检索结果 - George M. Martin

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  1. 1
    Epigenetic drift in aging identical twins

    Epigenetic drift in aging identical twins 由 George M. Martin

    出版 2005
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  2. 2
    Genetic Modulation of Senescent Phenotypes in Homo sapiens

    Genetic Modulation of Senescent Phenotypes in Homo sapiens 由 George M. Martin

    出版 2005
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  3. 3
    The biology of aging: 1985–2010 and beyond

    The biology of aging: 1985–2010 and beyond 由 George M. Martin

    出版 2011
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  4. 4
    Genetic Determinants of Human Health Span and Life Span: Progress and New Opportunities

    Genetic Determinants of Human Health Span and Life Span: Progress and New Opportunities 由 George M. Martin, Aviv Bergman, Nir Barzilai

    出版 2007
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  5. 5
    Healthy aging: The ultimate preventative medicine

    Healthy aging: The ultimate preventative medicine 由 Matt Kaeberlein, Peter S. Rabinovitch, George M. Martin

    出版 2015
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  6. 6
    Reinitiation of DNA synthesis in senescent human fibroblasts upon fusion with cells of unlimited growth potential.

    Reinitiation of DNA synthesis in senescent human fibroblasts upon fusion with cells of unlimited growth potential. 由 Thomas H. Norwood, W. Pendergrass, George M. Martin

    出版 1975
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  7. 7
    Oxidation of the Indole Nucleus of 5-Hydroxytryptamine and the Formation of Pigments: Isolation and Partial Characterization of a Dimer of 5-Hydroxytryptamine

    Oxidation of the Indole Nucleus of 5-Hydroxytryptamine and the Formation of Pigments: Isolation and Partial Characterization of a Dimer of 5-Hydroxytryptamine 由 Nils Eriksen, George M. Martin, Earl P. Benditt

    出版 1960
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  8. 8
    Mutator phenotype of Werner syndrome is characterized by extensive deletions.

    Mutator phenotype of Werner syndrome is characterized by extensive deletions. 由 Ken-ichiro Fukuchi, George M. Martin, Raymond J. Monnat

    出版 1989
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  9. 9
    Rapamycin decreases <scp>DNA</scp> damage accumulation and enhances cell growth of <scp>WRN</scp>‐deficient human fibroblasts

    Rapamycin decreases <scp>DNA</scp> damage accumulation and enhances cell growth of <scp>WRN</scp>‐deficient human fibroblasts 由 Bidisha Saha, A. Cypro, George M. Martin, Junko Oshima

    出版 2013
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  10. 10
    The mitochondrial theory of aging and its relationship to reactive oxygen species damage and somatic mtDNA mutations

    The mitochondrial theory of aging and its relationship to reactive oxygen species damage and somatic mtDNA mutations 由 Lawrence A. Loeb, Douglas C. Wallace, George M. Martin

    出版 2005
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  11. 11
    Cellular Aging in Werner's Syndrome: A Unique Phenotype?

    Cellular Aging in Werner's Syndrome: A Unique Phenotype? 由 Thomas H. Norwood, Holger Hoehn, Darrell Salk, George M. Martin

    出版 1979
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  12. 12
    Dominance of the Senescent Phenotype in Heterokaryons Between Replicative and Post-Replicative Human Fibroblast-Like Cells

    Dominance of the Senescent Phenotype in Heterokaryons Between Replicative and Post-Replicative Human Fibroblast-Like Cells 由 Thomas H. Norwood, William R. Pendergrass, Curtis A. Sprague, George M. Martin

    出版 1974
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  13. 13
    A Review of its Symptomatology, Natural History, Pathologic Features, Genetics And Relationship to the Natural Aging Process

    A Review of its Symptomatology, Natural History, Pathologic Features, Genetics And Relationship to the Natural Aging Process 由 CHARLES J. EPSTKIN, George M. Martin, Amelia L. Schultz, ARNO G. MOTULSKYS

    出版 1966
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  14. 14
    Accelerated telomere shortening and replicative senescence in human fibroblasts overexpressing mutant and wild-type lamin A

    Accelerated telomere shortening and replicative senescence in human fibroblasts overexpressing mutant and wild-type lamin A 由 Shurong Huang, Rosa Ana Risques, George M. Martin, Peter S. Rabinovitch, Junko Oshima

    出版 2007
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  15. 15
    Accelerated epigenetic aging in Werner syndrome

    Accelerated epigenetic aging in Werner syndrome 由 Anna Maierhofer, Julia Flunkert, Junko Oshima, George M. Martin, Thomas Haaf, Steve Horvath

    出版 2017
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  16. 16
    <scp>TNF</scp>‐α/<scp>IFN</scp>‐γ synergy amplifies senescence‐associated inflammation and <scp>SARS‐CoV</scp>‐2 receptor expression via hyper‐activated <scp>JAK</scp>/<scp>STAT1</scp>

    <scp>TNF</scp>‐α/<scp>IFN</scp>‐γ synergy amplifies senescence‐associated inflammation and <scp>SARS‐CoV</scp>‐2 receptor expression via hyper‐activated <scp>JAK</scp>/<scp>STAT1</... 由 Renuka Kandhaya‐Pillai, Xiaomeng Yang, Tamar Tchkonia, George M. Martin, James L. Kirkland, Junko Oshima

    出版 2022
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  17. 17
    Exceptional Cellular Resistance to Oxidative Damage in Long-Lived Birds Requires Active Gene Expression

    Exceptional Cellular Resistance to Oxidative Damage in Long-Lived Birds Requires Active Gene Expression 由 Charles E. Ogburn, Kristen Carlberg, Mary Ann Ottinger, D. J. Holmes, George M. Martin, Steven N. Austad

    出版 2001
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  18. 18
    Alzheimer's presenilin mutation sensitizes neural cells to apoptosis induced by trophic factor withdrawal and amyloid beta-peptide: involvement of calcium and oxyradicals.

    Alzheimer's presenilin mutation sensitizes neural cells to apoptosis induced by trophic factor withdrawal and amyloid beta-peptide: involvement of calcium and oxyradicals. 由 Qing Guo, Bryce L. Sopher, Katsutoshi Furukawa, Dao G. Pham, Nic Robinson, George M. Martin, Mark P. Mattson

    出版 1997
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  19. 19
    Somatic Mutations Are Frequent and Increase with Age in Human Kidney Epithelial Cells

    Somatic Mutations Are Frequent and Increase with Age in Human Kidney Epithelial Cells 由 George M. Martin, Charles E. Ogburn, Lorel M. Colgin, Allen M. Gown, Steven D. Edland, Raymond J. Monnat

    出版 1996
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  20. 20
    Mammalian mutator mutant with an aphidicolin-resistant DNA polymerase alpha.

    Mammalian mutator mutant with an aphidicolin-resistant DNA polymerase alpha. 由 Philip K. Liu, Chia Cheng Chang, James E. Trosko, Dipak K. Dube, George M. Martin, Lawrence A. Loeb

    出版 1983
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