Resultados de procura - Lüder Hinrich Meyer

  • Mostrando 1 - 6 Resultados de 6
Limitar resultados
  1. 1
    Diversity of Human Leukemia Xenograft Mouse Models: Implications for Disease Biology

    Diversity of Human Leukemia Xenograft Mouse Models: Implications for Disease Biology por Lüder Hinrich Meyer, Klaus‐Michael Debatin

    Publicado 2011
    Ligazón do recurso Ligazón do recurso
    Revisão
    Engadir a favoritos
    Gardado en:
  2. 2
    Central nervous system involvement in acute lymphoblastic leukemia is mediated by vascular endothelial growth factor

    Central nervous system involvement in acute lymphoblastic leukemia is mediated by vascular endothelial growth factor por Vera Münch, Luca Trentin, Julia Herzig, Salih Demir, Felix Seyfried, Johann M. Kraus, Hans A. Kestler, Rolf Köhler, Thomas F.E. Barth, Geertruy te Kronnie, Klaus‐Michael Debatin, Lüder Hinrich Meyer

    Publicado 2017
    Ligazón do recurso Ligazón do recurso
    Artigo
    Engadir a favoritos
    Gardado en:
  3. 3
    Therapeutic targeting of mutant p53 in pediatric acute lymphoblastic leukemia

    Therapeutic targeting of mutant p53 in pediatric acute lymphoblastic leukemia por Salih Demir, Elena Boldrin, Qian Sun, Stephanie Hampp, Eugen Tausch, Cornelia Eckert, Martin Ebinger, Rupert Handgretinger, Geertruy te Kronnie, Lisa Wiesmüller, Stephan Stilgenbauer, Galina Selivanova, Klaus‐Michael Debatin, Lüder Hinrich Meyer

    Publicado 2019
    Ligazón do recurso Ligazón do recurso
    Artigo
    Engadir a favoritos
    Gardado en:
  4. 4
    ETV6/RUNX1-positive relapses evolve from an ancestral clone and frequently acquire deletions of genes implicated in glucocorticoid signaling

    ETV6/RUNX1-positive relapses evolve from an ancestral clone and frequently acquire deletions of genes implicated in glucocorticoid signaling por Lilian Kuster, Reinhard Grausenburger, Gerhard Fuka, Ulrike Kaindl, Gerd Krapf, Andrea Inthal, Georg Mann, Maximilian Kauer, Johannes Rainer, Reinhard Kofler, Andrew G. Hall, Markus Metzler, Lüder Hinrich Meyer, Claus Meyer, Jochen Harbott, Rolf Marschalek, Sabine Strehl, Oskar A. Haas, Renate Panzer‐Grümayer

    Publicado 2011
    Ligazón do recurso Ligazón do recurso
    Artigo
    Engadir a favoritos
    Gardado en:
  5. 5
    Early Relapse in ALL Is Identified by Time to Leukemia in NOD/SCID Mice and Is Characterized by a Gene Signature Involving Survival Pathways

    Early Relapse in ALL Is Identified by Time to Leukemia in NOD/SCID Mice and Is Characterized by a Gene Signature Involving Survival Pathways por Lüder Hinrich Meyer, Sarah M. Eckhoff, Manon Queudeville, Johann M. Kraus, Marco Giordan, Jana Stursberg, Andrea Zangrando, Elena Vendramini, Anja Möricke, Martin Zimmermann, André Schrauder, Georgia Lahr, Karlheinz Holzmann, Martin Schrappe, Giuseppe Basso, Karsten Stahnke, Hans A. Kestler, Geertruy te Kronnie, Klaus‐Michael Debatin

    Publicado 2011
    Ligazón do recurso Ligazón do recurso
    Artigo
    Engadir a favoritos
    Gardado en:
  6. 6
    Prediction of venetoclax activity in precursor B-ALL by functional assessment of apoptosis signaling

    Prediction of venetoclax activity in precursor B-ALL by functional assessment of apoptosis signaling por Felix Seyfried, Salih Demir, Rebecca Louise Hörl, Felix Uli Stirnweiß, Jeremy Ryan, Annika Scheffold, Mariana Villalobos-Ortiz, Elena Boldrin, Julia Zinngrebe, Stefanie Enzenmüller, Silvia Jenni, Yi-Chien Tsai, Beat Bornhäuser, Axel Fürstberger, Johann M. Kraus, Hans A. Kestler, Jean‐Pierre Bourquin, Stephan Stilgenbauer, Anthony Letai, Klaus‐Michael Debatin, Lüder Hinrich Meyer

    Publicado 2019
    Ligazón do recurso Ligazón do recurso
    Artigo
    Engadir a favoritos
    Gardado en:

Ferramentas de procura: