Hakutulokset - Masayuki Sakiyama
- Näytetään 1 - 12 yhteensä 12 tuloksesta
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1
NRF2 Is a Key Target for Prevention of Noise-Induced Hearing Loss by Reducing Oxidative Damage of Cochlea Tekijä Yohei Honkura, Hirotaka Matsuo, Shohei Murakami, Masayuki Sakiyama, Kunio Mizutari, Akihiro Shiotani, Masayuki Yamamoto, Ichiro Morita, Nariyoshi Shinomiya, Tetsuaki Kawase, Yukio Katori, Hozumi Motohashi
Julkaistu 2016Artigo -
2
The effects of URAT1/SLC22A12 nonfunctional variants,R90H and W258X, on serum uric acid levels and gout/hyperuricemia progression Tekijä Masayuki Sakiyama, Hirotaka Matsuo, Seiko Shimizu, Hiroshi Nakashima, Takahiro Nakamura, Akiyoshi Nakayama, Toshihide Higashino, Mariko Naito, Shino Suma, Asahi Hishida, Takahiro Satoh, Yutaka Sakurai, Tappei Takada, Kimiyoshi Ichida, Hiroshi Ooyama, Toru Shimizu, Nariyoshi Shinomiya
Julkaistu 2016Artigo -
3
Identification of ABCG2 as an Exporter of Uremic Toxin Indoxyl Sulfate in Mice and as a Crucial Factor Influencing CKD Progression Tekijä Tappei Takada, Takehito Yamamoto, Hirotaka Matsuo, Jiang Tan, Keiko Ooyama, Masayuki Sakiyama, Hiroshi Miyata, Yoshihide Yamanashi, Yu Toyoda, Toshihide Higashino, Akiyoshi Nakayama, Akio Nakashima, Nariyoshi Shinomiya, Kimiyoshi Ichida, Hiroshi Ooyama, Shin Fujimori, Hiroshi Suzuki
Julkaistu 2018Artigo -
4
Hyperuricemia in acute gastroenteritis is caused by decreased urate excretion via ABCG2 Tekijä Hirotaka Matsuo, Tomoyuki Tsunoda, Keiko Ooyama, Masayuki Sakiyama, Tsuyoshi Sogo, Tappei Takada, Akio Nakashima, Akiyoshi Nakayama, Makoto Kawaguchi, Toshihide Higashino, Kenji Wakai, Hiroshi Ooyama, Ryota Hokari, Hiroshi Suzuki, Kimiyoshi Ichida, Inui Ayano, Shin Fujimori, Nariyoshi Shinomiya
Julkaistu 2016Artigo -
5
A common missense variant of monocarboxylate transporter 9 (MCT9/SLC16A9) gene is associated with renal overload gout, but not with all gout susceptibility Tekijä Akiyoshi Nakayama, Hirotaka Matsuo, Takuya Shimizu, Hiraku Ogata, Yuzo Takada, Hiroshi Nakashima, Takahiro Nakamura, Seiko Shimizu, Toshinori Chiba, Masayuki Sakiyama, Chisaki Ushiyama, Tappei Takada, Katsuhisa Inoue, Sayo Kawai, Asahi Hishida, Kenji Wakai, Nobuyuki Hamajima, Kimiyoshi Ichida, Yutaka Sakurai, Yukio Kato, Toru Shimizu, Nariyoshi Shinomiya
Julkaistu 2013Artigo -
6
Common dysfunctional variants of ABCG2 have stronger impact on hyperuricemia progression than typical environmental risk factors Tekijä Akiyoshi Nakayama, Hirotaka Matsuo, Hirofumi Nakaoka, Takahiro Nakamura, Hiroshi Nakashima, Yuzo Takada, Yuji Oikawa, Tappei Takada, Masayuki Sakiyama, Seiko Shimizu, Yusuke Kawamura, Toshinori Chiba, Junko Abe, Kenji Wakai, Sayo Kawai, Rieko Okada, Takashi Tamura, Yuka Shichijo, A Akashi, Hiroshi Suzuki, Tatsuo Hosoya, Yutaka Sakurai, Kimiyoshi Ichida, Nariyoshi Shinomiya
Julkaistu 2014Artigo -
7
ABCG2 dysfunction causes hyperuricemia due to both renal urate underexcretion and renal urate overload Tekijä Hirotaka Matsuo, Akiyoshi Nakayama, Masayuki Sakiyama, Toshinori Chiba, Seiko Shimizu, Yusuke Kawamura, Hiroshi Nakashima, Takahiro Nakamura, Yuzo Takada, Yuji Oikawa, Tappei Takada, Hirofumi Nakaoka, Junko Abe, Hiroki Inoue, Kenji Wakai, Sayo Kawai, Guang Yin, Hiroko Nakagawa, Toshimitsu Ito, Kazuki Niwa, Ken Yamamoto, Yutaka Sakurai, Hiroshi Suzuki, Tatsuo Hosoya, Kimiyoshi Ichida, Toru Shimizu, Nariyoshi Shinomiya
Julkaistu 2014Artigo -
8
Common dysfunctional variants in ABCG2 are a major cause of early-onset gout Tekijä Hirotaka Matsuo, Kimiyoshi Ichida, Tappei Takada, Akiyoshi Nakayama, Hiroshi Nakashima, Takahiro Nakamura, Yusuke Kawamura, Yuzo Takada, Ken Yamamoto, Hiroki Inoue, Yuji Oikawa, Mariko Naito, Asahi Hishida, Kenji Wakai, Chisa Okada, Seiko Shimizu, Masayuki Sakiyama, Toshinori Chiba, Hiraku Ogata, Kazuki Niwa, Makoto Hosoyamada, Atsuyoshi Mori, Nobuyuki Hamajima, Hiroshi Suzuki, Yoshikatsu Kanai, Yutaka Sakurai, Tatsuo Hosoya, Toru Shimizu, Nariyoshi Shinomiya
