channels in a Painful Inherited Neuropathy gan Theodore Cummins, Sulayman D. Dib‐Hajj, Stephen G. Waxman

Sodium channels in pain disorders: pathophysiology and prospects for treatment gan Sulayman D. Dib‐Hajj, Paul Geha, Stephen G. Waxman

Electrophysiological properties of two axonal sodium channels, Na<sub>v</sub>1.2 and Na<sub>v</sub>1.6, expressed in mouse spinal sensory neurones gan Anthony M. Rush, Sulayman D. Dib‐Hajj, Stephen G. Waxman

Voltage-Gated Sodium Channels: Therapeutic Targets for Pain gan Sulayman D. Dib‐Hajj, Joel A. Black, Stephen G. Waxman

Size Matters: Erythromelalgia Mutation S241T in Nav1.7 Alters Channel Gating gan Angelika Lampert, Sulayman D. Dib‐Hajj, Lynda Tyrrell, Stephen G. Waxman

Mutation I136V Alters Electrophysiological Properties of the Na<sub>V</sub>1.7 Channel in a Family with Onset of Erythromelalgia in the Second Decade gan Xiaoyang Cheng, Sulayman D. Dib‐Hajj, Lynda Tyrrell, Stephen G. Waxman

Glycosylation Alters Steady-State Inactivation of Sodium Channel Na<sub>v</sub>1.9/NaN in Dorsal Root Ganglion Neurons and Is Developmentally Regulated gan Lynda Tyrrell, Muthukrishnan Renganathan, Sulayman D. Dib‐Hajj, Stephen G. Waxman

Glial-Derived Neurotrophic Factor Upregulates Expression of Functional SNS and NaN Sodium Channels and Their Currents in Axotomized Dorsal Root Ganglion Neurons gan Theodore Cummins, Joel A. Black, Sulayman D. Dib‐Hajj, Stephen G. Waxman

Inherited Neuronal Ion Channelopathies: New Windows on Complex Neurological Diseases gan William A. Catterall, Sulayman D. Dib‐Hajj, Miriam H. Meisler, Daniela Pietrobon

NaN, a novel voltage-gated Na channel, is expressed preferentially in peripheral sensory neurons and down-regulated after axotomy gan Sulayman D. Dib‐Hajj, Lynda Tyrrell, J.A. Black, Stephen G. Waxman

Na<sub>v</sub>1.6 channels generate resurgent sodium currents in spinal sensory neurons gan Theodore Cummins, Sulayman D. Dib‐Hajj, Raimund I. Herzog, Stephen G. Waxman

Sodium channels and pain gan Stephen G. Waxman, Sulayman D. Dib‐Hajj, Theodore Cummins, J.A. Black

Expression of Nav1.7 in DRG Neurons Extends from Peripheral Terminals in the Skin to Central Preterminal Branches and Terminals in the Dorsal Horn gan Joel A. Black, Noémie Frezel, Sulayman D. Dib‐Hajj, Stephen G. Waxman

Down-regulation of transcripts for Na channel α-SNS in spinal sensory neurons following axotomy gan Sulayman D. Dib‐Hajj, J.A. Black, Paul A. Felts, Stephen G. Waxman

Virus-Mediated Knockdown of Nav1.3 in Dorsal Root Ganglia of STZ-Induced Diabetic Rats Alleviates Tactile Allodynia gan Andrew M. Tan, Omar Abdel Samad, Sulayman D. Dib‐Hajj, Stephen G. Waxman

The Role of Voltage-Gated Sodium Channels in Pain Signaling gan David Bennett, Alex Clark, Jianying Huang, Stephen G. Waxman, Sulayman D. Dib‐Hajj

Distinct repriming and closed-state inactivation kinetics of Nav1.6 and Nav1.7 sodium channels in mouse spinal sensory neurons gan Raimund I. Herzog, Theodore Cummins, Farshid Ghassemi, Sulayman D. Dib‐Hajj, Stephen G. Waxman

Two Nedd4-binding Motifs Underlie Modulation of Sodium Channel Nav1.6 by p38 MAPK gan Andreas Gasser, Xiaoyang Cheng, Elaine S. Gilmore, Lynda Tyrrell, Stephen G. Waxman, Sulayman D. Dib‐Hajj

Canlyniadau Chwilio - Sulayman D. Dib‐Hajj

Erythromelalgia: A hereditary pain syndrome enters the molecular era gan Stephen G. Waxman, Sulayman D. Dib‐Hajj

