Resultats de la cerca - Rebecca Deasy

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  1. 1
    CRISPR activation screen identifies BCL-2 proteins and B3GNT2 as drivers of cancer resistance to T cell-mediated cytotoxicity

    CRISPR activation screen identifies BCL-2 proteins and B3GNT2 as drivers of cancer resistance to T cell-mediated cytotoxicity per Julia Joung, Paul C. Kirchgatterer, Ankita Singh, Jang H. Cho, Suchita P. Nety, Rebecca C. Larson, Rhiannon K. Macrae, Rebecca Deasy, Yuen‐Yi Tseng, Marcela V. Maus, Feng Zhang

    Publicat 2022
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  2. 2
    A GPX4-dependent cancer cell state underlies the clear-cell morphology and confers sensitivity to ferroptosis

    A GPX4-dependent cancer cell state underlies the clear-cell morphology and confers sensitivity to ferroptosis per Yilong Zou, Michael J. Palte, Amy Deik, Haoxin Li, John K. Eaton, Wenyu Wang, Yuen‐Yi Tseng, Rebecca Deasy, Maria Kost‐Alimova, Vlado Dančík, Elizaveta S. Leshchiner, Vasanthi S. Viswanathan, Sabina Signoretti, Toni K. Choueiri, Jesse S. Boehm, Bridget K. Wagner, John G. Doench, Clary B. Clish, Paul A. Clemons, Stuart L. Schreiber

    Publicat 2019
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  3. 3
    WRN helicase is a synthetic lethal target in microsatellite unstable cancers

    WRN helicase is a synthetic lethal target in microsatellite unstable cancers per Edmond M. Chan, Tsukasa Shibue, James M. McFarland, Benjamin Gaeta, Mahmoud Ghandi, Nancy Dumont, Alfredo González, Justine S. McPartlan, Tianxia Li, Yanxi Zhang, Jie Bin Liu, Jean‐Bernard Lazaro, Peili Gu, Cortt G. Piett, Annie Apffel, Syed O. Ali, Rebecca Deasy, Paula Keskula, Raymond W.S. Ng, Emma A. Roberts, Elizaveta Reznichenko, Lisa Leung, Maria Alimova, Monica Schenone, Mirazul Islam, Yosef E. Maruvka, Yang Liu, Jatin Roper, Srivatsan Raghavan, Marios Giannakis, Yuen‐Yi Tseng, Zachary D. Nagel, Alan D. D’Andrea, David E. Root, Jesse S. Boehm, Gad Getz, Sandy Chang, Todd R. Golub, Aviad Tsherniak, Francisca Vázquez, Adam J. Bass

    Publicat 2019
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  4. 4
    Renal medullary carcinomas depend upon SMARCB1 loss and are sensitive to proteasome inhibition

    Renal medullary carcinomas depend upon SMARCB1 loss and are sensitive to proteasome inhibition per Andrew L. Hong, Yuen‐Yi Tseng, Jeremiah A. Wala, Won‐Jun Kim, Bryan D. Kynnap, Mihir B. Doshi, Guillaume Kugener, Gabriel J. Sandoval, Thomas P. Howard, Ji Li, Xiaoping Yang, Michelle Tillgren, Mahmhoud Ghandi, Abeer Sayeed, Rebecca Deasy, Abigail Ward, Brian McSteen, Katherine Labella, Paula Keskula, Adam Tracy, Cora Connor, Catherine Clinton, Alanna J. Church, Brian D. Crompton, Katherine A. Janeway, Barbara Van Hare, David Sandak, Ole Gjoerup, Pratiti Bandopadhayay, Paul A. Clemons, Stuart L. Schreiber, David E. Root, Prafulla C. Gokhale, Susan Chi, Elizabeth A. Mullen, Charles W.M. Roberts, Cigall Kadoch, Rameen Beroukhim, Keith L. Ligon, Jesse S. Boehm, William C. Hahn

    Publicat 2019
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