Rezultati - Mark W. Russell

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  1. 1
    KVLQT1 mutations in three families with familial or sporadic long QT syndrome

    KVLQT1 mutations in three families with familial or sporadic long QT syndrome od Mark W. Russell

    Izdano 1996
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  2. 2
    Obscurin Depletion Impairs Organization of Skeletal Muscle in Developing Zebrafish Embryos

    Obscurin Depletion Impairs Organization of Skeletal Muscle in Developing Zebrafish Embryos od Maide Ö. Raeker, Mark W. Russell

    Izdano 2011
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  3. 3
    Interstitial fluid flow and cyclic strain differentially regulate cardiac fibroblast activation via AT1R and TGF-β1

    Interstitial fluid flow and cyclic strain differentially regulate cardiac fibroblast activation via AT1R and TGF-β1 od Peter A. Galie, Mark W. Russell, Margaret V. Westfall, Jan P. Stegemann

    Izdano 2011
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  4. 4
    Advances in the Understanding of the Genetic Determinants of Congenital Heart Disease and Their Impact on Clinical Outcomes

    Advances in the Understanding of the Genetic Determinants of Congenital Heart Disease and Their Impact on Clinical Outcomes od Mark W. Russell, Wendy K. Chung, Jonathan R. Kaltman, Thomas A. Miller

    Izdano 2018
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  5. 5
    Cardiomyocyte-Specific Knockout and Agonist of Peroxisome Proliferator–Activated Receptor-γ Both Induce Cardiac Hypertrophy in Mice

    Cardiomyocyte-Specific Knockout and Agonist of Peroxisome Proliferator–Activated Receptor-γ Both Induce Cardiac Hypertrophy in Mice od Sheng Zhong Duan, Christine Y. Ivashchenko, Mark W. Russell, David S. Milstone, Richard M. Mortensen

    Izdano 2005
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  6. 6
    Characterization of a Lamellocyte Transcriptional Enhancer Located within the misshapen Gene of Drosophila melanogaster

    Characterization of a Lamellocyte Transcriptional Enhancer Located within the misshapen Gene of Drosophila melanogaster od Tsuyoshi Tokusumi, Richard Paul Sorrentino, Mark W. Russell, Roberto Ferrarese, Shubha Govind, Robert A. Schulz

    Izdano 2009
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  7. 7
    Obscurin-like 1, OBSL1, is a novel cytoskeletal protein related to obscurin

    Obscurin-like 1, OBSL1, is a novel cytoskeletal protein related to obscurin od Sarah Geisler, Dustin Robinson, Maria Hauringa, Maide Ö. Raeker, Andrei B. Borisov, Margaret V. Westfall, Mark W. Russell

    Izdano 2007
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  8. 8
    Functional analysis of candidate genes in 2q13 deletion syndrome implicates FBLN7 and TMEM87B deficiency in congenital heart defects and FBLN7 in craniofacial malformations

    Functional analysis of candidate genes in 2q13 deletion syndrome implicates FBLN7 and TMEM87B deficiency in congenital heart defects and FBLN7 in craniofacial malformations od Mark W. Russell, Maide Ö. Raeker, Sarah Geisler, Peedikayil E. Thomas, Tracy Simmons, John Bernat, Thor Thorsson, Jeffrey W. Innis

    Izdano 2014
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  9. 9
    Genotype-Dependent and -Independent Calcium Signaling Dysregulation in Human Hypertrophic Cardiomyopathy

    Genotype-Dependent and -Independent Calcium Signaling Dysregulation in Human Hypertrophic Cardiomyopathy od Adam S. Helms, Francisco Alvarado, Jaime Yob, Vi T. Tang, Francis D. Pagani, Mark W. Russell, Héctor H. Valdivia, Sharlene M. Day

    Izdano 2016
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  10. 10
    Obscurin modulates the assembly and organization of sarcomeres and the sarcoplasmic reticulum

    Obscurin modulates the assembly and organization of sarcomeres and the sarcoplasmic reticulum od Aikaterini Kontrogianni‐Konstantopoulos, Dawn H. Catino, John Strong, Sarah Sutter, Andrei B. Borisov, David W. Pumplin, Mark W. Russell, Robert J. Bloch

    Izdano 2006
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  11. 11
    Complement‐induced activation of the cardiac NLRP3 inflammasome in sepsis

    Complement‐induced activation of the cardiac NLRP3 inflammasome in sepsis od Miriam Kalbitz, Fatemeh Fattahi, Jamison Grailer, Lawrence Jajou, Elizabeth A. Malan, Firas S. Zetoune, Markus Huber‐Lang, Mark W. Russell, Peter A. Ward

    Izdano 2016
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  12. 12
    Complement‐induced activation of MAPKs and Akt during sepsis: role in cardiac dysfunction

