Resultats de la cerca - Karlheinz Holzmann

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  1. 1
    The Early Activation Marker CD69 Regulates the Expression of Chemokines and CD4 T Cell Accumulation in Intestine

    The Early Activation Marker CD69 Regulates the Expression of Chemokines and CD4 T Cell Accumulation in Intestine per Katarina Radulović, Valerio Rossini, Calin Manta, Karlheinz Holzmann, Hans A. Kestler, Jan Hendrik Niess

    Publicat 2013
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  2. 2
    CD69 Regulates Type I IFN-Induced Tolerogenic Signals to Mucosal CD4 T Cells That Attenuate Their Colitogenic Potential

    CD69 Regulates Type I IFN-Induced Tolerogenic Signals to Mucosal CD4 T Cells That Attenuate Their Colitogenic Potential per Katarina Radulović, Calin Manta, Valerio Rossini, Karlheinz Holzmann, Hans A. Kestler, Ursula Maria Wegenka, Toshinori Nakayama, Jan Hendrik Niess

    Publicat 2012
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  3. 3
    miR-146a-mediated suppression of the inflammatory response in human adipocytes

    miR-146a-mediated suppression of the inflammatory response in human adipocytes per Julian Roos, Eveliina Enlund, Jan‐Bernd Funcke, Daniel Tews, Karlheinz Holzmann, Klaus‐Michael Debatin, Martin Wabitsch, Pamela Fischer‐Posovszky

    Publicat 2016
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  4. 4
    Synergy between PI3K Signaling and MYC in Burkitt Lymphomagenesis

    Synergy between PI3K Signaling and MYC in Burkitt Lymphomagenesis per Sandrine Sander, Dinis Pedro Calado, Lakshmi Srinivasan, Karl Köchert, Baochun Zhang, Maciej Rosołowski, Scott J. Rodig, Karlheinz Holzmann, Stephan Stilgenbauer, Reiner Siebert, Lars Bullinger, Klaus Rajewsky

    Publicat 2012
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  5. 5
    Genomic DNA-Chip Hybridization Reveals a Higher Incidence of Genomic Amplifications in Pancreatic Cancer than Conventional Comparative Genomic Hybridization and Leads to the Identification of Novel Candidate Genes

    Genomic DNA-Chip Hybridization Reveals a Higher Incidence of Genomic Amplifications in Pancreatic Cancer than Conventional Comparative Genomic Hybridization and Leads to the Identi... per Karlheinz Holzmann, Holger Kohlhammer, Carsten Schwäenen, Swen Weßendorf, Hans A. Kestler, Alexandra Schwoerer, Bettina Rau, Bernhard Radlwimmer, Hartmut Döhner, Peter Lichter, Thomas M. Gress, Martin Bentz

    Publicat 2004
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  6. 6
    Dihydrofolate Reductase Deficiency Due to a Homozygous DHFR Mutation Causes Megaloblastic Anemia and Cerebral Folate Deficiency Leading to Severe Neurologic Disease

    Dihydrofolate Reductase Deficiency Due to a Homozygous DHFR Mutation Causes Megaloblastic Anemia and Cerebral Folate Deficiency Leading to Severe Neurologic Disease per Holger Cario, Desirée E.C. Smith, Henk J. Blom, Nenad Blau, Harald Bode, Karlheinz Holzmann, Ulrich Pannicke, Karl‐Peter Hopfner, Eva-Maria Rump, Zuleya Ayric, E. Kohne, Klaus‐Michael Debatin, Yvo M. Smulders, Klaus Schwarz

    Publicat 2011
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  7. 7
    Dysregulation of a novel miR-1825/TBCB/TUBA4A pathway in sporadic and familial ALS

    Dysregulation of a novel miR-1825/TBCB/TUBA4A pathway in sporadic and familial ALS per Anika M. Helferich, Sarah J. Brockmann, Jörg Reinders, Dhruva Deshpande, Karlheinz Holzmann, Dávid Brenner, Peter M. Andersen, Susanne Petri, Dietmar Rudolf Thal, Jens Michaelis, Markus Otto, Steffen Just, Albert C. Ludolph, Karin M. Danzer, Axel Freischmidt, Jochen H. Weishaupt

