Rezultati - Stephen M. Strittmatter

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  1. 1
    Overcoming Drug Development Bottlenecks With Repurposing: Old drugs learn new tricks

    Overcoming Drug Development Bottlenecks With Repurposing: Old drugs learn new tricks od Stephen M. Strittmatter

    Izdano 2014
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  2. 2
    Rac1 Mediates Collapsin-1-Induced Growth Cone Collapse

    Rac1 Mediates Collapsin-1-Induced Growth Cone Collapse od Jin Zhao, Stephen M. Strittmatter

    Izdano 1997
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  3. 3
    The N-Terminal Domain of Nogo-A Inhibits Cell Adhesion and Axonal Outgrowth by an Integrin-Specific Mechanism

    The N-Terminal Domain of Nogo-A Inhibits Cell Adhesion and Axonal Outgrowth by an Integrin-Specific Mechanism od Fenghua Hu, Stephen M. Strittmatter

    Izdano 2008
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  4. 4
    P2Y1 purinergic receptors in sensory neurons: contribution to touch-induced impulse generation.

    P2Y1 purinergic receptors in sensory neurons: contribution to touch-induced impulse generation. od Fumio Nakamura, Stephen M. Strittmatter

    Izdano 1996
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  5. 5
    PlexinA1 Autoinhibition by the Plexin Sema Domain

    PlexinA1 Autoinhibition by the Plexin Sema Domain od Takuya Takahashi, Stephen M. Strittmatter

    Izdano 2001
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  6. 6
    The reticulons: a family of proteins with diverse functions

    The reticulons: a family of proteins with diverse functions od Yvonne S. Yang, Stephen M. Strittmatter

    Izdano 2007
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  7. 7
    Delayed Systemic Nogo-66 Receptor Antagonist Promotes Recovery from Spinal Cord Injury

    Delayed Systemic Nogo-66 Receptor Antagonist Promotes Recovery from Spinal Cord Injury od Shuxin Li, Stephen M. Strittmatter

    Izdano 2003
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  8. 8
    Oligomers of Amyloid β Prevent Physiological Activation of the Cellular Prion Protein-Metabotropic Glutamate Receptor 5 Complex by Glutamate in Alzheimer Disease

    Oligomers of Amyloid β Prevent Physiological Activation of the Cellular Prion Protein-Metabotropic Glutamate Receptor 5 Complex by Glutamate in Alzheimer Disease od Laura T. Haas, Stephen M. Strittmatter

    Izdano 2016
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  9. 9
    The CRMP Family of Proteins and Their Role in Sema3A Signaling

    The CRMP Family of Proteins and Their Role in Sema3A Signaling od Eric F. Schmidt, Stephen M. Strittmatter

    Izdano 2007
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  10. 10
    A Family of Rat CRMP Genes Is Differentially Expressed in the Nervous System

    A Family of Rat CRMP Genes Is Differentially Expressed in the Nervous System od Li‐Hsien Wang, Stephen M. Strittmatter

    Izdano 1996
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  11. 11
    Angiotensin-Converting Enzyme in the Male Rat Reproductive System: Autoradiographic Visualization with [<sup>3</sup>H]Captopril*

    Angiotensin-Converting Enzyme in the Male Rat Reproductive System: Autoradiographic Visualization with [<sup>3</sup>H]Captopril* od Stephen M. Strittmatter, Solomon H. Snyder

    Izdano 1984
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  12. 12
    Can regenerating axons recapitulate developmental guidance during recovery from spinal cord injury?

    Can regenerating axons recapitulate developmental guidance during recovery from spinal cord injury? od Noam Y. Harel, Stephen M. Strittmatter

    Izdano 2006
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  13. 13
    β-amyloid oligomers and cellular prion protein in Alzheimer’s disease

    β-amyloid oligomers and cellular prion protein in Alzheimer’s disease od Erik C. Gunther, Stephen M. Strittmatter

    Izdano 2009
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  14. 14
    Properties of the separated catalytic and regulatory units of brain adenylate cyclase.

    Properties of the separated catalytic and regulatory units of brain adenylate cyclase. od Stephen M. Strittmatter, Eva J. Neer

    Izdano 1980
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  15. 15
    Cellular Prion Protein Mediates the Toxicity of β-Amyloid Oligomers

    Cellular Prion Protein Mediates the Toxicity of β-Amyloid Oligomers od Haakon B. Nygaard, Stephen M. Strittmatter

    Izdano 2009
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  16. 16
    Amyloid-β induced signaling by cellular prion protein and Fyn kinase in Alzheimer disease

    Amyloid-β induced signaling by cellular prion protein and Fyn kinase in Alzheimer disease od Ji Won Um, Stephen M. Strittmatter

    Izdano 2013
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  17. 17
    Nogo limits neural plasticity and recovery from injury

    Nogo limits neural plasticity and recovery from injury od Martin E. Schwab, Stephen M. Strittmatter

    Izdano 2014
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  18. 18
    Cellular prion protein as a receptor for amyloid-β oligomers in Alzheimer's disease

    Cellular prion protein as a receptor for amyloid-β oligomers in Alzheimer's disease od Santiago V. Salazar, Stephen M. Strittmatter

    Izdano 2016
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  19. 19
    Binding Sites for Amyloid-β Oligomers and Synaptic Toxicity

    Binding Sites for Amyloid-β Oligomers and Synaptic Toxicity od Levi M. Smith, Stephen M. Strittmatter

    Izdano 2016
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  20. 20
    Synaptotoxic Signaling by Amyloid Beta Oligomers in Alzheimer's Disease Through Prion Protein and mGluR5

    Synaptotoxic Signaling by Amyloid Beta Oligomers in Alzheimer's Disease Through Prion Protein and mGluR5 od A. Harrison Brody, Stephen M. Strittmatter

    Izdano 2017
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