Ohcanbohtosat - Marcin Dobaczewski

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  1. 1
    Chemokines and cardiac fibrosis

    Chemokines and cardiac fibrosis Dahkki Marcin Dobaczewski, Nikolaos G. Frangogiannis

    Almmustuhtton 2009
    Viečča ollesdeavstta
    Revisão
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  2. 2
    TGF-β signaling in fibrosis

    TGF-β signaling in fibrosis Dahkki Anna Biernacka, Marcin Dobaczewski, Nikolaos G. Frangogiannis

    Almmustuhtton 2011
    Viečča ollesdeavstta
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  3. 3
    Transforming growth factor (TGF)-β signaling in cardiac remodeling

    Transforming growth factor (TGF)-β signaling in cardiac remodeling Dahkki Marcin Dobaczewski, Wei Chen, Nikolaos G. Frangogiannis

    Almmustuhtton 2010
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  4. 4
    The extracellular matrix as a modulator of the inflammatory and reparative response following myocardial infarction

    The extracellular matrix as a modulator of the inflammatory and reparative response following myocardial infarction Dahkki Marcin Dobaczewski, Carlos Gonzalez‐Quesada, Nikolaos G. Frangogiannis

    Almmustuhtton 2009
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  5. 5
    The Extracellular Matrix Modulates Fibroblast Phenotype and Function in the Infarcted Myocardium

    The Extracellular Matrix Modulates Fibroblast Phenotype and Function in the Infarcted Myocardium Dahkki Marcin Dobaczewski, Judith J. de Haan, Nikolaos G. Frangogiannis

    Almmustuhtton 2012
    Viečča ollesdeavstta
    Revisão
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  6. 6
    CCR5 Signaling Suppresses Inflammation and Reduces Adverse Remodeling of the Infarcted Heart, Mediating Recruitment of Regulatory T Cells

    CCR5 Signaling Suppresses Inflammation and Reduces Adverse Remodeling of the Infarcted Heart, Mediating Recruitment of Regulatory T Cells Dahkki Marcin Dobaczewski, Ying Xia, Marcin Bujak, Carlos Gonzalez‐Quesada, Nikolaos G. Frangogiannis

    Almmustuhtton 2010
    Viečča ollesdeavstta
    Artigo
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  7. 7
    Regulatory T cells are recruited in the infarcted mouse myocardium and may modulate fibroblast phenotype and function

    Regulatory T cells are recruited in the infarcted mouse myocardium and may modulate fibroblast phenotype and function Dahkki Amit Saxena, Marcin Dobaczewski, Vikrant Rai, Zaffar Haque, Wei Chen, Na Li, Nikolaos G. Frangogiannis

    Almmustuhtton 2014
    Viečča ollesdeavstta
    Artigo
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  8. 8
    Interleukin-1 Receptor Type I Signaling Critically Regulates Infarct Healing and Cardiac Remodeling

    Interleukin-1 Receptor Type I Signaling Critically Regulates Infarct Healing and Cardiac Remodeling Dahkki Marcin Bujak, Marcin Dobaczewski, Khaled Chatila, Leonardo H. Mendoza, Na Li, Anilkumar K. Reddy, Nikolaos G. Frangogiannis

    Almmustuhtton 2008
    Viečča ollesdeavstta Viečča ollesdeavstta
    Artigo
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  9. 9
    Systematic Characterization of Myocardial Inflammation, Repair, and Remodeling in a Mouse Model of Reperfused Myocardial Infarction

    Systematic Characterization of Myocardial Inflammation, Repair, and Remodeling in a Mouse Model of Reperfused Myocardial Infarction Dahkki Panagiota Christia, Marcin Bujak, Carlos Gonzalez‐Quesada, Wei Chen, Marcin Dobaczewski, Anilkumar K. Reddy, Nikolaos G. Frangogiannis

    Almmustuhtton 2013
    Viečča ollesdeavstta Viečča ollesdeavstta
    Artigo
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  10. 10
    CXCR3-independent actions of the CXC chemokine CXCL10 in the infarcted myocardium and in isolated cardiac fibroblasts are mediated through proteoglycans