    Publicat 2018
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  8. 8
    Serum microRNAs in patients with genetic amyotrophic lateral sclerosis and pre-manifest mutation carriers

    Serum microRNAs in patients with genetic amyotrophic lateral sclerosis and pre-manifest mutation carriers per Axel Freischmidt, Kathrin Müller, Lisa Zondler, Patrick Weydt, Alexander E. Volk, Anže Božič, Michael Walter, Michael Bonin, Benjamin Mayer, Christine A. F. Von Arnim, Markus Otto, Christoph Dieterich, Karlheinz Holzmann, Peter M. Andersen, Albert C. Ludolph, Karin M. Danzer, Jochen H. Weishaupt

    Publicat 2014
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  9. 9
    Deficiency of Innate and Acquired Immunity Caused by an <i>IKBKB</i> Mutation

    Deficiency of Innate and Acquired Immunity Caused by an <i>IKBKB</i> Mutation per Ulrich Pannicke, Bernd Baumann, Sebastian Fuchs, Philipp Henneke, Anne Rensing‐Ehl, Marta Rizzi, Aleš Janda, Katrin Hese, Michael Schlesier, Karlheinz Holzmann, Stephan Borte, Constanze Laux, Eva-Maria Rump, Alan Rosenberg, Teresa Zelinski, Hubert Schrezenmeier, Thomas Wirth, Stephan Ehl, Marlis L. Schroeder, Klaus Schwarz

    Publicat 2013
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  10. 10
    Clinical impact of the genomic landscape and leukemogenic trajectories in non-intensively treated elderly acute myeloid leukemia patients

    Clinical impact of the genomic landscape and leukemogenic trajectories in non-intensively treated elderly acute myeloid leukemia patients per Nikolaus Jahn, Maral Saadati, Pierre Fenaux, Marco Gobbi, Gail J. Roboz, Lars Bullinger, Pavlo Lutsik, Anna Riedel, Christoph Plass, Nikolaus Jahn, Claudia Walter, Karlheinz Holzmann, Yong Hao, Sue Naim, Nicholas Schreck, Julia Krzykalla, Axel Benner, Harold Keer, Mohammad Azab, Konstanze Döhner, Hartmut Döhner

    Publicat 2023
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  11. 11
    Early Relapse in ALL Is Identified by Time to Leukemia in NOD/SCID Mice and Is Characterized by a Gene Signature Involving Survival Pathways

    Early Relapse in ALL Is Identified by Time to Leukemia in NOD/SCID Mice and Is Characterized by a Gene Signature Involving Survival Pathways per Lüder Hinrich Meyer, Sarah M. Eckhoff, Manon Queudeville, Johann M. Kraus, Marco Giordan, Jana Stursberg, Andrea Zangrando, Elena Vendramini, Anja Möricke, Martin Zimmermann, André Schrauder, Georgia Lahr, Karlheinz Holzmann, Martin Schrappe, Giuseppe Basso, Karsten Stahnke, Hans A. Kestler, Geertruy te Kronnie, Klaus‐Michael Debatin

    Publicat 2011
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  12. 12
    Clonal evolution in relapsed NPM1-mutated acute myeloid leukemia

    Clonal evolution in relapsed NPM1-mutated acute myeloid leukemia per Jan Krönke, Lars Bullinger, Veronica Teleanu, Florian Tschürtz, Verena I. Gaidzik, Michael W.M. Kühn, Frank G. Rücker, Karlheinz Holzmann, Peter Paschka, Silke Kapp-Schwörer, Daniela Späth, Thomas Kindler, Marcus M. Schittenhelm, Jürgen Krauter, Arnold Ganser, Gudrun Göhring, Brigitte Schlegelberger, Richard F. Schlenk, Hartmut Döhner, Konstanze Döhner

    Publicat 2013
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  13. 13
    High-resolution genomic profiling of chronic lymphocytic leukemia reveals new recurrent genomic alterations

