Resultados da pesquisa - Satoshi Sakai

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  1. 1
    Molecular Pathogenesis of Pulmonary Fibrosis, with Focus on Pathways Related to TGF-β and the Ubiquitin-Proteasome Pathway

    Molecular Pathogenesis of Pulmonary Fibrosis, with Focus on Pathways Related to TGF-β and the Ubiquitin-Proteasome Pathway Por Naoki Inui, Satoshi Sakai, Masatoshi Kitagawa

    Publicado em 2021
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  2. 2
    Increased production of endothelin‐1 in the hypertrophied rat heart due to pressure overload

    Increased production of endothelin‐1 in the hypertrophied rat heart due to pressure overload Por Ryosuke Yorikane, Satoshi Sakai, Takashi Miyauchi, Takeshi Sakurai, Yasuro Sugishita, Katsutoshi Goto

    Publicado em 1993
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  3. 3
    Myocardial fibrosis and diastolic dysfunction in deoxycorticosterone acetate-salt hypertensive rats is ameliorated by the peroxisome proliferator-activated receptor-alpha activator fenofibrate, partly by suppressing inflammatory responses associated with the nuclear factor-kappa-b pathway

    Myocardial fibrosis and diastolic dysfunction in deoxycorticosterone acetate-salt hypertensive rats is ameliorated by the peroxisome proliferator-activated receptor-alpha activator... Por Takehiro Ogata, Takashi Miyauchi, Satoshi Sakai, Masakatsu Takanashi, Yoko Irukayama‐Tomobe, Iwao Yamaguchi

    Publicado em 2004
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  4. 4
    Expression of endothelin-1, ET<sub>A</sub>and ET<sub>B</sub>receptors, and ECE and distribution of endothelin-1 in failing rat heart

    Expression of endothelin-1, ET<sub>A</sub>and ET<sub>B</sub>receptors, and ECE and distribution of endothelin-1 in failing rat heart Por Tsutomu Kobayashi, Takashi Miyauchi, Satoshi Sakai, Masahiko Kobayashi, Iwao Yamaguchi, Katsutoshi Goto, Yasuro Sugishita

    Publicado em 1999
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  5. 5
    RNA-binding motif protein 47 inhibits Nrf2 activity to suppress tumor growth in lung adenocarcinoma

    RNA-binding motif protein 47 inhibits Nrf2 activity to suppress tumor growth in lung adenocarcinoma Por Toshihiro Sakurai, Kazunobu Isogaya, Satoshi Sakai, Masato Morikawa, Y Morishita, Shogo Ehata, Kohei Miyazono, Daizo Koinuma

    Publicado em 2016
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  6. 6
    Physiological and pathological cardiac hypertrophy induce different molecular phenotypes in the rat

    Physiological and pathological cardiac hypertrophy induce different molecular phenotypes in the rat Por Motoyuki Iemitsu, Takashi Miyauchi, Seiji Maeda, Satoshi Sakai, Tsutomu Kobayashi, Nobuharu Fujii, Hitoshi Miyazaki, Mitsuo Matsuda, Iwao Yamaguchi

    Publicado em 2001
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  7. 7
    Contribution of endogenous endothelin-1 to the progression of cardiopulmonary alterations in rats with monocrotaline-induced pulmonary hypertension.

    Contribution of endogenous endothelin-1 to the progression of cardiopulmonary alterations in rats with monocrotaline-induced pulmonary hypertension. Por T. Miyauchi, Ryosuke Yorikane, Satoshi Sakai, Takeshi Sakurai, Masayoshi Okada, M. Nishikibe, M. Yano, Ichiro Yamaguchi, Y Sugishita, Katsutoshi Goto

    Publicado em 1993
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  8. 8
    Micro<scp>RNA</scp>‐155‐5p is associated with oral squamous cell carcinoma metastasis and poor prognosis

    Micro<scp>RNA</scp>‐155‐5p is associated with oral squamous cell carcinoma metastasis and poor prognosis Por Osamu Baba, Shogo Hasegawa, Hiroki Nagai, Fumihiko Uchida, Masanobu Yamatoji, Naomi Kanno, Kenji Yamagata, Satoshi Sakai, Toru Yanagawa, Hiroki Bukawa

    Publicado em 2015
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  9. 9
    Endothelin-1–Induced Cardiac Hypertrophy Is Inhibited by Activation of Peroxisome Proliferator–Activated Receptor-α Partly Via Blockade of c-Jun NH <sub>2</sub> -Terminal Kinase Pathway

    Endothelin-1–Induced Cardiac Hypertrophy Is Inhibited by Activation of Peroxisome Proliferator–Activated Receptor-α Partly Via Blockade of c-Jun NH <sub>2</sub> -Terminal Kinase Pa... Por Yoko Irukayama‐Tomobe, Takashi Miyauchi, Satoshi Sakai, Yoshitoshi Kasuya, Takehiro Ogata, Masakatsu Takanashi, Motoyuki Iemitsu, T Sudo, Katsutoshi Goto, Iwao Yamaguchi

    Publicado em 2004
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  10. 10
    Long Noncoding RNA <i>ELIT-1</i> Acts as a Smad3 Cofactor to Facilitate TGFβ/Smad Signaling and Promote Epithelial–Mesenchymal Transition

    Long Noncoding RNA <i>ELIT-1</i> Acts as a Smad3 Cofactor to Facilitate TGFβ/Smad Signaling and Promote Epithelial–Mesenchymal Transition Por Satoshi Sakai, Tatsuya Ohhata, Kyoko Kitagawa, Chiharu Uchida, Takuya Aoshima, Hiroyuki Niida, Tetsuro Suzuki, Yasumichi Inoue, Keiji Miyazawa, Masatoshi Kitagawa

    Publicado em 2019
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  11. 11
    Role of Endothelin in Deterioration of Heart Failure Due to Cardiomyopathy in Hamsters

    Role of Endothelin in Deterioration of Heart Failure Due to Cardiomyopathy in Hamsters Por Rikako Yamauchi‐Kohno, Takashi Miyauchi, Tomoko Hoshino, Tsutomu Kobayashi, Hajime Aihara, Satoshi Sakai, Hideo Yabana, Katsutoshi Goto, Yasuro Sugishita, Sakae Murata

    Publicado em 1999
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  12. 12
    Tenascin‐C Aggravates Autoimmune Myocarditis via Dendritic Cell Activation and Th17 Cell Differentiation

    Tenascin‐C Aggravates Autoimmune Myocarditis via Dendritic Cell Activation and Th17 Cell Differentiation Por Tomoko Machino‐Ohtsuka, Kazuko Tajiri, Taizo Kimura, Satoshi Sakai, Akira Sato, Toshimichi Yoshida, Michiaki Hiroe, Yasuhiro Yasutomi, Kazutaka Aonuma, Kyoko Imanaka‐Yoshida

    Publicado em 2014
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  13. 13
    MicroRNA-205-5p suppresses the invasiveness of oral squamous cell carcinoma by inhibiting TIMP‑2 expression

    MicroRNA-205-5p suppresses the invasiveness of oral squamous cell carcinoma by inhibiting TIMP‑2 expression Por Hiroki Nagai, Shogo Hasegawa, Fumihiko Uchida, Takehito Terabe, Naomi Kanno, Koroku Kato, Kenji Yamagata, Satoshi Sakai, Shuichi Kawashiri, Hiroshi Sato, Toru Yanagawa, Hiroki Bukawa

    Publicado em 2018
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  14. 14
    Elevated levels of plasma endothelin-1 in young patients with pulmonary hypertension caused by congenital heart disease are decreased after successful surgical repair

    Elevated levels of plasma endothelin-1 in young patients with pulmonary hypertension caused by congenital heart disease are decreased after successful surgical repair Por Shiro Ishikawa, Takashi Miyauchi, Satoshi Sakai, Hiroya Ushinohama, Kouichi Sagawa, Naoki Fusazaki, Hideaki Kado, Hiroshi Sunagawa, Shinji Honda, Hayao Ueno, Iwao Yamaguchi, Yasuro Sugishita, Katsutoshi Goto

    Publicado em 1995
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  15. 15
    Phosphorylated HBO1 at UV irradiated sites is essential for nucleotide excision repair

    Phosphorylated HBO1 at UV irradiated sites is essential for nucleotide excision repair Por Hiroyuki Niida, Ryoichi Matsunuma, Ryo Horiguchi, Chiharu Uchida, Yuka Nakazawa, Akira Motegi, Koji Nishimoto, Satoshi Sakai, Tatsuya Ohhata, Kyoko Kitagawa, Shinichi Moriwaki, Hideo Nishitani, Ayako Ui, Tomoo Ogi, Masatoshi Kitagawa

    Publicado em 2017
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  16. 16
    Activation of cardiac Cdk9 represses PGC-1 and confers a predisposition to heart failure

    Activation of cardiac Cdk9 represses PGC-1 and confers a predisposition to heart failure Por Motoaki Sano, Sam C. Wang, Manabu Shirai, Fernando Scaglia, Min Xie, Satoshi Sakai, Toru Tanaka, Prathit A. Kulkarni, Philip M. Barger, Keith A. Youker, George E. Taffet, Yasuo Hamamori, Lloyd H. Michael, William J. Craigen, Michael Schneider

    Publicado em 2004
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  17. 17
    Changes in donor lymphocyte infusion for relapsed patients post-hematopoietic stem cell transplantation: a 30-year single-center experience

    Changes in donor lymphocyte infusion for relapsed patients post-hematopoietic stem cell transplantation: a 30-year single-center experience Por Yusuke Uchibori, Shuhei Kurosawa, Yuho Najima, Kyoko Haraguchi, Daichi Sadato, Chizuko Hirama, Yasutaka Sadaga, Kaori Kondo, Chika Kato, Satoshi Sakai, Yasuhiro Kambara, Fumihiko Ouchi, Masashi Shimabukuro, Atsushi Jinguji, Naoki Shingai, Takashi Toya, Hiroaki Shimizu, Takeshi Kobayashi, Hironori Harada, Yuka Harada, Yoshiki Okuyama, Noriko Doki

    Publicado em 2025
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