    Complement‐induced activation of MAPKs and Akt during sepsis: role in cardiac dysfunction od Fatemeh Fattahi, Miriam Kalbitz, Elizabeth A. Malan, Elizabeth Abe, Lawrence Jajou, Markus Huber‐Lang, Markus Bosmann, Mark W. Russell, Firas S. Zetoune, Peter A. Ward

    Izdano 2017
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  13. 13
    Pleiotropic Phenotype of a Genomic Knock-In of an RGS-Insensitive G184S Gnai2 Allele

    Pleiotropic Phenotype of a Genomic Knock-In of an RGS-Insensitive G184S Gnai2 Allele od Xinyan Huang, Ying Fu, Raelene A. Charbeneau, Thomas L. Saunders, Douglas K Taylor, Kurt D. Hankenson, Mark W. Russell, Louis G. D’Alecy, Richard R. Neubig

    Izdano 2006
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  14. 14
    Fontan-Associated Protein-Losing Enteropathy and Plastic Bronchitis

    Fontan-Associated Protein-Losing Enteropathy and Plastic Bronchitis od Kurt R. Schumacher, Kathleen A. Stringer, Janet E. Donohue, Sunkyung Yu, Ashley Shaver, Regine L. Caruthers, Brian J. Zikmund‐Fisher, Carlen G. Fifer, Caren S. Goldberg, Mark W. Russell

    Izdano 2015
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  15. 15
    Deficient cMyBP-C protein expression during cardiomyocyte differentiation underlies human hypertrophic cardiomyopathy cellular phenotypes in disease specific human ES cell derived cardiomyocytes

    Deficient cMyBP-C protein expression during cardiomyocyte differentiation underlies human hypertrophic cardiomyopathy cellular phenotypes in disease specific human ES cell derived... od André Monteiro da Rocha, Guadalupe Guerrero‐Serna, Adam S. Helms, Carly Luzod, Sergey V. Mironov, Mark W. Russell, José Jalife, Sharlene M. Day, Gary D. Smith, Todd J. Herron

    Izdano 2016
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  16. 16
    Enhancing Literacy in Cardiovascular Genetics: A Scientific Statement From the American Heart Association

    Enhancing Literacy in Cardiovascular Genetics: A Scientific Statement From the American Heart Association od Seema Mital, Kiran Musunuru, Vidu Garg, Mark W. Russell, David E. Lanfear, Rajat Gupta, Kathleen T. Hickey, Michael J. Ackerman, Marco Pérez, Dan M. Roden, Daniel Woo, Caroline S. Fox, Stephanie M. Ware

    Izdano 2016
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  17. 17
    Genetic Basis for Congenital Heart Disease: Revisited: A Scientific Statement From the American Heart Association

    Genetic Basis for Congenital Heart Disease: Revisited: A Scientific Statement From the American Heart Association od Mary Ella Pierpont, Martina Brueckner, Wendy K. Chung, Vidu Garg, Ronald V. Lacro, Amy L. McGuire, Seema Mital, James R. Priest, William T. Pu, Amy E. Roberts, Stephanie M. Ware, Bruce D. Gelb, Mark W. Russell

    Izdano 2018
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  18. 18
    In vivo, Pikfyve generates PI(3,5)P <sub>2</sub> , which serves as both a signaling lipid and the major precursor for PI5P

    In vivo, Pikfyve generates PI(3,5)P <sub>2</sub> , which serves as both a signaling lipid and the major precursor for PI5P od Sergey N. Zolov, Dave Bridges, Yanling Zhang, Wei-Wei Lee, Ellen Riehle, Rakesh Kumar Verma, Guy M. Lenk, Kimber Converso‐Baran, Thomas Weide, Roger L. Albin, Alan R. Saltiel, Miriam H. Meisler, Mark W. Russell, Lois S. Weisman

    Izdano 2012
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  19. 19
    Sarcomere Mutation-Specific Expression Patterns in Human Hypertrophic Cardiomyopathy

    Sarcomere Mutation-Specific Expression Patterns in Human Hypertrophic Cardiomyopathy od Adam S. Helms, Frank M. Davis, David Coleman, Sarah N. Bartolone, Amelia A. Glazier, Francis D. Pagani, Jaime Yob, Sakthivel Sadayappan, Ellen Bøtker Pedersen, Robert Lyons, Margaret V. Westfall, Richard W. Jones, Mark W. Russell, Sharlene M. Day

    Izdano 2014
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  20. 20
    Role of extracellular histones in the cardiomyopathy of sepsis

    Role of extracellular histones in the cardiomyopathy of sepsis od Miriam Kalbitz, Jamison Grailer, Fatemeh Fattahi, Lawrence Jajou, Todd J. Herron, Katherine Campbell, Firas S. Zetoune, Markus Bosmann, J. Vidya Sarma, Markus Huber‐Lang, Florian Gebhard, Randall Loaiza, Héctor H. Valdivia, José Jalife, Mark W. Russell, Peter A. Ward

    Izdano 2015
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