Resultados da pesquisa - Nobuo Kanazawa

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  1. 1
    Nakajo-Nishimura Syndrome: An Autoinflammatory Disorder Showing Pernio-Like Rashes and Progressive Partial Lipodystrophy

    Nakajo-Nishimura Syndrome: An Autoinflammatory Disorder Showing Pernio-Like Rashes and Progressive Partial Lipodystrophy Por Nobuo Kanazawa

    Publicado em 2012
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  2. 2
    Early-onset sarcoidosis and CARD15 mutations with constitutive nuclear factor- B activation: common genetic etiology with Blau syndrome

    Early-onset sarcoidosis and CARD15 mutations with constitutive nuclear factor- B activation: common genetic etiology with Blau syndrome Por Nobuo Kanazawa

    Publicado em 2004
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  3. 3
    Dendritic Cell Immunoactivating Receptor, a Novel C-type Lectin Immunoreceptor, Acts as an Activating Receptor through Association with Fc Receptor γ Chain

    Dendritic Cell Immunoactivating Receptor, a Novel C-type Lectin Immunoreceptor, Acts as an Activating Receptor through Association with Fc Receptor γ Chain Por Nobuo Kanazawa, Kei Tashiro, Kayo Inaba, Yoshiki Miyachi

    Publicado em 2003
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  4. 4
    Calumenin, a Ca2+-binding Protein Retained in the Endoplasmic Reticulum with a Novel Carboxyl-terminal Sequence, HDEF

    Calumenin, a Ca2+-binding Protein Retained in the Endoplasmic Reticulum with a Novel Carboxyl-terminal Sequence, HDEF Por Daisuke Yabe, Tomoyuki Nakamura, Nobuo Kanazawa, Kei Tashiro, Tasuku Honjo

    Publicado em 1997
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  5. 5
    Efficacy and safety of baricitinib in Japanese patients with autoinflammatory type I interferonopathies (NNS/CANDLE, SAVI, And AGS)

    Efficacy and safety of baricitinib in Japanese patients with autoinflammatory type I interferonopathies (NNS/CANDLE, SAVI, And AGS) Por Nobuo Kanazawa, Taeko Ishii, Yasushi Takita, Atsushi Nishikawa, Ryuta Nishikomori

    Publicado em 2023
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  6. 6
    Presence of a Sporadic Case of Systemic Granulomatosis Syndrome with a CARD15 Mutation

    Presence of a Sporadic Case of Systemic Granulomatosis Syndrome with a CARD15 Mutation Por Nobuo Kanazawa, Satoko Matsushima, Naotomo Kambe, Takao Tachibana, Sonoko Nagai, Yoshiki Miyachi

    Publicado em 2004
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  7. 7
    DCIR Acts as an Inhibitory Receptor Depending on its Immunoreceptor Tyrosine-Based Inhibitory Motif11A preliminary report of these results was presented by the first author at the 61st annual meeting of SID in Chicago in the session “General Immunology”.

    DCIR Acts as an Inhibitory Receptor Depending on its Immunoreceptor Tyrosine-Based Inhibitory Motif11A preliminary report of these results was presented by the first author at the... Por Nobuo Kanazawa, Taku Okazaki, Hiroyuki Nishimura, Kei Tashiro, Kayo Inaba, Yoshiki Miyachi

    Publicado em 2002
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  8. 8
    Frequent microsatellite instability in papillary and solid-type, poorly differentiated adenocarcinomas of the stomach

    Frequent microsatellite instability in papillary and solid-type, poorly differentiated adenocarcinomas of the stomach Por Tomio Arai, Urara Sakurai, Motoji Sawabe, Naoko Honma, Junko Aida, Yasuko Ushio, Nobuo Kanazawa, Kojiro Kuroiwa, Kaiyo Takubo

    Publicado em 2012
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  9. 9
    Fractalkine and macrophage-derived chemokine: T cell-attracting chemokines expressed in T cell area dendritic cells

    Fractalkine and macrophage-derived chemokine: T cell-attracting chemokines expressed in T cell area dendritic cells Por Nobuo Kanazawa, Tomoyuki Nakamura, Kei Tashiro, Masamichi Muramatsu, Kazumasa Morita, Kozo Yoneda, Kayo Inaba, Sadao Imamura, Tasuku Honjo

    Publicado em 1999
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  10. 10
    BCL-6 gene product, a 92- to 98-kD nuclear phosphoprotein, is highly expressed in germinal center B cells and their neoplastic counterparts

    BCL-6 gene product, a 92- to 98-kD nuclear phosphoprotein, is highly expressed in germinal center B cells and their neoplastic counterparts Por Toshiko Onizuka, Masashi Moriyama, Toshiko Yamochi, Toshihiko Kuroda, A Kazama, Nobuo Kanazawa, Kazunari Sato, Takeharu Kato, Hidekazu Ota, Shigeki Mori

    Publicado em 1995
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  11. 11
    The Conduit System Transports Soluble Antigens from the Afferent Lymph to Resident Dendritic Cells in the T Cell Area of the Lymph Node

    The Conduit System Transports Soluble Antigens from the Afferent Lymph to Resident Dendritic Cells in the T Cell Area of the Lymph Node Por Michael Sixt, Nobuo Kanazawa, Manuel Selg, Thomas Samson, G Roos, Dieter P. Reinhardt, Reinhard Pabst, Manfred B. Lutz, Lydia Sorokin

    Publicado em 2005
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  12. 12
    Disease-associated mutations in CIAS1 induce cathepsin B–dependent rapid cell death of human THP-1 monocytic cells

