检索结果 - Murphy, Nathaniel

Advancements in the use of gene therapy for cardiac arrhythmia 由 Murphy, Nathaniel P., Lubbers, Ellen R., Mohler, Peter J.

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Advancing our understanding of AnkRD1 in cardiac development and disease 由 Murphy, Nathaniel P, Lubbers, Ellen R, Mohler, Peter J

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Aberrant expression of a non-muscle RBFOX2 isoform triggers cardiac conduction defects in myotonic dystrophy 由 Misra, Chaitali, Bangru, Sushant, Lin, Feikai, Lam, Kin, Koenig, Sara N., Lubbers, Ellen R., Hedhli, Jamila, Murphy, Nathaniel P., Parker, Darren J., Dobrucki, Lawrence W., Cooper, Thomas A., Tajkhorshid, Emad, Mohler, Peter J., Kalsotra, Auinash

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Molecular Mechanisms Underlying Cardiac Protein Phosphatase 2A Regulation in Heart 由 DeGrande, Sean T., Little, Sean C., Nixon, Derek J., Wright, Patrick, Snyder, Jedidiah, Dun, Wen, Murphy, Nathaniel, Kilic, Ahmet, Higgins, Robert, Binkley, Philip F., Boyden, Penelope A., Carnes, Cynthia A., Anderson, Mark E., Hund, Thomas J., Mohler, Peter J.

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A Novel Variant in the ANK2 Membrane-Binding Domain is Associated with Ankyrin-B Syndrome and Structural Heart Disease in a First Nations Population with a High Rate of Long QT Syn... 由 Swayne, Leigh Anne, Murphy, Nathaniel P., Asuri, Sirisha, Chen, Lena, Xu, Xiaoxue, McIntosh, Sarah, Wang, Chao, Lancione, Peter J., Roberts, Jason D., Kerr, Charles, Sanatani, Shubhayan, Sherwin, Elizabeth, Kline, Crystal F., Zhang, Mingjie, Mohler, Peter J., Arbour, Laura T.

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Protein Phosphatase 2A Regulates Cardiac Na(+) Channels 由 Refaey, Mona El, Musa, Hassan, Murphy, Nathaniel P., Lubbers, Ellen R., Skaf, Michel, Han, Mei, Cavus, Omer, Koenig, Sara N., Wallace, Michael J., Gratz, Daniel, Bradley, Elisa, Alsina, Katherina M., Wehrens, Xander H.T., Hund, Thomas J., Mohler, Peter J.

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Novel Mechanistic Roles for Ankyrin-G in Cardiac Remodeling and Heart Failure 由 Makara, Michael A., Curran, Jerry, Lubbers, Ellen R., Murphy, Nathaniel P., Little, Sean C., Musa, Hassan, Smith, Sakima A., Unudurthi, Sathya D., Rajaram, Murugesan V.S., Janssen, Paul M.L., Boyden, Penelope A., Bradley, Elisa A., Hund, Thomas J., Mohler, Peter J.

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Altered Expression of Zonula occludens-1 Affects Cardiac Na(+) Channels and Increases Susceptibility to Ventricular Arrhythmias 由 El Refaey, Mona, Coles, Sara, Musa, Hassan, Stevens, Tyler L., Wallace, Michael J., Murphy, Nathaniel P., Antwi-Boasiako, Steve, Young, Lindsay J., Manring, Heather R., Curran, Jerry, Makara, Michael A., Sas, Kelli, Han, Mei, Koenig, Sara N., Skaf, Michel, Kline, Crystal F., Janssen, Paul M. L., Accornero, Federica, Borzok, Maegen A., Mohler, Peter J.

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Defining new mechanistic roles for αII spectrin in cardiac function 由 Lubbers, Ellen R., Murphy, Nathaniel P., Musa, Hassan, Huang, Claire Yu-Mei, Gupta, Rohan, Price, Morgan V., Han, Mei, Daoud, Georges, Gratz, Daniel, El Refaey, Mona, Xu, Xianyao, Hoeflinger, Nicole K., Friel, Emma L., Lancione, Peter, Wallace, Michael J., Cavus, Omer, Simmons, Samantha L., Williams, Jordan L., Skaf, Michel, Koenig, Sara N., Janssen, Paul M. L., Rasband, Matthew N., Hund, Thomas J., Mohler, Peter J.

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Correction: Defining new mechanistic roles for α II spectrin in cardiac function. 由 Lubbers, Ellen R., Murphy, Nathaniel P., Musa, Hassan, Huang, Claire Yu-Mei, Gupta, Rohan, Price, Morgan V., Han, Mei, Daoud, Georges E., Gratz, Daniel, El Refaey, Mona, Xu, Xianyao, Hoeflinger, Nicole K., Friel, Emma L., Lancione, Peter, Wallace, Michael J., Cavus, Omer, Simmons, Samantha L., Williams, Jordan L., Skaf, Michel, Koenig, Sara N., Janssen, Paul M. L., Rasband, Matthew N., Hund, Thomas J., Mohler, Peter J.

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Inherited Variants in SCARB1 Cause Severe Early-Onset Coronary Artery Disease 由 Koenig, Sara N., Sucharski, Holly C., Jose, Elizabeth M., Dudley, Emma K., Madiai, Francesca, Cavus, Omer, Argall, Aaron D., Williams, Jordan L., Murphy, Nathaniel P., Keith, Caullin B. R., Refaey, Mona El, Gumina, Richard J., Boudoulas, Konstantinos D., Milks, M. Wesley, Sofowora, Gbemiga, Smith, Sakima A., Hund, Thomas J., Wright, Nathan T., Bradley, Elisa A., Zareba, Karolina M., Wold, Loren E., Mazzaferri, Ernest L., Mohler, Peter J.

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Ankyrin-B dysfunction predisposes to arrhythmogenic cardiomyopathy and is amenable to therapy 由 Roberts, Jason D., Murphy, Nathaniel P., Hamilton, Robert M., Lubbers, Ellen R., James, Cynthia A., Kline, Crystal F., Gollob, Michael H., Krahn, Andrew D., Sturm, Amy C., Musa, Hassan, El-Refaey, Mona, Koenig, Sara, Aneq, Meriam Åström, Hoorntje, Edgar T., Graw, Sharon L., Davies, Robert W., Rafiq, Muhammad Arshad, Koopmann, Tamara T., Aafaqi, Shabana, Fatah, Meena, Chiasson, David A., Taylor, Matthew R.G., Simmons, Samantha L., Han, Mei, van Opbergen, Chantal J.M., Wold, Loren E., Sinagra, Gianfranco, Mittal, Kirti, Tichnell, Crystal, Murray, Brittney, Codima, Alberto, Nazer, Babak, Nguyen, Duy T., Marcus, Frank I., Sobriera, Nara, Lodder, Elisabeth M., van den Berg, Maarten P., Spears, Danna A., Robinson, John F., Ursell, Philip C., Green, Anna K., Skanes, Allan C., Tang, Anthony S., Gardner, Martin J., Hegele, Robert A., van Veen, Toon A.B., Wilde, Arthur A.M., Healey, Jeff S., Janssen, Paul M.L., Mestroni, Luisa, van Tintelen, J. Peter, Calkins, Hugh, Judge, Daniel P., Hund, Thomas J., Scheinman, Melvin M., Mohler, Peter J.

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