Хайлтын үр дүнгүүд - Jin‐Sung Chung

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  1. 1
    Syndecan-4 Mediates the Coinhibitory Function of DC-HIL on T Cell Activation

    Syndecan-4 Mediates the Coinhibitory Function of DC-HIL on T Cell Activation -н Jin-Sung Chung, Irene Dougherty, Ponciano D. Cruz, Kiyoshi Ariizumi

    Хэвлэсэн 2007
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    Artigo
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  2. 2
    DC-HIL/Glycoprotein Nmb Promotes Growth of Melanoma in Mice by Inhibiting the Activation of Tumor-Reactive T Cells

    DC-HIL/Glycoprotein Nmb Promotes Growth of Melanoma in Mice by Inhibiting the Activation of Tumor-Reactive T Cells -н Mizuki Tomihari, Jin-Sung Chung, Hideo Akiyoshi, Ponciano D. Cruz, Kiyoshi Ariizumi

    Хэвлэсэн 2010
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    Artigo
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  3. 3
    DC-HIL is a negative regulator of T lymphocyte activation

    DC-HIL is a negative regulator of T lymphocyte activation -н Jin-Sung Chung, Kota Sato, Irene Dougherty, Ponciano D. Cruz, Kiyoshi Ariizumi

    Хэвлэсэн 2007
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    Artigo
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  4. 4
    The DC‐HIL/syndecan‐4 pathway inhibits human allogeneic T‐cell responses

    The DC‐HIL/syndecan‐4 pathway inhibits human allogeneic T‐cell responses -н Jin‐Sung Chung, Makoto Bonkobara, Mizuki Tomihari, Ponciano D. Cruz, Kiyoshi Ariizumi

    Хэвлэсэн 2009
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    Artigo
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  5. 5
    Gpnmb is a melanosome‐associated glycoprotein that contributes to melanocyte/keratinocyte adhesion in a RGD‐dependent fashion

    Gpnmb is a melanosome‐associated glycoprotein that contributes to melanocyte/keratinocyte adhesion in a RGD‐dependent fashion -н Mizuki Tomihari, Sun‐Hee Hwang, Jin‐Sung Chung, Ponciano D. Cruz, Kiyoshi Ariizumi

    Хэвлэсэн 2009
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    Artigo
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  6. 6
    Myeloid-Derived Suppressor Cells in Psoriasis Are an Expanded Population Exhibiting Diverse T-Cell–Suppressor Mechanisms

    Myeloid-Derived Suppressor Cells in Psoriasis Are an Expanded Population Exhibiting Diverse T-Cell–Suppressor Mechanisms -н Lauren Cao, Jin‐Sung Chung, Takahiro Teshima, Lawrence S. Feigenbaum, Ponciano D. Cruz, Heidi Jacobe, Benjamin F. Chong, Kiyoshi Ariizumi

    Хэвлэсэн 2016
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    Artigo
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  7. 7
    Acquired resistance to immune checkpoint therapy is caused by glycoprotein non-metastatic melanoma protein B signal cascade

    Acquired resistance to immune checkpoint therapy is caused by glycoprotein non-metastatic melanoma protein B signal cascade -н Jin‐Sung Chung, Vijay Ramani, Lei Guo, Vinita Popat, Ponciano D. Cruz, Lin Xu, Hans J. Hammers, Kiyoshi Ariizumi

    Хэвлэсэн 2025
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    Artigo
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  8. 8
    Blocking Monocytic Myeloid-Derived Suppressor Cell Function via Anti-DC-HIL/GPNMB Antibody Restores the <i>In Vitro</i> Integrity of T Cells from Cancer Patients

    Blocking Monocytic Myeloid-Derived Suppressor Cell Function via Anti-DC-HIL/GPNMB Antibody Restores the <i>In Vitro</i> Integrity of T Cells from Cancer Patients -н Masato Kobayashi, Jin-Sung Chung, Muhammad Shaalan Beg, Yull Arriaga, Udit Verma, Kevin D. Courtney, John C. Mansour, Barbara Haley, Saad A. Khan, Yutaka Horiuchi, Vijay Ramani, David Harker, Purva Gopal, Farshid Araghizadeh, Ponciano D. Cruz, Kiyoshi Ariizumi

    Хэвлэсэн 2018
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    Artigo
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