Хайлтын үр дүнгүүд - Hitoshi Satoh

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  1. 1
    RNA-Binding Protein Musashi2: Developmentally Regulated Expression in Neural Precursor Cells and Subpopulations of Neurons in Mammalian CNS

    RNA-Binding Protein Musashi2: Developmentally Regulated Expression in Neural Precursor Cells and Subpopulations of Neurons in Mammalian CNS -н Shin‐ichi Sakakibara, Yuki Nakamura, Hitoshi Satoh, Hideyuki Okano

    Хэвлэсэн 2001
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    Artigo
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  2. 2
    Proinflammatory Cytokine Expression in the Endolymphatic Sac During Inner Ear Inflammation

    Proinflammatory Cytokine Expression in the Endolymphatic Sac During Inner Ear Inflammation -н Hitoshi Satoh, Gary S. Firestein, Peter B. Billings, Jeffrey P. Harris, Elizabeth M. Keithley

    Хэвлэсэн 2003
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    Artigo
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  3. 3
    Intronic U50 small‐nucleolar‐RNA (snoRNA) host gene of no protein‐coding potential is mapped at the chromosome breakpoint t(3;6)(q27;q15) of human B‐cell lymphoma

    Intronic U50 small‐nucleolar‐RNA (snoRNA) host gene of no protein‐coding potential is mapped at the chromosome breakpoint t(3;6)(q27;q15) of human B‐cell lymphoma -н Ritsuko Tanaka, Hitoshi Satoh, Masatsugu Moriyama, Kasumi Satoh, Yasuyuki Morishita, Syouko Yoshida, Toshiki Watanabe, Yoshikazu Nakamura, Shigeo Mori

    Хэвлэсэн 2000
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    Artigo
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  4. 4
    BLNK suppresses pre–B-cell leukemogenesis through inhibition of JAK3

    BLNK suppresses pre–B-cell leukemogenesis through inhibition of JAK3 -н Joji Nakayama, Mutsumi Yamamoto, Katsuhiko Hayashi, Hitoshi Satoh, Kenji Bundo, Masato Kubo, Ryo Goitsuka, Michael A. Farrar, Daisuke Kitamura

    Хэвлэсэн 2008
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    Artigo
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  5. 5
    Regulation of Reactive Oxygen Species by <i>Atm</i> Is Essential for Proper Response to DNA Double-Strand Breaks in Lymphocytes

    Regulation of Reactive Oxygen Species by <i>Atm</i> Is Essential for Proper Response to DNA Double-Strand Breaks in Lymphocytes -н Keisuke Ito, Keiyo Takubo, Fumio Arai, Hitoshi Satoh, Sahoko Matsuoka, Masako Ohmura, Kazuhito Naka, Masaki Azuma, Kana Miyamoto, Kentaro Hosokawa, Yasuo Ikeda, Tak W. Mak, Toshio Suda, Atsushi Hirao

    Хэвлэсэн 2007
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    Artigo
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  6. 6
    Mice lacking a transcriptional corepressor Tob are predisposed to cancer

    Mice lacking a transcriptional corepressor Tob are predisposed to cancer -н Yutaka Yoshida, Takahisa Nakamura, Masato Komoda, Hitoshi Satoh, Toru Suzuki, Junko K. Tsuzuku, Takashi Miyasaka, Eri H. Yoshida, Hisashi Umemori, Reiko Kunisaki, Kenzaburo Tani, Shunsuke Ishii, Shigeo Mori, Masami Suganuma, Tetsuo Noda, Tadashi Yamamoto

    Хэвлэсэн 2003
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    Artigo
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  7. 7
    NOTCH3 Signaling Pathway Plays Crucial Roles in the Proliferation of ErbB2-Negative Human Breast Cancer Cells

    NOTCH3 Signaling Pathway Plays Crucial Roles in the Proliferation of ErbB2-Negative Human Breast Cancer Cells -н Noritaka Yamaguchi, Tetsunari Oyama, Emi Ito, Hitoshi Satoh, Sakura Azuma, Mitsuhiro Hayashi, Ken Shimizu, Reiko Honma, Yuka Yanagisawa, Akira Nishikawa, Mika Kawamura, Jun-ichi Imai, Susumu Ohwada, Kuniaki Tatsuta, Jun‐ichiro Inoue, Kentaro Semba, Shinya Watanabe

    Хэвлэсэн 2008
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    Artigo
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  8. 8
    Mitotic perturbation is a key mechanism of action of decitabine in myeloid tumor treatment

    Mitotic perturbation is a key mechanism of action of decitabine in myeloid tumor treatment -н Tomohiro Yabushita, Takumi Chinen, Atsuya Nishiyama, Shuhei Asada, Ruka Shimura, Tomoya Isobe, Keita Yamamoto, Naru Sato, Yutaka Enomoto, Yosuke Tanaka, Tomofusa Fukuyama, Hitoshi Satoh, Keiko Kato, Kaori Saitoh, Takamasa Ishikawa, Tomoyoshi Soga, Yasuhito Nannya, Tatsuo Fukagawa, Makoto Nakanishi, Daiju Kitagawa, Toshio Kitamura, Susumu Goyama

    Хэвлэсэн 2023
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    Artigo
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