Fibroblast Growth Factor Homologous Factor 1B Binds to the C Terminus of the Tetrodotoxin-resistant Sodium Channel rNav1.9a (NaN) gan Liu C, Sulayman D. Dib‐Hajj, Stephen G. Waxman

Electrophysiological Properties of Mutant Na<sub>v</sub>1.7 Sodium Channels in a Painful Inherited Neuropathy gan Theodore Cummins, Sulayman D. Dib‐Hajj, Stephen G. Waxman

Sodium channels in pain disorders: pathophysiology and prospects for treatment gan Sulayman D. Dib‐Hajj, Paul Geha, Stephen G. Waxman

Electrophysiological properties of two axonal sodium channels, Na<sub>v</sub>1.2 and Na<sub>v</sub>1.6, expressed in mouse spinal sensory neurones gan Anthony M. Rush, Sulayman D. Dib‐Hajj, Stephen G. Waxman

Voltage-Gated Sodium Channels: Therapeutic Targets for Pain gan Sulayman D. Dib‐Hajj, Joel A. Black, Stephen G. Waxman

Size Matters: Erythromelalgia Mutation S241T in Nav1.7 Alters Channel Gating gan Angelika Lampert, Sulayman D. Dib‐Hajj, Lynda Tyrrell, Stephen G. Waxman

Mutation I136V Alters Electrophysiological Properties of the Na<sub>V</sub>1.7 Channel in a Family with Onset of Erythromelalgia in the Second Decade gan Xiaoyang Cheng, Sulayman D. Dib‐Hajj, Lynda Tyrrell, Stephen G. Waxman

Glycosylation Alters Steady-State Inactivation of Sodium Channel Na<sub>v</sub>1.9/NaN in Dorsal Root Ganglion Neurons and Is Developmentally Regulated gan Lynda Tyrrell, Muthukrishnan Renganathan, Sulayman D. Dib‐Hajj, Stephen G. Waxman

Glial-Derived Neurotrophic Factor Upregulates Expression of Functional SNS and NaN Sodium Channels and Their Currents in Axotomized Dorsal Root Ganglion Neurons gan Theodore Cummins, Joel A. Black, Sulayman D. Dib‐Hajj, Stephen G. Waxman

Inherited Neuronal Ion Channelopathies: New Windows on Complex Neurological Diseases gan William A. Catterall, Sulayman D. Dib‐Hajj, Miriam H. Meisler, Daniela Pietrobon

NaN, a novel voltage-gated Na channel, is expressed preferentially in peripheral sensory neurons and down-regulated after axotomy gan Sulayman D. Dib‐Hajj, Lynda Tyrrell, J.A. Black, Stephen G. Waxman

Na<sub>v</sub>1.6 channels generate resurgent sodium currents in spinal sensory neurons gan Theodore Cummins, Sulayman D. Dib‐Hajj, Raimund I. Herzog, Stephen G. Waxman

Sodium channels and pain gan Stephen G. Waxman, Sulayman D. Dib‐Hajj, Theodore Cummins, J.A. Black

Expression of Nav1.7 in DRG Neurons Extends from Peripheral Terminals in the Skin to Central Preterminal Branches and Terminals in the Dorsal Horn gan Joel A. Black, Noémie Frezel, Sulayman D. Dib‐Hajj, Stephen G. Waxman

Down-regulation of transcripts for Na channel α-SNS in spinal sensory neurons following axotomy gan Sulayman D. Dib‐Hajj, J.A. Black, Paul A. Felts, Stephen G. Waxman

Virus-Mediated Knockdown of Nav1.3 in Dorsal Root Ganglia of STZ-Induced Diabetic Rats Alleviates Tactile Allodynia gan Andrew M. Tan, Omar Abdel Samad, Sulayman D. Dib‐Hajj, Stephen G. Waxman

The Role of Voltage-Gated Sodium Channels in Pain Signaling gan David Bennett, Alex Clark, Jianying Huang, Stephen G. Waxman, Sulayman D. Dib‐Hajj

Distinct repriming and closed-state inactivation kinetics of Nav1.6 and Nav1.7 sodium channels in mouse spinal sensory neurons gan Raimund I. Herzog, Theodore Cummins, Farshid Ghassemi, Sulayman D. Dib‐Hajj, Stephen G. Waxman

Two Nedd4-binding Motifs Underlie Modulation of Sodium Channel Nav1.6 by p38 MAPK gan Andreas Gasser, Xiaoyang Cheng, Elaine S. Gilmore, Lynda Tyrrell, Stephen G. Waxman, Sulayman D. Dib‐Hajj

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