検索結果 - Shiro Ikegawa

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  1. 1
    A Short History of the Genome-Wide Association Study: Where We Were and Where We Are Going

    A Short History of the Genome-Wide Association Study: Where We Were and Where We Are Going 著者: Shiro Ikegawa

    出版事項 2012
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    Artigo
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  2. 2
    SOX9-dependent and -independent Transcriptional Regulation of Human Cartilage Link Protein

    SOX9-dependent and -independent Transcriptional Regulation of Human Cartilage Link Protein 著者: Ikuyo Kou, Shiro Ikegawa

    出版事項 2004
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    Artigo
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  3. 3
    Human Genetic Disorders Caused by Mutations in Genes Encoding Biosynthetic Enzymes for Sulfated Glycosaminoglycans

    Human Genetic Disorders Caused by Mutations in Genes Encoding Biosynthetic Enzymes for Sulfated Glycosaminoglycans 著者: Shuji Mizumoto, Shiro Ikegawa, Kazuyuki Sugahara

    出版事項 2013
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    Revisão
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  4. 4
    Identification and Functional Characterization of RSPO2 as a Susceptibility Gene for Ossification of the Posterior Longitudinal Ligament of the Spine

    Identification and Functional Characterization of RSPO2 as a Susceptibility Gene for Ossification of the Posterior Longitudinal Ligament of the Spine 著者: Masahiro Nakajima, Ikuyo Kou, Hirofumi Ohashi, Shiro Ikegawa

    出版事項 2016
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    Artigo
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  5. 5
    Prediction model for knee osteoarthritis based on genetic and clinical information

    Prediction model for knee osteoarthritis based on genetic and clinical information 著者: Hiroshi Takahashi, Masahiro Nakajima, Kouichi Ozaki, Toshihiro Tanaka, Naoyuki Kamatani, Shiro Ikegawa

    出版事項 2010
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    Artigo
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  6. 6
    Prevalence of c.1559delT in ALPL, a common mutation resulting in the perinatal (lethal) form of hypophosphatasia in Japanese and effects of the mutation on heterozygous carriers

    Prevalence of c.1559delT in ALPL, a common mutation resulting in the perinatal (lethal) form of hypophosphatasia in Japanese and effects of the mutation on heterozygous carriers 著者: Atsushi Watanabe, Tatsuki Karasugi, Hideaki Sawai, Banyar Than Naing, Shiro Ikegawa, Hideo Orimo, Takashi Shimada

    出版事項 2010
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    Artigo
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  7. 7
    SIK3 is essential for chondrocyte hypertrophy during skeletal development in mice

    SIK3 is essential for chondrocyte hypertrophy during skeletal development in mice 著者: Satoru Sasagawa, Hiroshi Takemori, Tatsuya Uebi, Daisuke Ikegami, Kunihiko Hiramatsu, Shiro Ikegawa, Hideki Yoshikawa, Noriyuki Tsumaki

    出版事項 2012
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  8. 8
    Genome-wide DNA methylation profile implicates potential cartilage regeneration at the late stage of knee osteoarthritis

    Genome-wide DNA methylation profile implicates potential cartilage regeneration at the late stage of knee osteoarthritis 著者: Y. Zhang, Naoshi Fukui, M. Yahata, Yozo Katsuragawa, Tazuko Tashiro, Shiro Ikegawa, Ming Ta Michael Lee

    出版事項 2015
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    Artigo
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  9. 9
    Identification of DNA methylation changes associated with disease progression in subchondral bone with site-matched cartilage in knee osteoarthritis

    Identification of DNA methylation changes associated with disease progression in subchondral bone with site-matched cartilage in knee osteoarthritis 著者: Yanfei Zhang, Naoshi Fukui, M. Yahata, Yozo Katsuragawa, Toshiyuki Tashiro, Shiro Ikegawa, Ming Ta Michael Lee

    出版事項 2016
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    Artigo
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  10. 10
    Replication of the association of the aspartic acid repeat polymorphism in the asporin gene with knee-osteoarthritis susceptibility in Han Chinese

    Replication of the association of the aspartic acid repeat polymorphism in the asporin gene with knee-osteoarthritis susceptibility in Han Chinese 著者: Qing Jiang, Dongquan Shi, Long Yi, Shiro Ikegawa, Yong Wang, Takahiro Nakamura, Di Qiao, Cheng Liu, Jin Dai

    出版事項 2006
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    Artigo
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  11. 11
    PLAP-1/Asporin, a Novel Negative Regulator of Periodontal Ligament Mineralization

    PLAP-1/Asporin, a Novel Negative Regulator of Periodontal Ligament Mineralization 著者: Satoru Yamada, Miki Tomoeda, Yasuhiro Ozawa, Shinya Yoneda, Yoshimitsu Terashima, Kazuhiko Ikezawa, Shiro Ikegawa, Masahiro Saito, Satoru Toyosawa, Shinya Murakami

    出版事項 2007
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    Artigo
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  12. 12
    Molecular Classification of Knee Osteoarthritis

    Molecular Classification of Knee Osteoarthritis 著者: Zhongyang Lv, Yannick Xiaofan Yang, Jiawei Li, Yuxiang Fei, Hu Guo, Ziying Sun, Jun Lü, Xingquan Xu, Qing Jiang, Shiro Ikegawa, Dongquan Shi