    Disease-associated mutations in CIAS1 induce cathepsin B–dependent rapid cell death of human THP-1 monocytic cells Por Akihiro Fujisawa, Naotomo Kambe, Megumu K. Saito, Ryuta Nishikomori, Hideaki Tanizaki, Nobuo Kanazawa, Souichi Adachi, Toshio Heike, Junji Sagara, Takashi Suda, Tatsutoshi Nakahata, Yoshiki Miyachi

    Publicado em 2006
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  13. 13
    Steady state migratory RelB<sup>+</sup> langerin<sup>+</sup> dermal dendritic cells mediate peripheral induction of antigen‐specific CD4<sup>+</sup>CD25<sup>+</sup>Foxp3<sup>+</sup> regulatory T cells

    Steady state migratory RelB<sup>+</sup> langerin<sup>+</sup> dermal dendritic cells mediate peripheral induction of antigen‐specific CD4<sup>+</sup>CD25<sup>+</sup>Foxp3<sup>+</sup... Por Hiroaki Azukizawa, Anja Döhler, Nobuo Kanazawa, Arnab Nayak, Martin Lipp, Bernard Malissen, Ingo B. Autenrieth, Ichiro Katayama, Marc Riemann, Falk Weih, Friederike Berberich‐Siebelt, Manfred B. Lutz

    Publicado em 2011
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  14. 14
    Role of the NOD2 genotype in the clinical phenotype of Blau syndrome and early‐onset sarcoidosis

    Role of the NOD2 genotype in the clinical phenotype of Blau syndrome and early‐onset sarcoidosis Por Ikuo Okafuji, Ryuta Nishikomori, Nobuo Kanazawa, Naotomo Kambe, Akihiro Fujisawa, Shin Yamazaki, Megumu K. Saito, Takakazu Yoshioka, Tomoki Kawai, Hidemasa Sakai, Hideaki Tanizaki, Toshio Heike, Yoshiki Miyachi, Tatsutoshi Nakahata

    Publicado em 2008
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  15. 15
    Mesenchymal Bmp3b expression maintains skeletal muscle integrity and decreases in age-related sarcopenia

    Mesenchymal Bmp3b expression maintains skeletal muscle integrity and decreases in age-related sarcopenia Por Akiyoshi Uezumi, Madoka Ikemoto‐Uezumi, Heying Zhou, Tamaki Kurosawa, Yuki Yoshimoto, Masashi Nakatani, Keisuke Hitachi, Hisateru Yamaguchi, Shuji Wakatsuki, Toshiyuki Araki, Mitsuhiro Morita, Harumoto Yamada, Masashi Toyoda, Nobuo Kanazawa, Tatsu Nakazawa, Jun Hino, So‐ichiro Fukada, Kunihiro Tsuchida

    Publicado em 2020
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  16. 16
    Prevalence of frailty, cognitive impairment, and sarcopenia in outpatients with cardiometabolic disease in a frailty clinic

    Prevalence of frailty, cognitive impairment, and sarcopenia in outpatients with cardiometabolic disease in a frailty clinic Por Yoshiaki Tamura, Joji Ishikawa, Yoshinori Fujiwara, M. Tanaka, Nobuo Kanazawa, Yuko Chiba, Ai Iizuka, Sho Kaito, Jun Tanaka, Masamitsu Sugie, Takashi Nishimura, Akiko Kanemaru, Keigo Shimoji, Hirohiko Hirano, Ko Furuta, Akihiko Kitamura, Satoshi Seino, Shoji Shinkai, Kazumasa Harada, Shunei Kyo, Hideki Ito, Atsushi Araki

    Publicado em 2018
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  17. 17
    ISSAID/EMQN Best Practice Guidelines for the Genetic Diagnosis of Monogenic Autoinflammatory Diseases in the Next-Generation Sequencing Era

    ISSAID/EMQN Best Practice Guidelines for the Genetic Diagnosis of Monogenic Autoinflammatory Diseases in the Next-Generation Sequencing Era Por Yael Shinar, Isabella Ceccherini, Dorota Rowczenio, Ivona Aksentijevich, Juan I. Aróstegui, Eldad Ben‐Chetrit, Guilaine Boursier, Marco Gattorno, Hasmik Hayrapetyan, Hiroaki Ida, Nobuo Kanazawa, Helen J. Lachmann, Anna Mensa‐Vilaró, Ryuta Nishikomori, Christian Oberkanins, Laura Obici, Osamu Ohara, Seza Özen, Tamara Sarkisian, Katie Sheils, Nicola Wolstenholme, Evelien Zonneveld‐Huijssoon, Marielle E van Gijn, Isabelle Touitou

    Publicado em 2020
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  18. 18
    Proteasome assembly defect due to a proteasome subunit beta type 8 (PSMB8) mutation causes the autoinflammatory disorder, Nakajo-Nishimura syndrome

    Proteasome assembly defect due to a proteasome subunit beta type 8 (PSMB8) mutation causes the autoinflammatory disorder, Nakajo-Nishimura syndrome Por Kazuhiko Arima, Akira Kinoshita, Hiroyuki Mishima, Nobuo Kanazawa, Takeumi Kaneko, Tsunehiro Mizushima, Kunihiro Ichinose, Hideki Nakamura, Akira Tsujino, Atsushi Kawakami, Masahiro Matsunaka, Shimpei Kasagi, Seiji Kawano, Shunichi Kumagai, Koichiro Ohmura, Tsuneyo Mimori, Makito Hirano, Satoshi Ueno, Keiko Tanaka, Masami Tanaka, Itaru Toyoshima, Hirotoshi Sugino, A. Yamakawa, Keiji Tanaka, Norio Niikawa, Fukumi Furukawa, Shigeo Murata, Katsumi Eguchi, Hiroaki Ida, Koh-ichiro Yoshiura

    Publicado em 2011
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