Resultados de procura - Jonathan Neumann

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  1. 1
    Reduced Levels of microRNAs miR-124a and miR-150 Are Associated with Increased Proinflammatory Mediator Expression in Krüppel-like Factor 2 (KLF2)-deficient Macrophages

    Reduced Levels of microRNAs miR-124a and miR-150 Are Associated with Increased Proinflammatory Mediator Expression in Krüppel-like Factor 2 (KLF2)-deficient Macrophages por Palanikumar Manoharan, Joshua E. Basford, Robyn Pilcher-Roberts, Jonathan Neumann, David Y. Hui, Jerry B. Lingrel

    Publicado 2014
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  2. 2
    The α2 Isoform of Na,K-ATPase Mediates Ouabain-induced Cardiac Inotropy in Mice

    The α2 Isoform of Na,K-ATPase Mediates Ouabain-induced Cardiac Inotropy in Mice por Iva Dostanic, John N. Lorenz, Jo El J. Schultz, Ingrid L. Grupp, Jonathan Neumann, Maqsood A. Wani, Jerry B. Lingrel

    Publicado 2003
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  3. 3
    Targeted Ablation of Plasma Membrane Ca2+-ATPase (PMCA) 1 and 4 Indicates a Major Housekeeping Function for PMCA1 and a Critical Role in Hyperactivated Sperm Motility and Male Fertility for PMCA4

    Targeted Ablation of Plasma Membrane Ca2+-ATPase (PMCA) 1 and 4 Indicates a Major Housekeeping Function for PMCA1 and a Critical Role in Hyperactivated Sperm Motility and Male Fert... por Gbolahan W. Okunade, Marian L. Miller, Gail J. Pyne, Roy L. Sutliff, Kyle T. O'Connor, Jonathan Neumann, Anastasia Andringa, D. A. Miller, Vikram Prasad, Thomas Doetschman, Richard J. Paul, Gary E. Shull

    Publicado 2004
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  4. 4
    Systematic phenotyping and characterization of the 5xFAD mouse model of Alzheimer’s disease

    Systematic phenotyping and characterization of the 5xFAD mouse model of Alzheimer’s disease por Stefânia Forner, Shimako Kawauchi, Gabriela Balderrama-Gutierrez, Enikö A. Kramár, Dina P. Matheos, Jimmy Phan, Dominic I. Javonillo, Kristine M. Tran, Edna E. Hingco, Celia Da Cunha, Narges Rezaie, Joshua A. Alcantara, David Baglietto‐Vargas, Camden Jansen, Jonathan Neumann, Marcelo A. Wood, Grant R. MacGregor, A Mortazavi, Andrea J. Tenner, Frank M. LaFerla, Kim N. Green

    Publicado 2021
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  5. 5
    Systematic Phenotyping and Characterization of the 3xTg-AD Mouse Model of Alzheimer’s Disease

    Systematic Phenotyping and Characterization of the 3xTg-AD Mouse Model of Alzheimer’s Disease por Dominic I. Javonillo, Kristine M. Tran, Jimmy Phan, Edna E. Hingco, Enikö A. Kramár, Celia Da Cunha, Stefânia Forner, Shimako Kawauchi, Giedre Milinkeviciute, Ángela Gómez-Arboledas, Jonathan Neumann, Crystal E. Banh, Michelle Huynh, Dina P. Matheos, Narges Rezaie, Joshua A. Alcantara, A Mortazavi, Marcelo A. Wood, Andrea J. Tenner, Grant R. MacGregor, Kim N. Green, Frank M. LaFerla

    Publicado 2022
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  6. 6
    The <i>Abca7<sup>V1613M</sup></i> variant reduces Aβ generation, plaque load, and neuronal damage

    The <i>Abca7<sup>V1613M</sup></i> variant reduces Aβ generation, plaque load, and neuronal damage por Claire A. Butler, Adrian Mendoza Arvilla, Giedre Milinkeviciute, Celia Da Cunha, Shimako Kawauchi, Narges Rezaie, Heidi Yahan Liang, Dominic I. Javonillo, Annie Thach, Shuling Wang, Sherilyn Collins, Amber Walker, Kai‐Xuan Shi, Jonathan Neumann, Ángela Gómez-Arboledas, Caden M. Henningfield, Lindsay A. Hohsfield, Mark Mapstone, Andrea J. Tenner, Frank M. LaFerla, A Mortazavi, Grant R. MacGregor, Kim N. Green

    Publicado 2024
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  7. 7
    APOE Christchurch enhances a disease-associated microglial response to plaque but suppresses response to tau pathology

    APOE Christchurch enhances a disease-associated microglial response to plaque but suppresses response to tau pathology por Kristine M. Tran, Nellie Kwang, Claire A. Butler, Ángela Gómez-Arboledas, Shimako Kawauchi, Cassandra Mar, Donna Chao, Rocio A. Barahona, Celia Da Cunha, Kate I. Tsourmas, Zechuan Shi, Shuling Wang, Sherilyn Collins, Amber Walker, Kai‐Xuan Shi, Joshua A. Alcantara, Jonathan Neumann, Duc M. Duong, Nicholas T. Seyfried, Andrea J. Tenner, Frank M. LaFerla, Lindsay A. Hohsfield, Vivek Swarup, Grant R. MacGregor, Kim N. Green

    Publicado 2025
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  8. 8
    A Trem2R47H mouse model without cryptic splicing drives age- and disease-dependent tissue damage and synaptic loss in response to plaques

    A Trem2R47H mouse model without cryptic splicing drives age- and disease-dependent tissue damage and synaptic loss in response to plaques por Kristine M. Tran, Shimako Kawauchi, Enikö A. Kramár, Narges Rezaie, Heidi Yahan Liang, Jasmine Sakr, Ángela Gómez-Arboledas, Miguel A. Arreola, Celia Da Cunha, Jimmy Phan, Shuling Wang, Sherilyn Collins, Amber Walker, Kai‐Xuan Shi, Jonathan Neumann, Ghassan Filimban, Zechuan Shi, Giedre Milinkeviciute, Dominic I. Javonillo, Katelynn Tran, Magdalena Gantuz, Stefânia Forner, Vivek Swarup, Andrea J. Tenner, Frank M. LaFerla, Marcelo A. Wood, A Mortazavi, Grant R. MacGregor, Kim N. Green

    Publicado 2023
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