Julkaistu 2013Artigo -
9
Multiple common and rare variants of<i>ABCG2</i>cause gout Tekijä Toshihide Higashino, Tappei Takada, Hirofumi Nakaoka, Yu Toyoda, Blanka Stibůrková, Hiroshi Miyata, Yuki Ikebuchi, Hiroshi Nakashima, Seiko Shimizu, Makoto Kawaguchi, Masayuki Sakiyama, Akiyoshi Nakayama, A Akashi, Yuki Tanahashi, Yusuke Kawamura, Takahiro Nakamura, Kenji Wakai, Rieko Okada, Ken Yamamoto, Kazuyoshi Hosomichi, Tatsuo Hosoya, Kimiyoshi Ichida, Hiroshi Ooyama, Hiroshi Suzuki, Ituro Inoue, Tony R. Merriman, Nariyoshi Shinomiya, Hirotaka Matsuo
Julkaistu 2017Artigo -
10
Genome-wide association study of clinically defined gout identifies multiple risk loci and its association with clinical subtypes Tekijä Hirotaka Matsuo, Ken Yamamoto, Hirofumi Nakaoka, Akiyoshi Nakayama, Masayuki Sakiyama, Toshinori Chiba, Atsushi Takahashi, Takahiro Nakamura, Hiroshi Nakashima, Yuzo Takada, Inaho Danjoh, Seiko Shimizu, Junko Abe, Yusuke Kawamura, Sho Terashige, Hiraku Ogata, Seishiro Tatsukawa, Guang Yin, Rieko Okada, M. Emi, Mariko Naito, Atsumi Tokumasu, Hiroyuki Onoue, Keiichi Iwaya, Toshimitsu Ito, Tappei Takada, Katsuhisa Inoue, Yukio Kato, Yukio Nakamura, Yutaka Sakurai, Hiroshi Suzuki, Yoshikatsu Kanai, Tatsuo Hosoya, Nobuyuki Hamajima, Ituro Inoue, Michiaki Kubo, Kimiyoshi Ichida, Hiroshi Ooyama, Toru Shimizu, Nariyoshi Shinomiya
Julkaistu 2015Artigo -
11
GWAS of clinically defined gout and subtypes identifies multiple susceptibility loci that include urate transporter genes Tekijä Akiyoshi Nakayama, Hirofumi Nakaoka, Ken Yamamoto, Masayuki Sakiyama, Amara Shaukat, Yu Toyoda, Yukinori Okada, Yoichiro Kamatani, Takahiro Nakamura, Tappei Takada, Katsuhisa Inoue, Tomoya Yasujima, Hiroaki Yuasa, Yuko Shirahama, Hiroshi Nakashima, Seiko Shimizu, Toshihide Higashino, Yusuke Kawamura, Hiraku Ogata, Makoto Kawaguchi, Yasuyuki Ohkawa, Inaho Danjoh, Atsumi Tokumasu, Keiko Ooyama, Toshimitsu Ito, Takaaki Kondo, Kenji Wakai, Blanka Stibůrková, Karel Pavelká, Lisa K. Stamp, Nicola Dalbeth, Yutaka Sakurai, Hiroshi Suzuki, Makoto Hosoyamada, Shin Fujimori, Takashi Yokoo, Tatsuo Hosoya, Ituro Inoue, Atsushi Takahashi, Michiaki Kubo, Hiroshi Ooyama, Toru Shimizu, Kimiyoshi Ichida, Nariyoshi Shinomiya, Tony R. Merriman, Hirotaka Matsuo
Julkaistu 2016Revisão -
12
Genome-wide association study revealed novel loci which aggravate asymptomatic hyperuricaemia into gout Tekijä Yusuke Kawamura, Hirofumi Nakaoka, Akiyoshi Nakayama, Yukinori Okada, Ken Yamamoto, Toshihide Higashino, Masayuki Sakiyama, Toru Shimizu, Hiroshi Ooyama, Keiko Ooyama, Mitsuo Nagase, Yuji Hidaka, Yuko Shirahama, Kazuyoshi Hosomichi, Yuichiro Nishida, Ippei Shimoshikiryo, Asahi Hishida, Sakurako Katsuura‐Kamano, Seiko Shimizu, Makoto Kawaguchi, Hirokazu Uemura, Rie Ibusuki, Megumi Hara, Mariko Naito, Mikiya Takao, Mayuko Nakajima, Satoko Iwasawa, Hiroshi Nakashima, Keizo Ohnaka, Takahiro Nakamura, Blanka Stibůrková, Tony R. Merriman, Masahiro Nakatochi, Sahoko Ichihara, Mitsuhiro Yokota, Tappei Takada, Tatsuya Saitoh, Yoichiro Kamatani, Atsushi Takahashi, Kokichi Arisawa, Toshiro Takezaki, Keitaro Tanaka, Kenji Wakai, Michiaki Kubo, Tatsuo Hosoya, Kimiyoshi Ichida, Ituro Inoue, Nariyoshi Shinomiya, Hirotaka Matsuo
Julkaistu 2019Revisão
Työkalut:
Liittyvät aiheet
Internal medicine
Medicine
Biology
Gout
Gene
Uric acid
Genetics
Hyperuricemia
Endocrinology
Transporter
ATP-binding cassette transporter
Abcg2
Genome-wide association study
Genotype
Single-nucleotide polymorphism
Biochemistry
Bioinformatics
Genetic association
Gastroenterology
Audiology
Body mass index
Botany
Case-control study
Chemistry
Cochlea
Cohort
Computational biology
Dysfunctional family
Environmental health
Excretion