Torthaí cuardaigh - Rahn Kollander
- 1 - 2 toradh as 2 á dtaispeáint
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1
Endothelial cell expression of tissue factor in sickle mice is augmented by hypoxia/reoxygenation and inhibited by lovastatin de réir Anna Solovey, Rahn Kollander, Arun S. Shet, Liming Milbauer, Stephana Choong, Angela Panoskaltsis‐Mortari, Bruce R. Blazar, Robert J. Kelm, Robert P. Hebbel
Foilsithe / Cruthaithe 2004Artigo -
2
The HDAC inhibitors trichostatin A and suberoylanilide hydroxamic acid exhibit multiple modalities of benefit for the vascular pathobiology of sickle transgenic mice de réir Robert P. Hebbel, Gregory M. Vercellotti, Betty S. Pace, Anna Solovey, Rahn Kollander, Chine F. Abanonu, Julia Nguyen, Julie V. Vineyard, John D. Belcher, Fuad Abdulla, Shadé Osifuye, John W. Eaton, Robert J. Kelm, Arne Slungaard
Foilsithe / Cruthaithe 2010Artigo
Uirlisí cuardaigh:
Ábhair a bhaineann le hábhar
Biochemistry
Chemistry
Disease
Gene
Hypoxia (environmental)
Immunology
Medicine
Organic chemistry
Oxygen
Pathology
Sickle cell anemia
Biology
Cancer research
Cholesterol
Coagulation
Endocrinology
Endothelium
Genetically modified mouse
Histone
Histone deacetylase
Histone deacetylase inhibitor
Internal medicine
Lovastatin
Pharmacology
Tissue factor
Transgene
Trichostatin A