    出版事項 2021
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  13. 13
    Meta-analysis of association between the ASPN D-repeat and osteoarthritis

    Meta-analysis of association between the ASPN D-repeat and osteoarthritis 著者: Takahiro Nakamura, Dongquan Shi, Maria Tzetis, Julio A. Rodriguez‐Lopez, Yoshinari Miyamoto, Aspasia Tsezou, Antonio González, Qing Jiang, Naoyuki Kamatani, John Loughlin, Shiro Ikegawa

    出版事項 2007
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    Revisão
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  14. 14
    TRPV1 alleviates osteoarthritis by inhibiting M1 macrophage polarization via Ca2+/CaMKII/Nrf2 signaling pathway

    TRPV1 alleviates osteoarthritis by inhibiting M1 macrophage polarization via Ca2+/CaMKII/Nrf2 signaling pathway 著者: Zhongyang Lv, Xingquan Xu, Ziying Sun, Yannick Xiaofan Yang, Hu Guo, Jiawei Li, Kuoyang Sun, Rui Wu, Jia Xu, Qing Jiang, Shiro Ikegawa, Dongquan Shi

    出版事項 2021
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    Artigo
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  15. 15
    Association of the D repeat polymorphism in the ASPNgene with developmental dysplasia of the hip: a case-control study in Han Chinese

    Association of the D repeat polymorphism in the ASPNgene with developmental dysplasia of the hip: a case-control study in Han Chinese 著者: Dongquan Shi, Jin Dai, Pengsheng Zhu, Jianghui Qin, Lunqing Zhu, Hongtao Zhu, Baocheng Zhao, Xusheng Qiu, Zhihong Xu, Dongyang Chen, Long Yi, Shiro Ikegawa, Qing Jiang

    出版事項 2011
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    Artigo
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  16. 16
    Identification of a novel non-coding RNA, MIAT, that confers risk of myocardial infarction

    Identification of a novel non-coding RNA, MIAT, that confers risk of myocardial infarction 著者: Nobuaki Ishii, Kouichi Ozaki, Hiroshi Sato, Hiroya Mizuno, Susumu Saito, Atsushi Takahashi, Yoshinari Miyamoto, Shiro Ikegawa, Naoyuki Kamatani, Masatsugu Hori, Satoshi Saito, Yusuke Nakamura, Toshihiro Tanaka

    出版事項 2006
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    Artigo
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  17. 17
    Genetic analysis of quantitative traits in the Japanese population links cell types to complex human diseases

    Genetic analysis of quantitative traits in the Japanese population links cell types to complex human diseases 著者: Masahiro Kanai, Masato Akiyama, Atsushi Takahashi, Nana Matoba, Yukihide Momozawa, Masashi Ikeda, Nakao Iwata, Shiro Ikegawa, Makoto Hirata, Koichi Matsuda, Michiaki Kubo, Yukinori Okada, Yoichiro Kamatani

    出版事項 2018
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    Artigo
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  18. 18
    SNP rs11190870 near LBX1 is associated with adolescent idiopathic scoliosis in southern Chinese

    SNP rs11190870 near LBX1 is associated with adolescent idiopathic scoliosis in southern Chinese 著者: Yanhui Fan, You‐Qiang Song, Danny Chan, Yohei Takahashi, Shiro Ikegawa, Morio Matsumoto, Ikuyo Kou, Kathryn S.E. Cheah, Pak C. Sham, Kmc Cheung, K.D.K. Luk

    出版事項 2012
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    Artigo
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  19. 19
    Functional Investigation of a Non-coding Variant Associated with Adolescent Idiopathic Scoliosis in Zebrafish: Elevated Expression of the Ladybird Homeobox Gene Causes Body Axis Deformation

    Functional Investigation of a Non-coding Variant Associated with Adolescent Idiopathic Scoliosis in Zebrafish: Elevated Expression of the Ladybird Homeobox Gene Causes Body Axis De... 著者: Long Guo, Hiroshi Yamashita, Ikuyo Kou, Aki Takimoto, Makiko Meguro‐Horike, Shin‐ichi Horike, Tetsushi Sakuma, Shigenori Miura, Taiji ADACHI, Takashi Yamamoto, Shiro Ikegawa, Yuji Hiraki, Chisa Shukunami

    出版事項 2016
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    Artigo
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  20. 20
    Identification of loss-of-function mutations of SLC35D1 in patients with Schneckenbecken dysplasia, but not with other severe spondylodysplastic dysplasias group diseases

    Identification of loss-of-function mutations of SLC35D1 in patients with Schneckenbecken dysplasia, but not with other severe spondylodysplastic dysplasias group diseases 著者: Tatsuya Furuichi, Hülya Kayserili, Shûichi Hiraoka, Gen Nishimura, Hirofumi Ohashi, Yasemin Alanay, JUAN LERENA, Ayça Dilruba Aslanger, Haruhiko Koseki, Daniel H. Cohn, Andrea Superti‐Furga, Sheila Unger, Shiro Ikegawa

    出版事項 2009
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    Carta
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