    CXCR3-independent actions of the CXC chemokine CXCL10 in the infarcted myocardium and in isolated cardiac fibroblasts are mediated through proteoglycans Dahkki Amit Saxena, Marcin Bujak, Olga Frunza, Marcin Dobaczewski, Carlos Gonzalez‐Quesada, Bao Lu, Craig Gérard, Nikolaos G. Frangogiannis

    Almmustuhtton 2014
    Viečča ollesdeavstta Viečča ollesdeavstta
    Artigo
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  11. 11
    Smad3 Signaling Critically Regulates Fibroblast Phenotype and Function in Healing Myocardial Infarction

    Smad3 Signaling Critically Regulates Fibroblast Phenotype and Function in Healing Myocardial Infarction Dahkki Marcin Dobaczewski, Marcin Bujak, Na Li, Carlos Gonzalez‐Quesada, Leonardo H. Mendoza, Xiao‐Fan Wang, Nikolaos G. Frangogiannis

    Almmustuhtton 2010
    Viečča ollesdeavstta
    Artigo
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  12. 12
    Endogenous Thrombospondin 1 Protects the Pressure-Overloaded Myocardium by Modulating Fibroblast Phenotype and Matrix Metabolism

    Endogenous Thrombospondin 1 Protects the Pressure-Overloaded Myocardium by Modulating Fibroblast Phenotype and Matrix Metabolism Dahkki Ying Xia, Marcin Dobaczewski, Carlos Gonzalez‐Quesada, Wei Chen, Anna Biernacka, Na Li, Dong-Wook Lee, Nikolaos G. Frangogiannis

    Almmustuhtton 2011
    Viečča ollesdeavstta
    Artigo
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  13. 13
    Endogenous IRAK-M Attenuates Postinfarction Remodeling Through Effects on Macrophages and Fibroblasts

    Endogenous IRAK-M Attenuates Postinfarction Remodeling Through Effects on Macrophages and Fibroblasts Dahkki Wei Chen, Amit Saxena, Na Li, Jin-Yu Sun, Amit Gupta, Dong-Wook Lee, Qi Tian, Marcin Dobaczewski, Nikolaos G. Frangogiannis

    Almmustuhtton 2012
    Viečča ollesdeavstta Viečča ollesdeavstta
    Artigo
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  14. 14
    Thrombospondin-1 Induction in the Diabetic Myocardium Stabilizes the Cardiac Matrix in Addition to Promoting Vascular Rarefaction Through Angiopoietin-2 Upregulation

    Thrombospondin-1 Induction in the Diabetic Myocardium Stabilizes the Cardiac Matrix in Addition to Promoting Vascular Rarefaction Through Angiopoietin-2 Upregulation Dahkki Carlos Gonzalez‐Quesada, Michele Cavalera, Anna Biernacka, Ping Kong, Dong‐Wook Lee, Amit Saxena, Olga Frunza, Marcin Dobaczewski, Arti V. Shinde, Nikolaos G. Frangogiannis

    Almmustuhtton 2013
    Viečča ollesdeavstta
    Artigo
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  15. 15
    Induction of the CXC Chemokine Interferon-γ-Inducible Protein 10 Regulates the Reparative Response Following Myocardial Infarction

    Induction of the CXC Chemokine Interferon-γ-Inducible Protein 10 Regulates the Reparative Response Following Myocardial Infarction Dahkki Marcin Bujak, Marcin Dobaczewski, Carlos Gonzalez‐Quesada, Ying Xia, Thorsten M. Leucker, Paweł Zymek, Vikas Veeranna, Andrew M. Tager, Andrew D. Luster, Nikolaos G. Frangogiannis

    Almmustuhtton 2009
    Viečča ollesdeavstta Viečča ollesdeavstta
    Artigo
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  16. 16
    Smad3 Signaling Promotes Fibrosis While Preserving Cardiac and Aortic Geometry in Obese Diabetic Mice

    Smad3 Signaling Promotes Fibrosis While Preserving Cardiac and Aortic Geometry in Obese Diabetic Mice Dahkki Anna Biernacka, Michele Cavalera, Junhong Wang, Ilaria Russo, Arti V. Shinde, Ping Kong, Carlos Gonzalez‐Quesada, Vikrant Rai, Marcin Dobaczewski, Dong-Wook Lee, Xiao-Fan Wang, Nikolaos G. Frangogiannis

    Almmustuhtton 2015
    Viečča ollesdeavstta Viečča ollesdeavstta
    Artigo
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