نتائج البحث - Jan Dürig

تنقيح النتائج
  1. 1
    HLA-G expression is associated with an unfavorable outcome and immunodeficiency in chronic lymphocytic leukemia

    HLA-G expression is associated with an unfavorable outcome and immunodeficiency in chronic lymphocytic leukemia حسب Holger Nückel, Vera Rebmann, Jan Dürig, Ulrich Dührsen, Hans Grosse‐Wilde

    منشور في 2004
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  2. 2
    CD38 is a signaling molecule in B-cell chronic lymphocytic leukemia cells

    CD38 is a signaling molecule in B-cell chronic lymphocytic leukemia cells حسب Silvia Deaglio, Andrea Capobianco, L Bergui, Jan Dürig, Fortunato Morabito, Ulrich Dührsen, Fabio Malavasi

    منشور في 2003
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  3. 3
    Cellular origin and pathophysiology of chronic lymphocytic leukemia

    Cellular origin and pathophysiology of chronic lymphocytic leukemia حسب Marc Seifert, Ludger Sellmann, Johannes Bloehdorn, Frederik Wein, Stephan Stilgenbauer, Jan Dürig, Ralf Küppers

    منشور في 2012
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  4. 4
    Revision of the Human Hematopoietic Tree: Granulocyte Subtypes Derive from Distinct Hematopoietic Lineages

    Revision of the Human Hematopoietic Tree: Granulocyte Subtypes Derive from Distinct Hematopoietic Lineages حسب André Görgens, Stefan Radtke, Michael Möllmann, Michael Cross, Jan Dürig, Peter A. Horn, Bernd Giebel

    منشور في 2013
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  5. 5
    CD38 expression is an important prognostic marker in chronic lymphocytic leukaemia

    CD38 expression is an important prognostic marker in chronic lymphocytic leukaemia حسب Jan Dürig, M Naschar, U Schmücker, K. Renzing‐Köhler, Tanja Hölter, Andreas Hüttmann, Ulrich Dührsen

    منشور في 2002
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  6. 6
    Deorphanization and characterization of the ectopically expressed olfactory receptor OR51B5 in myelogenous leukemia cells

    Deorphanization and characterization of the ectopically expressed olfactory receptor OR51B5 in myelogenous leukemia cells حسب Stavros Manteniotis, Sebastian Wojcik, Joachim R. Göthert, Jan Dürig, Ulrich Dührsen, Günter Gisselmann, Hanns Hatt

    منشور في 2016
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  7. 7
    Dihydroartemisinin Induces Apoptosis by a Bak-Dependent Intrinsic Pathway

    Dihydroartemisinin Induces Apoptosis by a Bak-Dependent Intrinsic Pathway حسب René Handrick, Teona Ontikatze, Kerstin-Daniela Bauer, Florian Freier, Amelie Rübel, Jan Dürig, Claus Belka, Verena Jendrossek

    منشور في 2010
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  8. 8
    Multipotent Hematopoietic Progenitors Divide Asymmetrically to Create Progenitors of the Lymphomyeloid and Erythromyeloid Lineages

    Multipotent Hematopoietic Progenitors Divide Asymmetrically to Create Progenitors of the Lymphomyeloid and Erythromyeloid Lineages حسب André Görgens, Anna‐Kristin Ludwig, Michael Möllmann, Adalbert Krawczyk, Jan Dürig, Helmut Hanenberg, Peter A. Horn, Bernd Giebel

    منشور في 2014
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  9. 9
    Association of a novel regulatory polymorphism (−938C>A) in the BCL2 gene promoter with disease progression and survival in chronic lymphocytic leukemia

    Association of a novel regulatory polymorphism (−938C>A) in the BCL2 gene promoter with disease progression and survival in chronic lymphocytic leukemia حسب Holger Nückel, Ulrich H. Frey, Maja Bau, Ludger Sellmann, Jens Stanelle, Jan Dürig, Karl‐Heinz Jöckel, Ulrich Dührsen, Winfried Siffert

    منشور في 2006
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  10. 10
    Epigenetic Silencing of the Circadian Clock Gene CRY1 is Associated with an Indolent Clinical Course in Chronic Lymphocytic Leukemia

    Epigenetic Silencing of the Circadian Clock Gene CRY1 is Associated with an Indolent Clinical Course in Chronic Lymphocytic Leukemia حسب Maher Hanoun, Lewin Eisele, Masako Suzuki, John M. Greally, Andreas Hüttmann, Semra Aydin, René Scholtysik, Ludger Klein‐Hitpaß, Ulrich Dührsen, Jan Dürig

    منشور في 2012
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  11. 11
    Recurrent mutations in NF-κB pathway components, <i>KMT2D,</i> and <i>NOTCH1/2</i> in ocular adnexal MALT-type marginal zone lymphomas

    Recurrent mutations in NF-κB pathway components, <i>KMT2D,</i> and <i>NOTCH1/2</i> in ocular adnexal MALT-type marginal zone lymphomas حسب Patricia Johansson, Ludger Klein‐Hitpaß, Florian Grabellus, Georg J. Arnold, Wolfram Klapper, Roman Pförtner, Ulrich Dührsen, Anja Eckstein, Jan Dürig, Ralf Küppers

    منشور في 2016
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  12. 12
    <scp>HLA‐E</scp> allelic genotype correlates with <scp>HLA‐E</scp> plasma levels and predicts early progression in chronic lymphocytic leukemia

    <scp>HLA‐E</scp> allelic genotype correlates with <scp>HLA‐E</scp> plasma levels and predicts early progression in chronic lymphocytic leukemia حسب Bettina Wagner, Fabiola da Silva Nardi, Sabine Schramm, Thomas Kraemer, Alexander A. Celik, Jan Dürig, Peter A. Horn, Ulrich Dührsen, Holger Nückel, Vera Rebmann

