Suchergebnisse - Robling, Alexander G.

Muscle loss and bone loss: master and slave? von Robling, Alexander G.

Volltext Volltext Volltext

Is bone’s response to mechanical signals dominated by muscle forces? von Robling, Alexander G.

Volltext Volltext Volltext

The Interaction of Biological Factors with Mechanical Signals in Bone Adaptation: Recent Developments von Robling, Alexander G.

Volltext Volltext Volltext

The expanding role of Wnt signaling in bone metabolism von Robling, Alexander G.

Volltext Volltext Volltext

Mechanical Signaling for Bone Modeling and Remodeling von Robling, Alexander G., Turner, Charles H.

Volltext Volltext

New Insights into Wnt–Lrp5/6–β-Catenin Signaling in Mechanotransduction von Kang, Kyung Shin, Robling, Alexander G.

Volltext Volltext Volltext

WNT-Mediated Modulation of Bone Metabolism: Implications for WNT Targeting to Treat Extra-Skeletal Disorders von Bullock, Whitney A., Robling, Alexander G.

Volltext Volltext Volltext

The Osteocyte: New Insights von Robling, Alexander G., Bonewald, Lynda F.

Volltext Volltext Volltext

The Wnt pathway: an important control mechanism in bone’s response to mechanical loading von Choi, Roy B., Robling, Alexander G.

Volltext Volltext Volltext

Nmp4/CIZ: Road Block at the Intersection of PTH and Load von Childress, Paul, Robling, Alexander G, Bidwell, Joseph P

Volltext Volltext Volltext

Mechanotransduction in bone tissue: The A214V and G171V mutations in Lrp5 enhance load-induced osteogenesis in a surface-selective manner von Niziolek, Paul J., Warman, Matthew L., Robling, Alexander G.

Volltext Volltext Volltext

Postnatal β-catenin deletion from Dmp1-expressing osteocytes/osteoblasts reduces structural adaptation to loading, but not periosteal load-induced bone formation von Kang, Kyung Shin, Hong, Jung Min, Robling, Alexander G.

Volltext Volltext Volltext

In Vivo Axial Loading of the Mouse Tibia von Melville, Katherine M., Robling, Alexander G., van der Meulen, Marjolein C. H.

Volltext Volltext Volltext

Skeletal Functions of Voltage Sensitive Calcium Channels von Wright, Christian S., Robling, Alexander G., Farach-Carson, Mary C., Thompson, William R.

Volltext Volltext Volltext

Inactivation of Lrp5 in Osteocytes Reduces Young’s Modulus and Responsiveness to the Mechanical Loading von Zhao, Liming, Shim, Joon W., Dodge, Todd R., Robling, Alexander G., Yokota, Hiroki

Volltext Volltext Volltext

Bone Matrix Composition following PTH Treatment Is Not Dependent on Sclerostin Status von Ross, Ryan D., Mashiatulla, Maleeha, Robling, Alexander G., Miller, Lisa M., Sumner, D. Rick

Volltext Volltext Volltext

The emerging role of serotonin (5-hydroxytryptamine) in the skeleton and its mediation of the skeletal effects of low-density lipoprotein receptor-related protein 5 (LRP5) von Warden, Stuart J., Robling, Alexander G., Haney, Elizabeth M., Turner, Charles H., Bliziotes, Michael M.

Volltext Volltext Volltext

Mechanical stimulation and intermittent parathyroid hormone treatment induce disproportional osteogenic, geometric, and biomechanical effects in growing mouse bone von McAteer, Maureen E., Niziolek, Paul J., Ellis, Shana N., Alge, Daniel L., Robling, Alexander G.

Volltext Volltext Volltext

High-bone-mass-producing mutations in the Wnt signaling pathway result in distinct skeletal phenotypes von Niziolek, Paul J., Farmer, Takeisha, Cui, Yajun, Turner, Charles H., Warman, Matthew L., Robling, Alexander G.

Volltext Volltext Volltext

Quick and inexpensive paraffin-embedding method for dynamic bone formation analyses von Porter, Amy, Irwin, Regina, Miller, Josselyn, Horan, Daniel J., Robling, Alexander G., McCabe, Laura R.

Volltext Volltext Volltext