Resultats de la cerca - Stephen M. Strittmatter

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  1. 1
    Overcoming Drug Development Bottlenecks With Repurposing: Old drugs learn new tricks

    Overcoming Drug Development Bottlenecks With Repurposing: Old drugs learn new tricks per Stephen M. Strittmatter

    Publicat 2014
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  2. 2
    Rac1 Mediates Collapsin-1-Induced Growth Cone Collapse

    Rac1 Mediates Collapsin-1-Induced Growth Cone Collapse per Jin Zhao, Stephen M. Strittmatter

    Publicat 1997
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  3. 3
    The N-Terminal Domain of Nogo-A Inhibits Cell Adhesion and Axonal Outgrowth by an Integrin-Specific Mechanism

    The N-Terminal Domain of Nogo-A Inhibits Cell Adhesion and Axonal Outgrowth by an Integrin-Specific Mechanism per Fenghua Hu, Stephen M. Strittmatter

    Publicat 2008
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  4. 4
    P2Y1 purinergic receptors in sensory neurons: contribution to touch-induced impulse generation.

    P2Y1 purinergic receptors in sensory neurons: contribution to touch-induced impulse generation. per Fumio Nakamura, Stephen M. Strittmatter

    Publicat 1996
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  5. 5
    PlexinA1 Autoinhibition by the Plexin Sema Domain

    PlexinA1 Autoinhibition by the Plexin Sema Domain per Takuya Takahashi, Stephen M. Strittmatter

    Publicat 2001
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  6. 6
    The reticulons: a family of proteins with diverse functions

    The reticulons: a family of proteins with diverse functions per Yvonne S. Yang, Stephen M. Strittmatter

    Publicat 2007
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  7. 7
    Delayed Systemic Nogo-66 Receptor Antagonist Promotes Recovery from Spinal Cord Injury

    Delayed Systemic Nogo-66 Receptor Antagonist Promotes Recovery from Spinal Cord Injury per Shuxin Li, Stephen M. Strittmatter

    Publicat 2003
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  8. 8
    Oligomers of Amyloid β Prevent Physiological Activation of the Cellular Prion Protein-Metabotropic Glutamate Receptor 5 Complex by Glutamate in Alzheimer Disease

    Oligomers of Amyloid β Prevent Physiological Activation of the Cellular Prion Protein-Metabotropic Glutamate Receptor 5 Complex by Glutamate in Alzheimer Disease per Laura T. Haas, Stephen M. Strittmatter

    Publicat 2016
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  9. 9
    The CRMP Family of Proteins and Their Role in Sema3A Signaling

    The CRMP Family of Proteins and Their Role in Sema3A Signaling per Eric F. Schmidt, Stephen M. Strittmatter

    Publicat 2007
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  10. 10
    A Family of Rat CRMP Genes Is Differentially Expressed in the Nervous System

    A Family of Rat CRMP Genes Is Differentially Expressed in the Nervous System per Li‐Hsien Wang, Stephen M. Strittmatter

    Publicat 1996
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  11. 11
    Angiotensin-Converting Enzyme in the Male Rat Reproductive System: Autoradiographic Visualization with [<sup>3</sup>H]Captopril*

    Angiotensin-Converting Enzyme in the Male Rat Reproductive System: Autoradiographic Visualization with [<sup>3</sup>H]Captopril* per Stephen M. Strittmatter, Solomon H. Snyder

    Publicat 1984
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  12. 12
    Can regenerating axons recapitulate developmental guidance during recovery from spinal cord injury?

    Can regenerating axons recapitulate developmental guidance during recovery from spinal cord injury? per Noam Y. Harel, Stephen M. Strittmatter

    Publicat 2006
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  13. 13
    β-amyloid oligomers and cellular prion protein in Alzheimer’s disease

    β-amyloid oligomers and cellular prion protein in Alzheimer’s disease per Erik C. Gunther, Stephen M. Strittmatter

    Publicat 2009
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  14. 14
    Properties of the separated catalytic and regulatory units of brain adenylate cyclase.

    Properties of the separated catalytic and regulatory units of brain adenylate cyclase. per Stephen M. Strittmatter, Eva J. Neer

    Publicat 1980
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  15. 15
    Cellular Prion Protein Mediates the Toxicity of β-Amyloid Oligomers

    Cellular Prion Protein Mediates the Toxicity of β-Amyloid Oligomers per Haakon B. Nygaard, Stephen M. Strittmatter

    Publicat 2009
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  16. 16
    Amyloid-β induced signaling by cellular prion protein and Fyn kinase in Alzheimer disease

    Amyloid-β induced signaling by cellular prion protein and Fyn kinase in Alzheimer disease per Ji Won Um, Stephen M. Strittmatter

    Publicat 2013
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  17. 17
    Nogo limits neural plasticity and recovery from injury

    Nogo limits neural plasticity and recovery from injury per Martin E. Schwab, Stephen M. Strittmatter

    Publicat 2014
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  18. 18
    Cellular prion protein as a receptor for amyloid-β oligomers in Alzheimer's disease

    Cellular prion protein as a receptor for amyloid-β oligomers in Alzheimer's disease per Santiago V. Salazar, Stephen M. Strittmatter

    Publicat 2016
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  19. 19
    Binding Sites for Amyloid-β Oligomers and Synaptic Toxicity

    Binding Sites for Amyloid-β Oligomers and Synaptic Toxicity per Levi M. Smith, Stephen M. Strittmatter

    Publicat 2016
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  20. 20
    Synaptotoxic Signaling by Amyloid Beta Oligomers in Alzheimer's Disease Through Prion Protein and mGluR5

    Synaptotoxic Signaling by Amyloid Beta Oligomers in Alzheimer's Disease Through Prion Protein and mGluR5 per A. Harrison Brody, Stephen M. Strittmatter

    Publicat 2017
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