    High-resolution genomic profiling of chronic lymphocytic leukemia reveals new recurrent genomic alterations per Jennifer Edelmann, Karlheinz Holzmann, Florian Miller, Dirk Winkler, Andreas Bühler, Thorsten Zenz, Lars Bullinger, Michael W.M. Kühn, Andreas Gerhardinger, Johannes Bloehdorn, Ina Radtke, Xiaoping Su, Jing Ma, Stanley Pounds, Michael Hallek, Peter Lichter, Jan O. Korbel, Raymonde Busch, Daniel Mertens, James R. Downing, Stephan Stilgenbauer, Hartmut Döhner

    Publicat 2012
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  14. 14
    TP53 alterations in acute myeloid leukemia with complex karyotype correlate with specific copy number alterations, monosomal karyotype, and dismal outcome

    TP53 alterations in acute myeloid leukemia with complex karyotype correlate with specific copy number alterations, monosomal karyotype, and dismal outcome per Frank G. Rücker, Richard F. Schlenk, Lars Bullinger, Sabine Käyser, Veronica Teleanu, H. Kett, Marianne Habdank, Carla-Maria Kugler, Karlheinz Holzmann, Verena I. Gaidzik, Peter Paschka, Gerhard Held, Marie von Lilienfeld‐Toal, Michael Lübbert, Stefan Fröhling, Thorsten Zenz, Jürgen Krauter, Brigitte Schlegelberger, Arnold Ganser, Peter Lichter, Konstanze Döhner, Hartmut Döhner

    Publicat 2011
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  15. 15
    High-resolution genomic profiling of adult and pediatric core-binding factor acute myeloid leukemia reveals new recurrent genomic alterations

    High-resolution genomic profiling of adult and pediatric core-binding factor acute myeloid leukemia reveals new recurrent genomic alterations per Michael W.M. Kühn, Ina Radtke, Lars Bullinger, Salil Goorha, Jinjun Cheng, Jennifer Edelmann, Juliane Gohlke, Xiaoping Su, Peter Paschka, Stanley Pounds, Jürgen Krauter, Arnold Ganser, Asmâa Quessar, Raul C. Ribeiro, Verena I. Gaidzik, Sheila Shurtleff, Jan Krönke, Karlheinz Holzmann, Jing Ma, Richard F. Schlenk, Jeffrey E. Rubnitz, Konstanze Döhner, Hartmut Döhner, James R. Downing

    Publicat 2012
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  16. 16
    Genomic alterations in high-risk chronic lymphocytic leukemia frequently affect cell cycle key regulators and NOTCH1-regulated transcription

    Genomic alterations in high-risk chronic lymphocytic leukemia frequently affect cell cycle key regulators and NOTCH1-regulated transcription per Jennifer Edelmann, Karlheinz Holzmann, Eugen Tausch, Emily A. Saunderson, Billy Michael Chelliah Jebaraj, Daniela Steinbrecher, Anna Dolnik, Tamara J. Blätte, Dan A. Landau, Jenny Saub, Sven Estenfelder, Stefan Ibach, Florence Cymbalista, Véronique Leblond, Alain Delmer, Jasmin Bahlo, Sandra Robrecht, Kirsten Fischer, Valentin Goede, Lars Bullinger, Catherine J. Wu, Daniel Mertens, Gabriella Ficz, John G. Gribben, Michael Hallek, Hartmut Döhner, Stephan Stilgenbauer

    Publicat 2019
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  17. 17
    Impaired DNA damage response signaling by FUS-NLS mutations leads to neurodegeneration and FUS aggregate formation

    Impaired DNA damage response signaling by FUS-NLS mutations leads to neurodegeneration and FUS aggregate formation per Marcel Naumann, Arun Pal, Anand Goswami, Xenia Lojewski, Julia Japtok, Anne Vehlow, Maximilian Naujock, René Günther, Mengmeng Jin, Nancy Stanslowsky, Peter Reinhardt, Jared Sterneckert, Marie Frickenhaus, Francisco Pan‐Montojo, Erik Storkebaum, Ina Poser, Axel Freischmidt, Jochen H. Weishaupt, Karlheinz Holzmann, Dirk Troost, Albert C. Ludolph, Tobias M. Boeckers, Stefan Liebau, Susanne Petri, Nils Cordes, Anthony A. Hyman, Florian Wegner, Stephan W. Grill, Joachim Weis, Alexander Storch, Andreas Hermann

    Publicat 2018
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