检索结果 - Navaraj, Arunasalam

Repression of c-Myc and inhibition of G(1) exit in cells conditionally overexpressing p300 that is not dependent on its histone acetyltransferase activity 由 Baluchamy, Sudhakar, Rajabi, Hasan N., Thimmapaya, Rama, Navaraj, Arunasalam, Thimmapaya, Bayar

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Reduced cell death, invasive and angiogenic features conferred by BRCA1-deficiency in mammary epithelial cells transformed with H-Ras 由 Navaraj, Arunasalam, Finnberg, Niklas, Dicker, David T., Yang, Wensheng, Matthew, Elizabeth M., El-Deiry, Wafik S.

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Targeting of Chk2 as a countermeasure to dose-limiting toxicity triggered by topoisomerase-II (TOP2) poisons 由 Gokare, Prashanth, Navaraj, Arunasalam, Zhang, Shengliang, Motoyama, Noboru, Sung, Shen-Shu, Finnberg, Niklas K.

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AMG-232 sensitizes high MDM2-expressing tumor cells to T-cell-mediated killing 由 Sahin, Ilyas, Zhang, Shengliang, Navaraj, Arunasalam, Zhou, Lanlan, Dizon, Don, Safran, Howard, El-Deiry, Wafik S.

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Correction: AMG-232 sensitizes high MDM2-expressing tumor cells to T-cell-mediated killing 由 Sahin, Ilyas, Zhang, Shengliang, Navaraj, Arunasalam, Zhou, Lanlan, Dizon, Don, Safran, Howard, El-Deiry, Wafik S.

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The p53 target Plk2 interacts with TSC proteins impacting mTOR signaling, tumor growth and chemosensitivity under hypoxic conditions 由 Matthew, Elizabeth M., Hart, Lori S., Astrinidis, Aristotelis, Navaraj, Arunasalam, Dolloff, Nathan G., Dicker, David T., Henske, Elizabeth P., El-Deiry, Wafik S.

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Agonists of the TRAIL death receptor DR5 sensitize intestinal stem cells to chemotherapy-induced cell death and trigger gastrointestinal toxicity 由 Finnberg, Niklas K., Gokare, Prashanth, Navaraj, Arunasalam, Lang Kuhs, Krystle A., Cerniglia, George, Yagita, Hideo, Takeda, Kazuyoshi, Motoyama, Noboru, El-Deiry, Wafik S.

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Combination of ONC201 and TLY012 induces selective, synergistic apoptosis in vitro and significantly delays PDAC xenograft growth in vivo 由 Jhaveri, Aakash V., Zhou, Lanlan, Ralff, Marie D., Lee, Young S., Navaraj, Arunasalam, Carneiro, Benedito A., Safran, Howard, Prabhu, Varun V., Ross, Eric A., Lee, Seulki, El-Deiry, Wafik S.

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Natural Killer cell activation, reduced ACE2, TMPRSS2, cytokines G-CSF, M-CSF and SARS-CoV-2-S pseudovirus infectivity by MEK inhibitor treatment of human cells 由 Zhou, Lanlan, Huntington, Kelsey, Zhang, Shengliang, Carlsen, Lindsey, So, Eui-Young, Parker, Cassandra, Sahin, Ilyas, Safran, Howard, Kamle, Suchitra, Lee, Chang-Min, Lee, Chun Geun, Elias, Jack A., Campbell, Kerry S., Naik, Mandar T., Atwood, Walter J., Youssef, Emile, Pachter, Jonathan A., Navaraj, Arunasalam, Seyhan, Attila A., Liang, Olin, El-Deiry, Wafik S.

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MEK inhibitors reduce cellular expression of ACE2, pERK, pRb while stimulating NK-mediated cytotoxicity and attenuating inflammatory cytokines relevant to SARS-CoV-2 infection 由 Zhou, Lanlan, Huntington, Kelsey, Zhang, Shengliang, Carlsen, Lindsey, So, Eui-Young, Parker, Cassandra, Sahin, Ilyas, Safran, Howard, Kamle, Suchitra, Lee, Chang-Min, Geun Lee, Chun, A. Elias, Jack, S. Campbell, Kerry, T. Naik, Mandar, J. Atwood, Walter, Youssef, Emile, A. Pachter, Jonathan, Navaraj, Arunasalam, A. Seyhan, Attila, Liang, Olin, El-Deiry, Wafik S.

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