Výsledky vyhledávání - Katrine Almind

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  1. 1
    Genetic Determinants of Energy Expenditure and Insulin Resistance in Diet-Induced Obesity in Mice

    Genetic Determinants of Energy Expenditure and Insulin Resistance in Diet-Induced Obesity in Mice Autor Katrine Almind, C. Ronald Kahn

    Vydáno 2004
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  2. 2
    A common amino acid polymorphism in insulin receptor substrate-1 causes impaired insulin signaling. Evidence from transfection studies.

    A common amino acid polymorphism in insulin receptor substrate-1 causes impaired insulin signaling. Evidence from transfection studies. Autor Katrine Almind, Gen Inoue, Oluf Pedersen, C. Ronald Kahn

    Vydáno 1996
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  3. 3
    Ectopic brown adipose tissue in muscle provides a mechanism for differences in risk of metabolic syndrome in mice

    Ectopic brown adipose tissue in muscle provides a mechanism for differences in risk of metabolic syndrome in mice Autor Katrine Almind, Monia Manieri, William I. Sivitz, Saverio Cinti, C. Ronald Kahn

    Vydáno 2007
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  4. 4
    Effects of Diet and Genetic Background on Sterol Regulatory Element-Binding Protein-1c, Stearoyl-CoA Desaturase 1, and the Development of the Metabolic Syndrome

    Effects of Diet and Genetic Background on Sterol Regulatory Element-Binding Protein-1c, Stearoyl-CoA Desaturase 1, and the Development of the Metabolic Syndrome Autor Sudha B. Biddinger, Katrine Almind, Makoto Miyazaki, Efi Kokkotou, James M. Ntambi, C. Ronald Kahn

    Vydáno 2005
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  5. 5
    A Systems Biology Approach Identifies Inflammatory Abnormalities Between Mouse Strains Prior to Development of Metabolic Disease

    A Systems Biology Approach Identifies Inflammatory Abnormalities Between Mouse Strains Prior to Development of Metabolic Disease Autor Marcelo A. Mori, Manway Liu, Olivier Bézy, Katrine Almind, Hagit Shapiro, Simon Kasif, C. Ronald Kahn

    Vydáno 2010
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  6. 6
    Impact of Genetic Background on Development of Hyperinsulinemia and Diabetes in Insulin Receptor/Insulin Receptor Substrate-1 Double Heterozygous Mice

    Impact of Genetic Background on Development of Hyperinsulinemia and Diabetes in Insulin Receptor/Insulin Receptor Substrate-1 Double Heterozygous Mice Autor Rohit Kulkarni, Katrine Almind, H. Joseph Goren, Jonathon N. Winnay, Kohjiro Ueki, Terumasa Okada, C. Ronald Kahn

    Vydáno 2003
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  7. 7
    Combined Analyses of 20 Common Obesity Susceptibility Variants

    Combined Analyses of 20 Common Obesity Susceptibility Variants Autor Camilla H. Sandholt, Thomas Sparsø, Niels Grarup, Anders Albrechtsen, Katrine Almind, Lars Hansen, Ulla Toft, Torben Jørgensen, Torben Hansen, Oluf Pedersen

    Vydáno 2010
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  8. 8
    Studies of Metabolic Phenotypic Correlates of 15 Obesity Associated Gene Variants

    Studies of Metabolic Phenotypic Correlates of 15 Obesity Associated Gene Variants Autor Camilla H. Sandholt, M. A. Vestmar, Dorthe S. Bille, Anders Borglykke, Katrine Almind, Lars Hansen, Annelli Sandbæk, Torsten Lauritzen, Daniel R. Witte, Torben Jørgensen, Oluf Pedersen, Torben Hansen

    Vydáno 2011
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  9. 9
    Insulin resistance: interactions between obesity and a common variant of insulin receptor substrate-1

    Insulin resistance: interactions between obesity and a common variant of insulin receptor substrate-1 Autor J. O. Clausen, Torben Hansen, Christian Bjørbæk, S. M. Echwald, Søren A. Urhammer, S. Rasmussen, Carsten Andersen, Lars Hansen, Katrine Almind, Oluf Pedersen, J. O. Clausen, Knut Borch-Johnsen, K. Winther, Jóhanna Haraldsdóttir

    Vydáno 1995
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  10. 10
    Body Composition Is the Main Determinant for the Difference in Type 2 Diabetes Pathophysiology Between Japanese and Caucasians

    Body Composition Is the Main Determinant for the Difference in Type 2 Diabetes Pathophysiology Between Japanese and Caucasians Autor Jonas Bech Møller, Maria Pedersen, Haruhiko Tanaka, Mitsuru Ohsugi, Rune Viig Overgaard, Jan Lynge, Katrine Almind, Nina-Maria Vasconcelos, Pernille Poulsen, Charlotte Keller, Kohjiro Ueki, Steen H. Ingwersen, Bente Klarlund Pedersen, Takashi Kadowaki

    Vydáno 2013
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  11. 11
    Novel MODY3 Mutations in the Hepatocyte Nuclear Factor-1α Gene: Evidence for a Hyperexcitability of Pancreatic β-cells to Intravenous Secretagogues in a Glucose-Tolerant Carrier of a P447L Mutation

    Novel MODY3 Mutations in the Hepatocyte Nuclear Factor-1α Gene: Evidence for a Hyperexcitability of Pancreatic β-cells to Intravenous Secretagogues in a Glucose-Tolerant Carrier of... Autor Torben Hansen, Hans Eiberg, Mathias Rouard, Martine Vaxillaire, Ann Merete Møller, Sören K. Rasmussen, Marianne Fridberg, Søren A. Urhammer, Jens J. Holst, Katrine Almind, Søren M. Echwald, Lars Hansen, Graeme I. Bell, Oluf Pedersen

    Vydáno 1997
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