Resultados da pesquisa - Kamilla E. Jensen

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  1. 1
    Enhanced efficacy of combined HDAC and PARP targeting in glioblastoma

    Enhanced efficacy of combined HDAC and PARP targeting in glioblastoma Por Rikke Rasmussen, Madhavsai Gajjar, Kamilla E. Jensen, Petra Hamerlik

    Publicado em 2016
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  2. 2
    Targeting glioma stem‐like cell survival and chemoresistance through inhibition of lysine‐specific histone demethylase <scp>KDM</scp>2B

    Targeting glioma stem‐like cell survival and chemoresistance through inhibition of lysine‐specific histone demethylase <scp>KDM</scp>2B Por Mikkel Staberg, Rikke Rasmussen, Signe Regner Michaelsen, Henriette Pedersen, Kamilla E. Jensen, Mette Villingshøj, Jane Skjøth‐Rasmussen, Jannick Brennum, Kristoffer Vitting‐Seerup, Hans Skovgaard Poulsen, Petra Hamerlik

    Publicado em 2018
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  3. 3
    BRCA1-regulated RRM2 expression protects glioblastoma cells from endogenous replication stress and promotes tumorigenicity

    BRCA1-regulated RRM2 expression protects glioblastoma cells from endogenous replication stress and promotes tumorigenicity Por Rikke Rasmussen, Madhavsai Gajjar, Lucie Tučková, Kamilla E. Jensen, Apolinar Maya‐Mendoza, Camilla Bjørnbak Holst, Kjeld Møllgaard, Jane S. Rasmussen, Jannick Brennum, Jiří Bártek, Martin Syrůček, Eva Sedlákova, Klaus K. Andersen, Marie Frederiksen, Jiri Bartek, Petra Hamerlik

    Publicado em 2016
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  4. 4
    Structural and Functional Insight into How the Plasmodium falciparum VAR2CSA Protein Mediates Binding to Chondroitin Sulfate A in Placental Malaria

    Structural and Functional Insight into How the Plasmodium falciparum VAR2CSA Protein Mediates Binding to Chondroitin Sulfate A in Placental Malaria Por Thomas Mandel Clausen, S. Christoffersen, Madeleine Dahlbäck, Annette E. Langkilde, Kamilla E. Jensen, Mafalda Resende, Mette Ø. Agerbæk, Daniel B. Andersen, Besim Berisha, Sisse B. Ditlev, Vera V. Pinto, Morten A. Nielsen, Thor G. Theander, Sine Larsen, Ali Salanti

    Publicado em 2012
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  5. 5
    VEGF-C sustains VEGFR2 activation under bevacizumab therapy and promotes glioblastoma maintenance

    VEGF-C sustains VEGFR2 activation under bevacizumab therapy and promotes glioblastoma maintenance Por Signe Regner Michaelsen, Mikkel Staberg, Henriette Pedersen, Kamilla E. Jensen, Wiktor W. Majewski, Helle Broholm, Mette Kjølhede Nedergaard, Christopher Meulengracht, Thomas Urup, Mette Villingshøj, Slávka Lukacova, Jane Skjøth‐Rasmussen, Jannick Brennum, Andreas Kjær, Ulrik Lassen, Marie‐Thérése Stockhausen, Hans Skovgaard Poulsen, Petra Hamerlik

    Publicado em 2018
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