Torthaí cuardaigh - Ho Yin Edwin Chan
- 1 - 16 toradh as 16 á dtaispeáint
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1
Drosophila models of human neurodegenerative disease de réir Ho Yin Edwin Chan, Nancy M. Bonini
Foilsithe / Cruthaithe 2000Revisão -
2
Measuring the evolutionary rate of protein–protein interaction de réir Wenfeng Qian, Xionglei He, Ho Yin Edwin Chan, Huailiang Xu, Jianzhi Zhang
Foilsithe / Cruthaithe 2011Artigo -
3
The SARS-Coronavirus Membrane protein induces apoptosis through modulating the Akt survival pathway de réir Chak-Ming Chan, Cheuk-Wing Ma, Wood Yee Chan, Ho Yin Edwin Chan
Foilsithe / Cruthaithe 2007Artigo -
4
Perturbation of U2AF65/NXF1-mediated RNA nuclear export enhances RNA toxicity in polyQ diseases de réir Ho Tsoi, Terrence Chi‐Kong Lau, Kwok‐Fai Lau, Ho Yin Edwin Chan
Foilsithe / Cruthaithe 2011Artigo -
5
Genetic modulation of polyglutamine toxicity by protein conjugation pathways in Drosophila de réir Ho Yin Edwin Chan, John M. Warrick, Isabella Andriola, Diane E. Merry, Nancy M. Bonini
Foilsithe / Cruthaithe 2002Artigo -
6
CAG expansion induces nucleolar stress in polyglutamine diseases de réir Ho Tsoi, Terrence Chi‐Kong Lau, Suk Ying Tsang, Kwok‐Fai Lau, Ho Yin Edwin Chan
Foilsithe / Cruthaithe 2012Artigo -
7
Mechanisms of chaperone suppression of polyglutamine disease: selectivity, synergy and modulation of protein solubility in Drosophila de réir Ho Yin Edwin Chan, John M. Warrick, Gladys L. Gray-Board, Henry L. Paulson, Nancy M. Bonini
Foilsithe / Cruthaithe 2000Artigo -
8
The role of ubiquitin linkages on α‐synuclein induced‐toxicity in a <i>Drosophila</i> model of Parkinson’s disease de réir Francesca K. M. Lee, Azaria Kam Yan Wong, Yuk Wa Lee, Oi Wan Wan, Ho Yin Edwin Chan, Kenny K. K. Chung
Foilsithe / Cruthaithe 2009Artigo -
9
Isorhynchophylline, a natural alkaloid, promotes the degradation of alpha-synuclein in neuronal cells via inducing autophagy de réir Jiahong Lu, Jieqiong Tan, Siva Sundara Kumar Durairajan, Liang‐Feng Liu, Zhuohua Zhang, Long Ma, Han‐Ming Shen, Ho Yin Edwin Chan, Min Li
Foilsithe / Cruthaithe 2012Artigo -
10
The ion channel activity of the SARS-coronavirus 3a protein is linked to its pro-apoptotic function de réir Chak-Ming Chan, Ho Tsoi, Wing-Man Chan, Shenyu Zhai, Ching‐On Wong, Xiaoqiang Yao, Wood Yee Chan, Stephen Kwok‐Wing Tsui, Ho Yin Edwin Chan
Foilsithe / Cruthaithe 2009Artigo -
11
Nuclear retention of full-length HTT RNA is mediated by splicing factors MBNL1 and U2AF65 de réir Xin Sun, Pan P. Li, Shanshan Zhu, Rachael Cohen, Leonard O. Marque, Christopher A. Ross, Stefan M. Pulst, Ho Yin Edwin Chan, Russell L. Margolis, Dobrila D. Rudnicki
Foilsithe / Cruthaithe 2015Artigo -
12
Chronic alcohol metabolism results in <scp>DNA</scp> repair infidelity and cell cycle‐induced senescence in neurons de réir Jacquelyne Ka‐Li Sun, Deng Wu, Genper Chi‐Ngai Wong, Tsun Ming Lau, Meigui Yang, Ronald P. Hart, Kin Ming Kwan, Ho Yin Edwin Chan, Hei‐Man Chow
Foilsithe / Cruthaithe 2023Artigo -
13
Intrinsically cell-penetrating multivalent and multitargeting ligands for myotonic dystrophy type 1 de réir Ju‐Yeon Lee, Yugang Bai, Ullas V. Chembazhi, Shaohong Peng, Kevin Yum, Long M. Luu, Lauren D. Hagler, Julio F. Serrano, Ho Yin Edwin Chan, Auinash Kalsotra, Steven C. Zimmerman
Foilsithe / Cruthaithe 2019Artigo -
14
Targeting Toxic RNAs that Cause Myotonic Dystrophy Type 1 (DM1) with a Bisamidinium Inhibitor de réir Chun‐Ho Wong, Lien Nguyen, Jessie Peh, Long M. Luu, Jeannette S. Sanchez, Stacie L. Richardson, Tiziano Tuccinardi, Ho Tsoi, Wood Yee Chan, Ho Yin Edwin Chan, Anne M. Baranger, Paul J. Hergenrother, Steven C. Zimmerman
Foilsithe / Cruthaithe 2014Artigo -
15
Pharmacologically reversible zonation-dependent endothelial cell transcriptomic changes with neurodegenerative disease associations in the aged brain de réir Lei Zhao, Zhongqi Li, Joaquim S. L. Vong, Xinyi Chen, H. M. Lai, Leo Y. C. Yan, Junzhe Huang, Samuel K. H. Sy, Xiao Yu Tian, Yü Huang, Ho Yin Edwin Chan, Hon‐Cheong So, Wai‐Lung Ng, Yamei Tang, Wei‐Jye Lin, Vincent Mok, Ho Ko
Foilsithe / Cruthaithe 2020Artigo -
16
A novel missense mutation in<i>CCDC88C</i>activates the JNK pathway and causes a dominant form of spinocerebellar ataxia de réir Ho Tsoi, Allen Chi-Shing Yu, Zhefan Stephen Chen, Nelson K N Ng, Anne Chan, Liz Y. P. Yuen, Jill Abrigo, Suk Ying Tsang, Stephen Kwok‐Wing Tsui, Tony M F Tong, Ivan F. M. Lo, Stephen T.S. Lam, Vincent Mok, Ka Sing Wong, Jacky Chi Ki Ngo, Kwok‐Fai Lau, Ting‐Fung Chan, Ho Yin Edwin Chan
Foilsithe / Cruthaithe 2014Artigo
Uirlisí cuardaigh:
Ábhair a bhaineann le hábhar
Biology
Cell biology
Gene
Genetics
Biochemistry
Medicine
RNA
Disease
Molecular biology
Apoptosis
Chemistry
Neuroscience
Internal medicine
Neurodegeneration
Nucleus
Pathology
RNA-binding protein
Allele
Autophagy
Biophysics
Cell
Cell nucleus
DNA
Drosophila melanogaster
Function (biology)
Messenger RNA
Mutant
Myotonic dystrophy
Nuclear export signal
PI3K/AKT/mTOR pathway