    منشور في 2016
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  13. 13
    A Novel Nonobese Diabetic/Severe Combined Immunodeficient Xenograft Model for Chronic Lymphocytic Leukemia Reflects Important Clinical Characteristics of the Disease

    A Novel Nonobese Diabetic/Severe Combined Immunodeficient Xenograft Model for Chronic Lymphocytic Leukemia Reflects Important Clinical Characteristics of the Disease حسب Jan Dürig, Peter R. Ebeling, Florian Grabellus, Ursula R. Sorg, Michael Möllmann, Philipp Schütt, Joachim R. Göthert, Ludger Sellmann, S. Seeber, Michael Flaßhove, Ulrich Dührsen, Thomas Moritz

    منشور في 2007
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  14. 14
    miR-34a as part of the resistance network in chronic lymphocytic leukemia

    miR-34a as part of the resistance network in chronic lymphocytic leukemia حسب Thorsten Zenz, Julia Mohr, Eric Eldering, Arnon P. Kater, Andreas Bühler, Dirk Kienle, Dirk Winkler, Jan Dürig, Marinus H. J. van Oers, Daniel Mertens, Hartmut Döhner, Stephan Stilgenbauer

    منشور في 2008
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  15. 15
    Expression of ribosomal and translation-associated genes is correlated with a favorable clinical course in chronic lymphocytic leukemia

    Expression of ribosomal and translation-associated genes is correlated with a favorable clinical course in chronic lymphocytic leukemia حسب Jan Dürig, Holger Nückel, Andreas Hüttmann, Elisabeth Kruse, Tanja Hölter, Katja Halfmeyer, Anja Führer, R. Rudolph, Naser Kalhori, Arnd Nusch, Silvia Deaglio, Fabio Malavasi, Tarik Möröy, Ludger Klein‐Hitpaß, Ulrich Dührsen

    منشور في 2003
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  16. 16
    SAMHD1 is recurrently mutated in T-cell prolymphocytic leukemia

    SAMHD1 is recurrently mutated in T-cell prolymphocytic leukemia حسب Patricia Johansson, Ludger Klein‐Hitpaß, Axel Choidas, Peter Habenberger, Bijan Mahboubi, Baek Kim, Anke K. Bergmann, René Scholtysik, Martina Brauser, Anna Lollies, Reiner Siebert, Thorsten Zenz, Ulrich Dührsen, Ralf Küppers, Jan Dürig

    منشور في 2018
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  17. 17
    Poor efficacy and tolerability of <scp>R‐CHOP</scp> in relapsed/refractory chronic lymphocytic leukemia and <scp>R</scp>ichter transformation

    Poor efficacy and tolerability of <scp>R‐CHOP</scp> in relapsed/refractory chronic lymphocytic leukemia and <scp>R</scp>ichter transformation حسب Petra Langerbeins, Raymonde Busch, Nadine Anheier, Jan Dürig, Michael Bergmann, Maria‐Elisabeth Goebeler, Hans‐Jürgen Hurtz, Martina Stauch, Stephan Stilgenbauer, Hartmut Döhner, Anna‐Maria Fink, Paula Cramer, Kirsten Fischer, Clemens‐Martin Wendtner, Michael Hallek, Barbara Eichhorst

    منشور في 2014
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  18. 18
    Two Years Rituximab Maintenance Vs. Observation after First Line Treatment with Bendamustine Plus Rituximab (B-R) in Patients with Waldenström's Macroglobulinemia (MW): Results of a Prospective, Randomized, Multicenter Phase 3 Study (the StiL NHL7-2008 MAINTAIN trial)

    Two Years Rituximab Maintenance Vs. Observation after First Line Treatment with Bendamustine Plus Rituximab (B-R) in Patients with Waldenström's Macroglobulinemia (MW): Results of... حسب Mathias Rummel, Christian Lerchenmüller, Manfred Hensel, Martin Göerner, Christian Buske, H. Schulz, Burkhard Schmidt, G. Kojouharoff, Elisabeth Lange, Wolfgang Willenbacher, Jan Dürig, Erik Engel, Frank Kauff, Jürgen Barth, Alexander Burchardt, Axel Hinke, Jasmin Wupperfeld, Richard Greil

    منشور في 2019
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  19. 19
    JAK/STAT-Activating Genomic Alterations Are a Hallmark of T-PLL

    JAK/STAT-Activating Genomic Alterations Are a Hallmark of T-PLL حسب Linus Wahnschaffe, Till Braun, Sanna Timonen, Anil K. Giri, Alexandra Schrader, Prerana Wagle, Henrikki Almusa, Patricia Johansson, Dorine Bellanger, Cristina López, Claudia Haferlach, Marc‐Henri Stern, Jan Dürig, Reiner Siebert, Satu Mustjoki, Tero Aittokallio, Marco Herling

    منشور في 2019
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  20. 20
    Genes encoding members of the <scp>JAK</scp>‐<scp>STAT</scp> pathway or epigenetic regulators are recurrently mutated in T‐cell prolymphocytic leukaemia

    Genes encoding members of the <scp>JAK</scp>‐<scp>STAT</scp> pathway or epigenetic regulators are recurrently mutated in T‐cell prolymphocytic leukaemia حسب Cristina López, Anke K. Bergmann, Ulrike Paul, Eva Maria Murga Penas, Inga Nagel, Matthew J. Betts, Patricia Johansson, Matthias Ritgen, Tycho Baumann, Marta Aymerich, Sandrine Jayne, Robert B. Russell, Elı́as Campo, Martin J.S. Dyer, Jan Dürig, Reiner Siebert

    منشور في 2016
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